That would be an interesting surprise, I personally doubt that this would be the case: with N-benzylation you create a rather selective 5-HT2A agonist and I think the empathogenic 2C-B-FLY sort of effects are likely to be thanks to effects on other receptors like other serotonergic ones, maybe a monoamine transporter, who knows (let me look that up)?
I do admit this doesn't explain some of the NBOMe's being reminiscent of their parent compounds in peculiar ways but still... Seems better to let go of analogies with the parent compound.
At low doses aren't other NBOMe's gentle?
With the longer duration you may very well be right because metabolism is being inhibited for a part. On the other hand, these are not known to bind to the 5-HT2A for extremely long times like acid so NBOMe's may just still distribute over the body and bind to plasma protein, tissues, etc. PCP has long duration too though despite being lipophilic, and low dosage, so yeah could be.
I do admit this doesn't explain some of the NBOMe's being reminiscent of their parent compounds in peculiar ways but still... Seems better to let go of analogies with the parent compound.
At low doses aren't other NBOMe's gentle?
With the longer duration you may very well be right because metabolism is being inhibited for a part. On the other hand, these are not known to bind to the 5-HT2A for extremely long times like acid so NBOMe's may just still distribute over the body and bind to plasma protein, tissues, etc. PCP has long duration too though despite being lipophilic, and low dosage, so yeah could be.