Warning for vapers
If you are choosing to vapourise this compound please ensure you are actually vapourising it and not thermally decomposing/degrading by combustion.
3-FPM should run clear, the liquid phase is translucent. If you are getting brown/dark amber/black trails you are
burning rather than converting to the gaseous state. It is not a semantic difference. It is not a minor thing. Do not think otherwise. Combustion of Phenylamines (all Amphetamines/Phenylmorpholines and a vast amount more) by excessive heating releases vastly toxic byproducts once the molecular bonds are broken.
In particular, Nitric Oxide (NO) fumes are given off readily from the amine moiety - NO is a
free radical molecule which oxidises in air to form NO2. NO2 is highly toxic when inhaled and of a characteristic amber-red-brown colouration dependent on temperature, getting darker as the heat increases.
Both NO and NO2 are
highly biotoxic when inhaled, causing severe respiratory damage and destruction to mucousal bodies via peroxidation of lipid membranes. The hydrolysis of NO2 upon contact with water forms Nitric (HNO2) and Nitrous (HNO3) acid which are directly corrosive to tissue membranes.
From Wiki: "Exposure to high level of nitrogen dioxide may lead to inflammation of the mucous membrane and the lower and upper respiratory tracts. The symptoms of acute poisoning are non-specific and have a semblance with ammonia poisoning, chlorine gas poisoning, and carbon monoxide poisoning. The symptoms also resemble that of pneumonia or viral infection and other inhalational injuries, but common symptoms includes rhinitis, wheezing or coughing, conjunctivitis, headache, throat irritation and dyspnea which may progress to nasal fissures, ulcerations, or perforation".
NO is a potent inflammatory agent and a direct cytotoxin -
this paper details the resultant apoptosis of somatic cells and
the capacity of NO to suppress the immune response of phagocytic and T-cells and actively damage DNA.
Eventual consequences include
pulmonary oedema, pulmonary obstruction, bronchiolitis and damage to Haemoglobin function
The reduction in both oxygen capacity of Hb and eventual haemolysis leads to severe hypoxia on a systemic level, with symptoms reminiscent of cyanosis.