dopamimetic
Bluelighter
Is there anything new related to the cardiotoxicity of the 2-fluorinated amphetamines? If you know anything, how that person who had heart damage due to 2-FMA is doing now, what the docs have found out etc.. -please- share it!
I have only used them occasionally and in low dose yet, and as most people report, it feels so smooth and functional with much less hangover than medical d-amp (where after even two or three doses I have a strong feeling of a toxic metabolite lingering- maybe alpha-methyldopamine). I've noticed increase in pulse and BP of course, but not more than expected.
So this is really sad and I / we really need more background information on this!
Is there really some specific toxic metabolite to 2-FMA that builds up in fatty tissue? How does this one cause the damage, by vasoconstriction, adrenergic activity, 5-HT2B agonism, ...? Does this apply to 2-FA too or not? If we have it to do with something that is adrenergic / cardio related, would one be able to detect the changes by monitoring blood pressure and pulse- and since I take 150mcg of clonidine regularly before sleep (clonidine lets me sleep even with amps still active by shutting down norepinephrine) and sometimes 75mcg together with a stimulant to counter-act side effects, is it a possibility that this might alleviate the cardiotoxicity?
I have only used them occasionally and in low dose yet, and as most people report, it feels so smooth and functional with much less hangover than medical d-amp (where after even two or three doses I have a strong feeling of a toxic metabolite lingering- maybe alpha-methyldopamine). I've noticed increase in pulse and BP of course, but not more than expected.
So this is really sad and I / we really need more background information on this!
Is there really some specific toxic metabolite to 2-FMA that builds up in fatty tissue? How does this one cause the damage, by vasoconstriction, adrenergic activity, 5-HT2B agonism, ...? Does this apply to 2-FA too or not? If we have it to do with something that is adrenergic / cardio related, would one be able to detect the changes by monitoring blood pressure and pulse- and since I take 150mcg of clonidine regularly before sleep (clonidine lets me sleep even with amps still active by shutting down norepinephrine) and sometimes 75mcg together with a stimulant to counter-act side effects, is it a possibility that this might alleviate the cardiotoxicity?