Can we have some discussion on the metabolism of dimethocaine? If, as Hammilton mentioned, the ester is cleaved via pseduocholinesterase, PABA and 3-(diethylamino)-2,2-dimethylpropan-1-ol would be the expected products.
So then, are there any forseeable problems that could arise from repeated dosing and/or regular daily use? PABA, when taken as a supplement (for those without pseudocholinesterase deficiency) is recommended at ~400mg max/ day. However, having known a few coacaine users over the years, it's quite conceivable this level might be exceeded if dimethocaine was used as a replacement.
If the second metabolite is 3-(diethylamino)-2,2-dimethylpropan-1-ol, what sort of side effects could result high dosage use? Would it be excreted fast enough to avoid issues of toxicity? I believe there's been some research done on it as a possible anticancer agent, although from PubChem it appears it was inactive in all assays. The anion is also found as an ester in Syntropan (a parasympatholytic).