This was more of a "scientific curiosity" - of whether or not the oral activity was true oral activity, or may be falsified by rapid absorption on mucuous membranes before entering the stomach. I don't remember where exactly, but there was one post where some chemist claimed oral activity for some NBOx, and later had to correct themselves after encapsulating the compound. Dissolving in a huge volume of liquid and chugging it down may keep absorbtion down to a small level. Using a capsule is the better option for controlling the variables in this experiment. Though clearly, eating a tab instead of using it sublingually is also not really harm reduction at all for obvious reasons: 1) it seems clear that NBOMe/NBOH compounds may be absorbed before the stomach or they may be orally active, 2) there are plenty of compounds which fit on a tab which are usually not desired (i.e. not LSD) and which definitely are active orally.
I only have a single experience on a NBOH-type compound (25E-NBOH), sublingually, and it was a disaster enough to have a very healthy respect for this class of compounds now and me not wishing to repeat an experiment in the foreseeable future.
I'm still in the process of figuring stuff out and writing a trip report for that one, although I'm not sure how, when and if I will publish it. Harm reduction efforts demand it though, so in the meantime I can only say: 1.2 mg of the hydrochloride were decidedly too much, and even for a reasonable experienced psychonaut, the stimulation, the sensory overload from all the visuals and the bad stigma of the NBOx compounds make for an excellent combination for substance-induced psychosis and panic loops.