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GHB vs GBL

What do you mean, possibility of dying.

Well just like any other CNS depressant, take too much or mix it with other depressants and you might just give up the ghost. Of course if you're sensible with dosage, mixing with other drugs etc then it's one of the safer ones, but not everyone is sensible.

20ml a day just sounds like it's reached the point of being a psychological crutch to me, that's what I meant (as I said, I know all about psychological crutches of the amphetamine variety)
 
Ok, well several times in the past Ive been forced to quit. Even from about 12mls a day for a months on end. Just had to quit all of a sudden and it never affected me one bit. I mean it sucked to not be able to take it when i wanted to, but i got used to it ratehr quickly and was able to lead a normal happy life all the same.
Maybe Im lucky, but I still havent found an asnwer as to why what I am doing is all that bad. Like i said before tho, I am going to get on a more appropriate dosing schedule, but even still i consider 8-10 a day to be appropriate.
 
Yeah, we've been through this before on this board, and I sent my blood panels to two or three people who PM'ed me and \asked to see them. They showed conclusively that GBL and/or GHB consumption directly impacts potassium levels. Its that simple. Monitored by a doctor, multiple tests taken over several months, heavy use. Tested both GHB and GBL. Potassium levels dropped, period. Significantly.

Mild acidosis also occurred with GBL. Irrevocable evidence. A direct, purposeful study of blood panels with several of my friends taking GBL showed identicle results -- lower potassium , mild acidosis. Occurred over several weeks with heavy use. Doctors all concurred that GBL consumption was the direct cause.

Furthermore, by supplementing GBL consumtion with an alkalyzing agent (green kamut ) and potassium (white grapefruit juice or potassium citrate -- either will work and grapefruit is the better choice), the effects disappeared completely. Also, the amount of green kamut ingested did not result in excessive or rapid alkalyzation, just stability, suggesting that the alkalyzing effects were being countered by something -- which would be the GBL consumption.

I know that Fastandbulbous gives a compelling scientific argument against this possibility, but I submit respectfully that Fastandbulbous is theorizing, whereas I have directly examined the empirical data. My observations and direct experience conflict with Fastandbulbous' theory.

Perhaps there is a process that we have yet to understand about GBL metabolism. We know that the pH of urine is affected. This, in and of itself, is indicative of a process not covered by Fastandbulbous' theory.

In any event, I do not want to start a flame war, or even communicate any disrespect, but I think it is irresponsible to advise that GBL consumption CANNOT result in either acidosis or potassium depletion. One must at least take the position that it is possible, but not yet understood, if for no other reason than because the body of empirical and anecdotal evidences strongly suggest this to be the case.

Peace.
 
synchrojet said:
Doctors all concurred that GBL consumption was the direct cause.

Thanks for the post - but what do you class as 'heavy use'? :)
 
I know that Fastandbulbous gives a compelling scientific argument against this possibility, but I submit respectfully that Fastandbulbous is theorizing, whereas I have directly examined the empirical data. My observations and direct experience conflict with Fastandbulbous' theory.

Expect certain thread closure and possible banning!!!!!!!!!!!!!!!!
 
I know that Fastandbulbous gives a compelling scientific argument against this possibility, but I submit respectfully that Fastandbulbous is theorizing, whereas I have directly examined the empirical data. My observations and direct experience conflict with Fastandbulbous' theory.

Does it? If you read all my previous posts in this thread you'll see that I don't discount the possibility of acidosis & potassium depletion; I only state that it does not happen because of the conversion of GBL to GHB. I've repeatedly referred to the fact that it might be due to endocrine activity (after all, GHB causes changes in the levels of human growth hormone - HGH), but that I've seen no mention of such in any medical journals. Increases of levels of corticosteroids can cause both acidosis and potassium depletion, that could be a possible mechanism linked to the effects on dopamine. My main conjecture here is that you can pretty much treat GBL and GHB as slightly different versions of the same drug, differing only in their pharmacokinetics. No doubt taking equivalent, chronic high doses of either GBL or GHB will cause both the same sort of effects on the human body (in terms of metabolic status and mineral ion levels).

I'm theorizing when it comes to the possible mechanism in action should any acidosis/potassium loss occur. What I'm stating as indesputable fact is that hydrolsis of GBL to GHB in the blood cannot be the cause. My other contention is that because of its incredibly short half life, any side effects seen with GBL will be the same as those seen with GHB, only that they may be slighty more prevalent with GBL bacause of the higher GHB plasma levels produced if given identical doses.


Expect certain thread closure and possible banning!!!!!!!!!!!!!!!!

Why? It wasn't insulting or facetious and was logical in it's arguement (well would have been if he'd read all my posts all the way through). Well we'll know who'll become a power mad megalomaniac if they're given moderator status. Moderators are here (shock horror!) to moderate behaviour and discussion, not to act like some ignorant feudal lord over you peasants!
 
I really cant follow this argument that well. C6H6 sed he woz sitting on a large supply of GBL. HE has not been around lately although it would be interesting to see what his consenses is on the matter. Even if it is only in lay-mans terms, it'd be interesting to see if he approves/disapproves of its utility.
 
Furthermore, by supplementing GBL consumtion with an alkalyzing agent (green kamut ) and potassium (white grapefruit juice or potassium citrate -- either will work and grapefruit is the better choice), the effects disappeared completely.
So how much potassium citrate per ml GBL or gram GHB is advised?
 
Why does something that melts plastic cups be good for the stomach or body
 
put a little in the syringe and see if it melts.

F&B- If gbl has no ph then whats makes it an irritant?
 
what about storing GBL with a vial that has a cork stopper-

has GBL been known to make its way through cork? aanyone with any experience?
 
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