Kratom is stimulating, at least in low doses. I think there's reason to believe that it hits some of the adrenergic receptors at any dose. If I recall correctly, one of the alkaloids in kratom is a delta opiate agonist, which means that it lowers the seizure threshold, making a seizure more likely. Adding selegiline compounds these mechanisms. Phenibut will counteract this excitability, but I wouldn't count on it. If you can't, or won't, see a real doctor to deal with your problems constructively, start with a very low dose, and work your way up.
Antidepressants aren't known for that. There are also plenty of stimulating antidepressants on the market (pristiq, effexor, cymbalta, wellbutrin, some MAOIs).
Selegiline doesn't really have a documented antidepressant effect at lower doses, because at these doses it pretty selectively inhibits MAO-B, which deals with sympathomimetic amines (norepinephrine, dopamine, trace amines). At higher doses it inhibits MAO-B and MAO-A, which means that it additionally slows down the metabolism of serotonin. Like it or not, the great majority of antidepressants are effective at doses that somehow (if ephemerally) increase synaptic concentrations of serotonin.
If you decide to take selegiline at lower doses, be extremely careful taking any other stimulants with it. If you're taking it at higher doses, also stay away from hallucinogens, and you should go on a diet to eliminate various neurotransmitter precursors.
So in summary, what you're proposing is pretty dangerous and reckless. People have died in great pain because they didn't follow the diet or because they abused drugs while on MAOIs.
