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Importing Laws - prohibited substances

Very interesting...are you able to provide us with a copy of the preliminary analysis or a more detailed summary or anything? Give our thanks to the guys that did this as well!
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Considering the work involved in getting this far, it would be unfair and unethical to release what I've been privy to, particualrly as qualification of the compounds has yet to be conclusively proven. That will require more work. So far, afaik the compounds have been tentatively identified using the Wiley/NIST database mentioned above. From last communications, for most of the compounds, identification is >70% probability.

I'm sure the BLer will be aware of your appreciation.

If I may ask, who were these guys? Were they related to BlueLight or Enlighten or another HM organization?
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The group has worked with Enlighten in the past. If you read the neorg. report on page 6 of the neodoves II thread, it explains something about this organisation.
 
Considering the work involved in getting this far, it would be unfair and unethical to release what I've been privy to, particualrly as qualification of the compounds has yet to be conclusively proven.
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No worries, PD, and let me just say your integrity is very admirable. :)
 
Costly? How much. If the blers out there might be intrested in dropping cash in the said org via internet banking... .
 
Ok, so (in theory) I have a substance I would like to import for resale....I dont know what it is(a propriatory blend), but when it is mixed with plaster of paris, it stops the plaster from going off too quickly. and gives me more time to finish the sculpture. It is not intended for human consumption. If I wish to import it, do I need to arrange for a full on GC/MA test to get it through, as I would not be willing to take the punt and as the vendor is not willing to reveal the contents , as it is "propriatory"? Any thoughts? (especially from PD)
 
bustabraincell said:
Ok, so (in theory) I have a substance I would like to import for resale....I dont know what it is(a propriatory blend), but when it is mixed with plaster of paris, it stops the plaster from going off too quickly. and gives me more time to finish the sculpture. It is not intended for human consumption. If I wish to import it, do I need to arrange for a full on GC/MA test to get it through, as I would not be willing to take the punt and as the vendor is not willing to reveal the contents , as it is "propriatory"? Any thoughts? (especially from PD)
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Whether it is for human consumption or not doesn't make much difference in australia [big diff in USA law though].
To get a conviction the prosecution has to show that you had intent of breaking the law. For this you need to be
1) aware what is illegal
2) aware that your import contained an illegal substance.
Depending on which act is applied there can be different burdens of proof, ranging from 'reasonably expected to know', to the defendant having to prove beyond reasonable doubt that he didn't know. Somethign like a drug book at home or membership at Bluelight can be enough to seriously undermine your credibility when faced with the burden of proof.

ie, if my nanna googles 'CD cleaner' online and ends up ordering from one of those GBL websites overseas that mislabel their product for deceptive purposes, then the cops will search nanna's computer and her home, and if they find no drug related stuff then they are unlikely to take the matter any further. The sender may face customs false declaration charges in his own country though.

If nanna however orders under a false name, asks for the GBL bottle to be deceptively labelled, or has a rhodium recipe on how to cook it up into GHB on her desk then nanna is toast.

if you're a 19 year old raver who's been busted for pill possession then you've got no chance of convincing a judge that you didn't know.
 
Interesting list.

I noticed that tramadol isn't on the list. Very interesting.........
 
ya perhaps carisopradol and tramadol can be imported :) but then tram is S4 here so technically not :P I cant find any reference to carisopradol's legality for possession import/export
 
My understanding is that carisopradol, like most prescription-only medicines can be taken by customs and you will be asked for a prescription to release it.
 
If something is not listed in any schedules in australia [including prescription schedules] then it is legal to import as long as it is not an analog of a prohibited drug.

Also, just because something is not in the prescription schedules doesn't mean it can't be prescribed. Docs can prescribe whatever they want. If they think ginger tea helps then they can write a script for that. not that most docs will - in fact most will only prescribe whatever is listed in the MIMS.
 
can I get a script for a big mac? and its drive thru so I might need an authority script :/
 
1. PD is DA MAN!
2. CB ags/antags dont necessarily have to be cannabinoids
3. Customs in OZ are predictably unpredictable and randomly unselective.
4. some chems that will surprise are hunted globally and actively, human rights not being a consideration
5. a little blue bird was heard to whisper 'caution, discretion, wisdom' and words to that effect, for it flew in on foul winds, it also said.
 
Phase dancer, any new information on the contents of spice gold? You say that because it was not completely antagonised by a CB1 antagonist that it can't be a synthetic cannabinoid. I disagree. Firstly, couldn't the binding affinity (for the CB receptors) of the purported synthetic canabinoid be higher than that of rimonabant, thus making its presence irrelevant? Secondly, although this is less likely, could not CB2 be reponsible for some of the effects (although I have heard that this is not psychoactive). I have smoked both spice gold and marijuana and its obvious that there is definitely cannabinoid activity of some form occuring here. There is a certain feeling that you will only get with a cannabinoid that you get with spice gold.
 
Nothing new as yet.


As I did not do the bioassay I can only pass on what has been communicated. From what I've been told, the partial reversal experienced with rimonabant was not the only thing to convince the analysts that no synthetic cannabinoids were present. However, it's certainly conceivable that some synthetic cannabinoids could have higher binding affinities.

The compounds which are thought to be responsible (at the time of receiving the last update) are believed to have indirect actions on CB1 receptors via GABA modulation. If the suspected compounds are confirmed I'm sure the mechanisms by which this occurs will be detailed. I have looked at a patent which describes the actions of this class of compound, and at this point it seems to offer a plausible explanation in accordance with what's been discovered so far.
 
Very interesting, I understand now why it is being kept under wraps, as the discovery of a new compound like that could potentially be very lucrative.

The theory of DSI (Depolarisation-induced suppresion of inhibition) claims that depolarisation of a cell causes the release of endocannabinoids, which in turn agonise the CB1 receptor (presynaptically) in order to decrease the presynaptic release of GABA.

Going from that then the only way that would make sense for GABA to reduce cannabinoid activity would be for an increased activity of GABA perhaps, which could cause an increase agonism of CB1 in order to compensate. But then again, that would make spice very sedating, which it is not, and not much in neuroscience makes sense anyway. It was nice of you to divulge a bit more though, thanks :)
 
lol nice work, gotta love this biochemical detective work!

And the spice gold just indulged myself in a smoke of it. Some of the indirect cannabinoid agonism sounds consistent with the sluggish onset and lack of any headrush on toking it I suppose.

Because it isnt so phsycoactive and lack of a rush makes it have a lower addictive/recreation potential than typical cannabis imo. But in terms of less (mental) side effects (I assume) and as a relaxant/sedative it is quite good. (and as a tool for someone addicted to pot)
 
I guess that also makes sense considering that some people say when they smoke it for like 24 hours, it begins to lose effect. Which would suggest that it is a form of indirect agonism. And yes I agree it is less psychedelic, less of a head high than weed.
 
phase_dancer said:
Nothing new as yet.


As I did not do the bioassay I can only pass on what has been communicated. From what I've been told, the partial reversal experienced with rimonabant was not the only thing to convince the analysts that no synthetic cannabinoids were present. However, it's certainly conceivable that some synthetic cannabinoids could have higher binding affinities.

The compounds which are thought to be responsible (at the time of receiving the last update) are believed to have indirect actions on CB1 receptors via GABA modulation. If the suspected compounds are confirmed I'm sure the mechanisms by which this occurs will be detailed. I have looked at a patent which describes the actions of this class of compound, and at this point it seems to offer a plausible explanation in accordance with what's been discovered so far.
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this is all very interesting indeed, as it seems that lately quite a few companies have jumped in the market with effective smoking blends, that feels exactly like spice in theyr mechanism of action (beside "smoke", wich im pretty sure it might contain an aminoalkylindole.)

its years people wonders of the exact mechanism of action of the spice, being the first effective legal blend to hit the market.

i know the spice "herbal" matter is comprised of only marshmallow, red clover and rose petals, but you are saying that NO phytochemicals of the listed plants on the package have been identified trough the gc/ms? (id imagine it would be relatively easy to find nuciferine and apomorphine from the blue lotus, for example)

i know this is my first post, but im extremely interested in the spice, as i quite like the product myself, and knowing what exactly makes the spice so.. "spicey" would be of huge interests for a lot of people.
its obvius there is some sort of cb1 and cb2 agonism involved in the effects, but that leaves a lot to speculate still.

any news for us phase_dancer?

and cheers :)
 
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