is this possible?
One doesn't need "full doses", one needs a full cycle. That's the way the human body and hormonal system works. Stable blood levels. All you'll do by taking random doses is throw your HPTA off balance and shut your testosterone down. I know this is a fleeting concept you can't take one dose and immediately notice results like you do with oxycontin.why not??
why do you need full doses too notice an effect?
i've heard somewhere that stanozolol lowers SHBG at lower doses (5 mg / day). so it can "release" natural testosterone, but then again, why not just go with artificial testosterone in the first place???
One doesn't need "full doses", one needs a full cycle. That's the way the human body and hormonal system works. Stable blood levels. All you'll do by taking random doses is throw your HPTA off balance and shut your testosterone down. I know this is a fleeting concept you can't take one dose and immediately notice results like you do with oxycontin.
The only thing you need to enhance your results is a gallon of milk a day and a good routine. Since you're probably doing neither of these things, you should go back to the drawing board and evaluate your diet and lifestyle before doing something you may soon regret.
i've heard somewhere that stanozolol lowers SHBG at lower doses (5 mg / day). so it can "release" natural testosterone, but then again, why not just go with artificial testosterone in the first place???
If you want to reduce side effects and shutdown, it's better to run a short cycle at a reasonable dosage than to run a long underdosed cycle. For example you could do 8 weeks of Test E at 500mg/week (you still need an AI and PCT!).what would be a good steroid for enhancing workouts?
i dont want too go full out, just take very small doses too enhance results a little bit ?
is this possible?
If you were a woman I would want to have sex with you because of the posts you make.
The effects of Winstrol or Proviron on SHBG don't really make a difference in free testosterone. That's probably because free testosterone is metabolized more quickly. They are also useless as anti-estrogens. If you want more free testosterone, you have to raise your total T by injecting more of it. If you want to lower your estrogen, use an AI.
Proviron is an androgen without any anabolic effects and therefore only useful as a libido enhancer ON CYLCE (Proviron is suppressive just like any other androgen, so off cycle use will shut you down). Winstrol is only useful for contest prep. It's a very weak anabolic that really only drys you out (especially your joints).
Thats why low dose Winstrol or even Proviron is added to a cycle for their synergistic effects of lowering SHBG therefore freeing up more plasma Testosterone, plus by their nature they are suppressive of Estrogen....
Fuck Winstrol. People should use either Proviron, Masteron or even Epistane.
I Think Winstrol is a bit like most compounds Guido... Person & dose dependant..!!
Used at 20-30mg/day with a base of Test' plus Boldernone I've used it for years & never had any issues....
It can be great as part of a pre-contest cut, I think the trick is to not let the dose get too high...
But I know what you mean, if its not your thing it can be quite harsh on some people....
More free testosterone also means quicker metabolization. It's true that SHBG inactivates testosterone, but it also protects it from metabolization. So even if a steroid did free up more testosterone (I'm not sure if this has actually been proven), the eleveation of free testosterone probably wouldn't last for more than a few days.It is my understanding DHT derivatives like Masteron, Winstrol, Proviron bind well to SHBG, thus they can free up more testosterone in blood plasma which is then biologically active and able to enter a cell and activate its receptor.....
Why would anyone rely on DHT derivatives (that may or may not be weak AIs) if strong, scientifically proven AIs are readily availabe for estrogen control?All are excellent when added to a cycle for their synergistic effects of lowering SHBG and as a mild anti-E by binding to the aromatise enzyme...
Why would anyone rely on DHT derivatives (that may or may not be weak AIs) if strong, scientifically proven AIs are readily availabe for estrogen control?