《Plasticity》
Bluelight Crew
- Joined
- Sep 21, 2013
- Messages
- 3,115
Oddly, acquisition of tolerance to kratom didn't affect my response to hydrocodone much.
You see, this has been the case for me and I've been an everyday user for ~4 years now. I just don't think plain leaf is a strong enough mu agonist to cause any sort of significant tolerance to full agonist opioids, I think it's only when people get into extracts that people get significant cross-tolerance. I mean I can get high off nearly the same doses of opioids as I was able to before getting on kratom.
I think the main reason amanitadine experiences extreme tolerance is because he uses insane doses of these synthetic extracts, in the kratom megathread he states that his dose of UEI is 7 grams on top of his normal plain leaf dose and his dose of FST is a whole 2ml bottle 8(, no wonder he's experiencing severe tolerance. Also he has said a few times that he has a huge history of prior opiate abuse and we all know that once you get addicted even once how easy it is to re instate your previous tolerance, especially with these insane extract doses as well as having an increased severity of withdrawals despite the mu agonist in question.
For example, say someone has a 250mg a day oxy habit and switches to kratom. This persons tolerance would be reduced but if this person starts eating huge doses of these potent extracts equivalent to god knows how much oxy your tolerance is gonna shoot right back up, I believe this phenomena is known as "kindling" and it seems very apparent this is the cause of such a huge tolerance in amanita's case. There's no fucking way any amount of plain leaf will cause a tolerance so significant to render 180mgs of oxy useless, especially when I dose near 30gs daily and still get high from 40mgs of oxy. The combination of extract use and prior history is to blame IMO no doubt, maybe kratom didn't help but you can't blame your tolerance issues solely on kratom, that's extremely unfair.