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The Big & Dandy 2C-C Thread

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After reading through this thread a bit it sounds to me like the closest comparison would be to 2C-D. I've never had the opportunity to try 2C-C, but may in the near future. At this point I'm leaning more towards 2C-T-2 though. Anyone have favorable/disfavorable comparisons of these two?
 
I prefer 2C-T-2 by a good margin, although granted it is dear to me because it helped kick me out of depression years back. I've tried both, actually I've used 2C-C more by now just because I've only ever had 100mg of T-2. For me, I never was able to get anywhere special on 2C-C. It seems sort of like a generic 2C-X feeling that's smoother than most but without anything to really set it apart... no insights or anything. Nice, but not a favorite.

2C-T-2 is several level more psychedelic for me. It seems to have the effect of splitting the mind apart into its various component parts and elevating you as the observer above all of the parts of the ego... I was able to watch objectively how my mind was working against itself and learn how to help fix that. And it's done with an edge of calm and peace. I really learned a lot about myself with 2C-T-2... it's one of my favorites.
 
Yeah 2C-C was one of the last psychedelics I took at a decent dose (25mg, not bad for a first time...) and thus the only state I can remember with some clarity... I'm thinking of trying 2C-T-2 soon so if I realize any interesting parallels between the mind states I'll be sure to write them down and come back to report to you guys :)
 
I really really wonder what mixing 4-aco-dmt with 2c-c would be like.
If they are both gentle and subtle in their own right.
i bet a 50mg + 50mg trip would be a fantastical experience.
lol can anyone give me a reason not too?
 
I wouldn't take quite that much psilacetin... that might be more than a little overwhelming, based on my experience anyway. Much above 30mg and I start to have lapses in memory etc
 
^ That's what I said to him, but for this guy 50mg of 4-AcO-DMT is half his highest dose! My only concerns with that combo is the length, the body high might overwhelm even the biggest hardhead if 50mg of 2C-C and the same of 4-AcO-DMT sneak up on each other... Perhaps staggering the doses into 2x25mg doses of each would help deal with the relative shortness of both chemicals?
 
So I have horrible body load and nausea from 2c-e and the one time I tried 2c-i, I had similar problems. So much so that I have abandoned bboth substances, albeit I am going to revisit to 2c-e plugged. Is 2c-c right for me?

I'm looking for responses particularly from people who have been turned off by intense nausea and body load from 2c-i and 2c-e but have used 2c-c successfully. Thank you.
 
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I haven't done 2c-i or 2c-e, but i've managed to avoid nausea from 2c-b/d/c by plugging.

Oral doses of 2c-b and 2c-d made me feel queasy but I didn't get sick at all from plugging.
 
ungelesene_bettlek said:
can it also be taken nasally? if yes, how much more potent is it that way? does it hurt as bad as 2C-B?
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Personally, I find 2C-C much less painful than 2C-B nasally; although sometimes the drip makes my throat a little sore. I think it's around 2x more potent nasally than orally, although I've only rarely used it nasally on its own... I found 20 mg nasally in two doses spaced over about 10 minutes (from sober) to produce considerable euphoria but little psychedelia; whereas 10 mg nasally on top of various other (oral) psychedelics (on separate occasions: 100 mg DiPT; 25 mg 2C-B; 66 mg 2C-C; for instance) has had rather strong effects (typically pushing me over from a moderate ++ to a full-on +++).
 
visual/ euphoria connection

djfriendly said:
^ I've seen the clockwork also, closed-eye. Multi-colored gears and cogs, everywhere. It somehow meshed with my emotional state, and I felt I was experiencing what it was to be the various colors. For me, 2C-C often combines emotions with other stimuli.
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Yes I also know of someone who took about 80-90 mg of 2cc and said it was a great time and lasted almost 12 hours. The visuals were cool and some how connected with waves of euphoria, she said when concentrating on the visuals certain patterns would bring on these waves of esctasy.
 
fastandbulbous said:
For the sake of comparison, 2C-C is a different creature alltogether compared with 2C-D. The nature of the substituent on the 4 position determines the basic nature of the experience (4-halo compounds have a common theme as do 4-thioalkyl and 4-alkyl). For 'depth', I think the 4-alkyl substituted compounds are the one required, for entactogenic activity the 4-halogen and the 4-thioalkyl are somewhere inbetween.

All to do with size, shape and electron withdrawing capacity when seated in the 5HT2a receptor.

Then there are just the 'weirdos' like 2C-T-21 & 2C-N which are a sort of 'pick 'n' mix' type of experience!
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It sounds like you understand some things I have been trying to figure out. These 5ht2 agonists actually bind to the receptor in place of 5ht or are both still binding? So in your opinion does 5htp increase the activity of any of these? How do ssri's work if all that 5ht is just sitting in the synapse but can't be taken up? Any one? Cocaine/ methamp does the same thing but with dopamine what going in?
 
I really enjoyed 2c-c <3 it came with quite an uneasy come up for me, lots of vomiting and shivvers and aches but once I got past that I got some really interesting psychedelic effects. The total duration of the trip was 6ish hours.

My favorite and most memorable hallucination was an auditory one; my nebighors were speaking in polish (which i dont speak) outside our window and I understood what the were saying in my mind but I was hearing it in Spanish (which I do speak)... very strange. I find that languages become very intermingled in my mind while tripping quite often, but I never have understood a language which I do not speak. ;) <3

The trip was very mellow, not quite as mellow and sedated as I expected it to be, as a matter of fact it was much more of a trip than I was prepared for, but that turned out for the best, as it usually does.
 
lsg said:
It sounds like you understand some things I have been trying to figure out. These 5ht2 agonists actually bind to the receptor in place of 5ht or are both still binding? So in your opinion does 5htp increase the activity of any of these? How do ssri's work if all that 5ht is just sitting in the synapse but can't be taken up? Any one? Cocaine/ methamp does the same thing but with dopamine what going in?
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Psychedelics bind to the 5-HT2A receptors in place of serotonin. Different effects are perceived due to the differences in chemical structure between these molecules and serotonin.

5HTP is merely a precursor for serotonin and provides help if serotonin levels in the bain are running low. For example, monoamine releasers such as MDMA deplete serotonin, which is why the comedown is so harsh and also why supplementing with serotonin precursors such as 5HTP and L-Tryptophan helps to alleviate the crash.

SSRIs simply bind to SERT (serotonin transporter), effectively inhibiting the uptake of serotonin back into the presynaptic cell. This in turn causes higher levels of serotonin in the synapses. They don't block the binding of serotonin to the receptors (which would be called antagonism instead of reuptake inhibition).
 
Recept said:
Psychedelics bind to the 5-HT2A receptors in place of serotonin. Different effects are perceived due to the differences in chemical structure between these molecules and serotonin.
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how does this difference work exactly? as I understand it, a recepter can either be active or not.

Recept said:
SSRIs simply bind to SERT (serotonin transporter), effectively inhibiting the uptake of serotonin back into the presynaptic cell. This in turn causes higher levels of serotonin in the synapses. They don't block the binding of serotonin to the receptors (which would be called antagonism instead of reuptake inhibition).
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well, there are actually some atypical antidepressants (like trazodone, for example) which act as serotonine antagonist. I never understood why that makes them antidepressant, though...
 
ungelesene_bettlek said:
how does this difference work exactly? as I understand it, a recepter can either be active or not.
Click to expand...
I'm not sure, I could be wrong entirely but to my best understanding, the receptor is activated when a chemical mimicking the neurotransmitter (serotonin in this case) binds to it. But regarding the difference in effects, could be that different molecules have different electrical charge and cause different signals to be mediated? Or maybe just different molecules access different areas of the brain to cause different effects? I don't know, I'm confused now, perhaps someone with better understanding of pharmacology could chip in and explain this better :D

ungelesene_bettlek said:
well, there are actually some atypical antidepressants (like trazodone, for example) which act as serotonine antagonist. I never understood why that makes them antidepressant, though...
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Me neither. Serotonin antagonism sounds nasty to me.
 
Most psych's affect various 5HT receptors, not just 2a. These differences in affinity cause some pharmacological differences in various psychedelics. There's also significant differences in bioavailability, metabolism, metabolites, absorption, elimination, and plenty of other factors as well for various psych's.
 
GREAT chemical, loving the bodyhigh it's centering and it feels worthwhile, this is such clean clear stuff! Took about 15 mg IN op top of R-(-)-ketamine and just a pinch of mephedrone, and I feel so much better in my skin this is awesome!
 
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