• 🇳🇿 🇲🇲 🇯🇵 🇨🇳 🇦🇺 🇦🇶 🇮🇳
    Australian & Asian
    Drug Discussion

    Welcome Guest!
    Posting Rules Bluelight Rules
  • AADD Moderators: andyturbo

GHB/1,4-B/GBL Discussion and use- Part II

It's likely 1,4-b that's been diluted with water. Any water at all will throw off the freeze point drastically.
 
I tried GHB for the second time a few weeks ago (the first time was about 5 years ago).

All I know is that my partner and I fucked like rabbits and it was the first time I've ever had a multiple orgasm (or perhaps it was one loooooong one, but god-damn it was awesome.)

4 loads in about 90 seconds and I thought I'd gone blind.
 
I dont think you'll find it will freeze at any temp. It's well known that even a small amount of dilution will throw the freeze point out to something completely unrelated to the dilution ratio.
 
I dont think you'll find it will freeze at any temp. It's well known that even a small amount of dilution will throw the freeze point out to something completely unrelated to the dilution ratio.
Click to expand...

This is bullshit. I'd explain but there's lots to consider/write and I can't do that right now. Chemical equilibrium is the fundamental reasoning here.

Hektissity said:
Just picked up some g which I believed to be 1,4-B. Slight bitterness to it not a terrible taste, no smell off 20ml. Chucked it in the fridge for close to 24 hours and it hasn't froze.

Is this most likely not 1,4-B anyone?
Click to expand...

Could be 1,4-B diluted in various different ways. Could also be GHB or GBL. It's hard to say.
 
^LOL, it's not that simple buddy.

The actual freezing temp jumps around rapidly - it's really not easy to distinguish the point from the ratio - because you have no clue what the ratio is in the first place. But i'll let you know that at no temp between 20 and 0 celcius did 2:1 (33% water) dilution of 1,4-b freeze, or thicken much at all.

READ: Apollos GHB/1,4-B guide... you'll find im not mistaken.
 
okay, sure - don't listen to what i say.

What you're trying to point out is that if it's freezing temp was 20 degrees c, at 1:1 dilution it would freeze at 10? this is just wrong.

Don't quote me but from reading i've done (ON THIS WEBSITE) the freezing points drop to things like sub-zero temps... don't patronize me as a moron because of my post count.

Otherwise, fuck, just go test it yourself then.
 
Six overdoses in one night spur Fantasy drug warning

POLICE are warning people against taking the drug GHB, known as Fantasy, after six people needed hospital treatment in Canberra on Friday night.

An ACT Policing spokesman said six people admitted to Canberra Hospital had ingested an illicit drug suspected to be GHB (Gamma Hydroxybutyrate).

The liquid drug is also known as Grievous Bodily Harm (GBH), liquid ecstasy, liquid E and blue nitro.

The six were in a serious condition when admitted but some if not all have been released, the spokesman said today.

Detectives are investigating the overdoses which all happened within the Canberra nightclub district in the city's centre, Civic.

ACT Policing's criminal investigations team has seized substances and is investigating the overdoses.

Police have urged anyone who witnesses an overdose to call ambulance services immediately on 000.

http://www.dailytelegraph.com.au/ne...asy-drug-warning/story-fndo2j43-1226484782493
 
juice, ghb

I'm getting some GHB in Melbourne, people I know call it 'juice'.

From what i've read its fairly safe if you dose correctly, etc, but people keep telling me, "don't do it, it's dirty"

Some of these people are on shards every weekend, even 3-4 times a week, they've also done GHB a number of times.

Is there something wrong with the GHB in Melbourne? (the source is reliable)

Also planning on taking 1ml with ecstasy and then another 1ml in about 2-3 hours, will it be fine to take 1mg xanax before sleep?
 
well technically, any drug is fairly safe if you dose correctly... IMO, GHB is one of the more dangerous 'party' drugs because it requires very small doses, with alot of other drugs (mainly alcohol) that can drastically increase the effects when combined (keeping in mind purity can vary from 'a shot glass worth put me on a good level' to 'a had a couple of ml and fell asleep for a few days'....

if you are not experienced with it, you shouldnt mix it with anything until you are more familiar with what dose works for you.. should probably mention my advice is not from experience but from what people have told me so your better off gettin some advice from melbourne 'juice' heads... (I know one thing about it from experience.. was at a festival a year or two ago n met some dude from melburn whom kept tellin me how he was charging on 'juice'.. from my observations, a clear side-effect of this drug is the uncontrollable urge to run up poles and bolt off in random directions whilst waving your arms frantically in the air) :\

does anyone know why Gxx is so popular in melbs? is practically non-existant in WA... I've only had it once and it was an off chance I got when a good mate's sister's boyfriend drove back a waterbottle full from, you guessed it, melburn...
 
γ-Hydroxybutyric acid (GHB), also known as 4-hydroxybutanoic acid and sodium oxybate (INN), is a naturally occurring substance found in the human central nervous system, as well as in wine, beef, small citrus fruits, and almost all animals in small amounts.[1] It is also categorized as an illegal drug in many countries.[2] It is currently regulated in Australia and New Zealand, Canada, most of Europe and in the US. GHB as the sodium salt, known as sodium oxybate, is sold by Jazz Pharmaceuticals under the name Xyrem[3] to treat cataplexy[4] and excessive daytime sleepiness in patients with narcolepsy.
GHB has been used in a medical setting as a general anesthetic, to treat conditions such as insomnia, clinical depression, narcolepsy, and alcoholism, and to improve athletic performance.[5] It is also used as an intoxicant (illegally in many jurisdictions) or as a date rape drug.[6] GHB is naturally produced in the human body's cells and is structurally related to the ketone body beta-hydroxybutyrate. As a supplement/drug, it is used most commonly in the form of a salt, for example sodium gamma-hydroxybutyrate (Na.GHB, sodium oxybate, or under the brand name Xyrem.) or potassium gamma-hydroxybutyrate (K.GHB, potassium oxybate). GHB is also produced as a result of fermentation, and so is found in small quantities in some beers and wines. Succinic semialdehyde dehydrogenase deficiency is a disease that causes GHB to accumulate in the blood.
Click to expand...

GHB is a CNS depressant used as an intoxicant.[10] It has many street names, including "Georgia Home Boy", "Juice", "Liquid Ecstasy", "Mils", "G", "Liquid X", and "Liquid G", as well as "Fantasy" and the reordered initialism GBH (abbreviation of grievous bodily harm in English law). Its effects have been described anecdotally as comparable with alcohol and ecstasy use, such as euphoria, disinhibition, enhanced sensuality and empathogenesis. At higher doses, GHB may induce nausea, dizziness, drowsiness, agitation, visual disturbances, depressed breathing, amnesia, unconsciousness, and death. The effects of GHB can last from 1.5 to 3 hours, or even longer if large doses have been consumed.[11] Consuming GHB with alcohol is dangerous as it can lead to vomiting in combination with unrouseable sleep, a potentially lethal combination.[12]
In general, the doses used recreationally are between 500 mg and 3,000 mg. When used as a recreational drug, GHB may be found as the sodium or potassium salt, which is a white crystalline powder, or as GHB salt dissolved in water to form a clear solution. The sodium salt of GHB has a salty taste.[11] Other salt forms such as calcium GHB and magnesium GHB have also been reported, but the sodium salt is by far the most common.
Some chemicals convert to GHB in the stomach and blood stream. GBL, or gamma-butyrolactone, is one such prodrug. Other prodrugs include 1,4-butanediol. There may be additional toxicity concerns with these precursors. 1,4-B and GBL are normally found as pure liquids, although they may be mixed with other more harmful solvents when intended for industrial use, e.g., as paint stripper or varnish thinner.
GHB can be easily manufactured at home with very little knowledge of chemistry, as it only involves the mixing of its two precursors, GBL and an alkali hydroxide (such as sodium hydroxide) to form the resulting GHB salt. Due to the ease of manufacture and the availability of its precursors, its production is not done in relatively few illicit laboratories like most other synthetic drugs, but in private homes by low level producers instead. While available as a prescription for rare and severe forms of sleep disorders such as narcolepsy in some other countries, notably most of Europe, GHB was banned (in the U.S.) by the FDA in 1990. However, on 17 July 2002, GHB was approved for treatment of cataplexy, often associated with narcolepsy. GHB is "colourless and odorless".[13]
Click to expand...

GHB is often taken because users find that it enhances their experiences of being in a club, party, or rave; small doses of GHB can act as a stimulant and aphrodisiac. GHB is sometimes referred to as G, liquid ecstasy, liquid X, or liquid E due to its tendency to produce euphoria and sociability and its use in the dance party scene.[14] Despite this nickname, GHB has entirely separate chemical and pharmacological modes of action compared to ecstasy.
Click to expand...

One report has suggested that Xyrem (pharmaceutical GHB, or "Sodium Oxybate") overdose may be fatal, based on deaths of three patients who had been prescribed the drug.[27] However, for two of the three cases, post-mortem GHB concentrations were 141 and 110 mg/L, which is within the expected range of concentrations for GHB after death, and the third case was a patient with a history of intentional drug overdose.[28]
One publication has investigated 226 deaths attributed to GHB.[29] Of 226 deaths included, 213 suffered cardiorespiratory arrest and 13 suffered fatal accidents. Seventy-one deaths (34%) had no co-intoxicants. Postmortem blood GHB was 18–4400 mg/L (median=347) in deaths negative for co-intoxicants.
GHB is produced in the body in very small amounts, and blood levels may climb after death to levels in the range of 30–50 mg/L.[30] Levels higher than this are found in GHB deaths. Levels lower than this may be due to GHB or to postmortem endogenous elevations.
A UK parliamentary committee commissioned report found the use of GHB to be less dangerous than tobacco and alcohol in social harms, physical harm and addiction.[31]
Click to expand...

Overdose of GHB can be difficult to treat because of its multiple effects on the body.[5][32][33] GHB tends to cause rapid unconsciousness at doses above 3500 mg, with single doses over 7000 mg often causing life-threatening respiratory depression, and higher doses still inducing bradycardia and cardiac arrest. Other side-effects include convulsions (especially when combined with stimulants), and nausea/vomiting (especially when combined with alcohol).[10]
The greatest life threat due to GHB overdose (with or without other substances) is respiratory arrest.[2][10] Other relatively common causes of death due to GHB ingestion include aspiration of vomitus, positional asphyxia, and trauma sustained while intoxicated (e.g., motor vehicle accidents while driving under the influence of GHB).[citation needed] The risk of aspiration pneumonia and positional asphyxia risk can be reduced by laying the patient down in the recovery position. People are most likely to vomit as they become unconscious, and as they wake up. It is important to keep the patient/friend awake and moving, plus do not allow them to be alone as death through vomiting can easily happen. Frequently they will be in a good mood but this does not mean they are not in danger. GHB overdose is a medical emergency and immediate assessment in an emergency department is needed.
Convulsions from GHB can be treated with diazepam or lorazepam,[10] even though these are also CNS depressants they are GABAA agonists, whereas GHB is primarily a GABAB agonist, so the benzodiazepines do not worsen CNS depression as much as might be expected.[citation needed]
Because of the faster and more complete absorption of GBL relative to GHB, its dose-response curve is steeper, and overdoses of GBL tend to be more dangerous and problematic than overdoses involving only GHB or 1,4-B. Any GHB/GBL overdose is a medical emergency and should be cared for by appropriately trained personnel.
A newer synthetic drug SCH-50911, which acts as a selective GABAB antagonist, quickly reverses GHB overdose in mice.[34] However, this treatment has yet to be tried in humans, and it is unlikely that it will be researched for this purpose in humans due to the illegal nature of clinical trials of GHB, and the lack of medical indemnity coverage inherent in using an untested treatment for a life-threatening overdose.[original research?]
Click to expand...

Although there have been reported fatalities due to GHB withdrawal, reports are inconclusive and further research is needed.[44] Addiction occurs when repeated drug use disrupts the normal balance of brain circuits that control rewards, memory and cognition, ultimately leading to compulsive drug taking.[45][46]
Click to expand...

GHB has also been associated with a withdrawal syndrome of insomnia, anxiety, and tremor that usually resolves within three to twenty-one days.[10][44][49] The withdrawal syndrome can be severe producing acute delirium and may require hospitalization in an intensive care unit for management.[10] The mainstay of treatment for severe withdrawal is supportive care and benzodiazepines for control of acute delirium, but larger doses are often required compared to acute delirium of other causes (e.g. > 100 mg/d of diazepam). Baclofen has been suggested as an alternative or adjunct to benzodiazepines based on anecdotal evidence and some animal data.[50] However, there is less experience with the use of baclofen for GHB withdrawal, and additional research in humans is needed. Baclofen was first suggested as an adjunct because benzodiazepines do not affect GABAB receptors and thus have no cross-tolerance with GHB while baclofen, which works via GABAB receptors, is cross-tolerant with GHB and may be more effective in alleviating withdrawal effects of GHB.[51]
GHB withdrawal is not widely discussed in text books and some psychiatrists, general practitioners, and even hospital emergency physicians may not be familiar with this withdrawal syndrome.[52]
Click to expand...

GHB is also produced as a result of fermentation and so is found in small quantities in some beers and wines, in particular fruit wines. However, the amount of GHB found in wine is insignificant and not sufficient to produce any effects.[60]
Click to expand...

GHB has at least two distinct binding sites[61] in the central nervous system. GHB is an agonist at the newly characterized GHB receptor, which is excitatory,[62][63] and it is a weak agonist at the GABAB receptor, which is inhibitory.[63] GHB is a naturally occurring substance that acts in a similar fashion to some neurotransmitters in the mammalian brain.[64] GHB is probably synthesized from GABA in GABAergic neurons, and released when the neurons fire.[63]
If taken orally, GABA itself does not effectively cross the blood-brain-barrier.[65]
GHB induces the accumulation of either a derivative of tryptophan or tryptophan itself in the extracellular space, possibly by increasing tryptophan transport across the blood–brain barrier. The blood content of certain neutral amino-acids, including tryptophan, is also increased by peripheral GHB administration. GHB-induced stimulation of tissue serotonin turnover may be due to an increase in tryptophan transport to the brain and in its uptake by serotonergic cells. As the serotonergic system may be involved in the regulation of sleep, mood, and anxiety, the stimulation of this system by high doses of GHB may be involved in certain neuropharmacological events induced by GHB administration.
However, at therapeutic doses, GHB reaches much higher concentrations in the brain and activates GABAB receptors, which are primarily responsible for its sedative effects.[66] GHB's sedative effects are blocked by GABAB antagonists.
The role of the GHB receptor in the behavioural effects induced by GHB is more complex. GHB receptors are densely expressed in many areas of the brain, including the cortex and hippocampus, and these are the receptors that GHB displays the highest affinity for. There has been somewhat limited research into the GHB receptor; however, there is evidence that activation of the GHB receptor in some brain areas results in the release of glutamate, the principal excitatory neurotransmitter.[56] Drugs that selectively activate the GHB receptor cause absence seizures in high doses, as do GHB and GABA(B) agonists.[67]
Click to expand...

http://en.wikipedia.org/wiki/Gamma-Hydroxybutyric_acid
 
Juice isn't any 'dirtier' than most recreational drugs, in fact it has less side effects than most (including alcohol) and is much less likely to be impure or contaminated.

In terms of effects, I've found it to be rather similar to alcohol, except it isn't as harsh and uncomfortable on the body, and it induces more euphoria (and more consistently).

The problem is that people are stupid about dosing it. As a fairly concentrated liquid of unknown purity, you have to start slow when measuring a new batch, always use a plunger and track your intake, don't just sip it here and there whenever you feel like it. And it should go without saying, but for the love of god don't combine it with alcohol or other sedatives.
 
Hey mazzoj I merged your thread with this one given it is our current thread on use of GHB.

First off, poly drug use (that is combining multiple drugs) can be dangerous at the best of times, it is especially so when using one or more substances you are unfamiliar with as part of that mix. I would atleast get a couple doses of G and nothing else under your belt before combining it with anything. MDMA and GHB I don't *think* is an overly risky combination when both drugs are used in reasonable doses, however DON'T take that as gospel I just don't remember reading about any interactions. The xanax is a different story though, I would think by mixing the two CNS depressants you are putting yourself at a real risk of serious respiratory depression.

There is nothing 'wrong' with G in Melbourne (unless a batch has been contaminated!) but you are a lot more likely to be getting 1,4b or GBL than GHB, especially 1,4b seems to be by far the most common here ime. 1,4b and GBL both put more stress on the body than GHB purely because the body has to work to convert them into GHB. There does seem to be a perception amongst a lot of drug users, even hard drug users, that G is a dirty or low drug to be using, I really don't know where it stems from but I do know they tend to like to regurgitate "its DRAIN CLEANER" etc. etc. kind of ironic that the same people will drink alcohol which has also been used as an industrial solvent.

If you do decide to get some start small because you can always work your way up and take more but you can't untake your dose once its down the hatch. Better safe than sorry, its easy to OD on G.
 
You must log in or register to reply here.
Top