narco anonomous
Bluelighter
- Joined
- Mar 2, 2006
- Messages
- 229
Yeah, there's a slight difference, but its just as bad in my opinion.
That's nice, it is after all your opinion, nothing more, nothing less.
I know a couple people who were put on amphetamines even younger than 7 years old, one is a psychopathic bi polar raging drug addict, the other has psychosis and is convinced that the crips and the bloods are fighting over him and trying to recruit him, and claims to hear them talking about him on the radio. Both used to be very good friends. And the second dude i mentioned, he never abused drugs. Only took his adderal as prescribed and smoked weed. He went crazy when he decided to stop taking it. And first guy i mentioned, psychopathic doesn't mean he kills people. Thats a sociopath.
This is the second post I've read by you where you have gotten your facts very wrong. The first was when you mentioned a study suggesting kids could get heroin more easily than alcohol. It was weed, the idea that a kid could get heroin more easily than alcohol is absurd, but that's irrelevant.
A psychopath is considered an organic brain disorder in which the limbic system does not function correctly or functions in a very different fashion. This predisposes the inflicted individual to crime, as they are more inclined, and better able to commit them. When you scan a psychopaths brain with an fMRI while say... using word association, words like baby and rape will elicit the same emotional response, that is none what so ever. Usually when a word is processed by the sensory cortex it gets sent along a feedback loop through the frontal, temporal, and other peripheral brain areas involved with language such as wernicke's area where the cortexes meet. In a psychopath this loop skips the parts of the brain involved with emotion, temporal lobe, and goes strait to the verbal areas. It has superficial similarities to the situation high functioning autistics find themselves in, they do not have the social structure others find or pick up innately. That is why it is so pointless to treat psychopaths, and sociopaths to a lesser extent as well but not much less, because they will use group therapy to study how normal people emote.
Sociopath is less organic, although still involving neurology it has more arrows pointing down from environment and society than up from genetics and neurology. They are very similar, although the sociopath would in theory be less methodical and organized in their criminal activities. These are people who were not necessarily born this way, and it isn't so much that they cannot emote, rather something is interfering with this process, or they have become less receptive to it. Again it's irrelevant, the idea that childhood amphetamine was THE causative agent in your friends unfortunate schizophrenia development is implausible, there are too many other variables, genetics, and as you said marijuana use. That the symptoms intensified as he discontinued his medication indicates nothing clearly, as correlation does not imply causation.
"I have seen research published that suggests amphetamines are toxic in therapeutic doses. Regardless, making your childs brain develop on speed is almost as bad as giving a developed brain meth."
I have seen research that suggests MDMA puts holes in your brain, and that vaccines cause autism. Now, in mice, there is evidence that amphetamine administered to adolescent white mice (comparable to a 5-9 year old child, in theory) causes an up to 25% reduction in the amount of striatal dopamine receptors found in adulthood. Obviously this isn't any kind of conclusive evidence, and the dosage used was not necessarily exact in mimicking therapeutic doses. That said there isn't enough evidence to suggest that therapeutic dosage of amphetamine after age 15 causes significant brain damage. I have been using amphetamine to augment my focusing capacity for nearly 8 years, and although I take weekend breaks, I still feel that my anecdotal evidence suggests that whatever damage it may do is insignificant. I make that claim because I am sharper than an ice pick. Now, methylphenidate on the other hand is actually thought to be a neuro protective, so I think that in and of it's self should dispel any further conversation about ADD medication and street meth amphetamine being analogous. AND, if brain damage is significant, I still feel that this medication provides a valuable tool for many people, just as it might be possible to reduce or eliminate the development of opiate tolerance, it would be possible through NMDA antagonists, which assuming the secondary medication was safe as well, reduce or eliminate striatal dopamine depletion and thus any significant damage that might result.
I actually can't find the article about striatal dopamine in mice sorry. Also it's possible that amphetamine use could trigger the release of free radicals, but again that would indicate that treatment should be augmented not discontinued.
http://www.ncbi.nlm.nih.gov/pubmed/1436384 Article about NMDA receptor antagonists
http://amphetamines.com/adhd/drugsbrain.html Overview of ADD medication and possible risks.
http://www.sciencedirect.com/scienc...3891e563313bea51bad43fc821ce2751&searchtype=a article about frontal cortex damage increasing the brains receptivity to amphetamine, which I think is quite indicative of amphetamines value in dealing with neural incongruence.
Oh and http://www.ncbi.nlm.nih.gov/pubmed/16736241 article titled methylphenidate exerts no neurotoxic, but neuroprotective effects in vitro.
That's nice, it is after all your opinion, nothing more, nothing less.
I know a couple people who were put on amphetamines even younger than 7 years old, one is a psychopathic bi polar raging drug addict, the other has psychosis and is convinced that the crips and the bloods are fighting over him and trying to recruit him, and claims to hear them talking about him on the radio. Both used to be very good friends. And the second dude i mentioned, he never abused drugs. Only took his adderal as prescribed and smoked weed. He went crazy when he decided to stop taking it. And first guy i mentioned, psychopathic doesn't mean he kills people. Thats a sociopath.
This is the second post I've read by you where you have gotten your facts very wrong. The first was when you mentioned a study suggesting kids could get heroin more easily than alcohol. It was weed, the idea that a kid could get heroin more easily than alcohol is absurd, but that's irrelevant.
A psychopath is considered an organic brain disorder in which the limbic system does not function correctly or functions in a very different fashion. This predisposes the inflicted individual to crime, as they are more inclined, and better able to commit them. When you scan a psychopaths brain with an fMRI while say... using word association, words like baby and rape will elicit the same emotional response, that is none what so ever. Usually when a word is processed by the sensory cortex it gets sent along a feedback loop through the frontal, temporal, and other peripheral brain areas involved with language such as wernicke's area where the cortexes meet. In a psychopath this loop skips the parts of the brain involved with emotion, temporal lobe, and goes strait to the verbal areas. It has superficial similarities to the situation high functioning autistics find themselves in, they do not have the social structure others find or pick up innately. That is why it is so pointless to treat psychopaths, and sociopaths to a lesser extent as well but not much less, because they will use group therapy to study how normal people emote.
Sociopath is less organic, although still involving neurology it has more arrows pointing down from environment and society than up from genetics and neurology. They are very similar, although the sociopath would in theory be less methodical and organized in their criminal activities. These are people who were not necessarily born this way, and it isn't so much that they cannot emote, rather something is interfering with this process, or they have become less receptive to it. Again it's irrelevant, the idea that childhood amphetamine was THE causative agent in your friends unfortunate schizophrenia development is implausible, there are too many other variables, genetics, and as you said marijuana use. That the symptoms intensified as he discontinued his medication indicates nothing clearly, as correlation does not imply causation.
"I have seen research published that suggests amphetamines are toxic in therapeutic doses. Regardless, making your childs brain develop on speed is almost as bad as giving a developed brain meth."
I have seen research that suggests MDMA puts holes in your brain, and that vaccines cause autism. Now, in mice, there is evidence that amphetamine administered to adolescent white mice (comparable to a 5-9 year old child, in theory) causes an up to 25% reduction in the amount of striatal dopamine receptors found in adulthood. Obviously this isn't any kind of conclusive evidence, and the dosage used was not necessarily exact in mimicking therapeutic doses. That said there isn't enough evidence to suggest that therapeutic dosage of amphetamine after age 15 causes significant brain damage. I have been using amphetamine to augment my focusing capacity for nearly 8 years, and although I take weekend breaks, I still feel that my anecdotal evidence suggests that whatever damage it may do is insignificant. I make that claim because I am sharper than an ice pick. Now, methylphenidate on the other hand is actually thought to be a neuro protective, so I think that in and of it's self should dispel any further conversation about ADD medication and street meth amphetamine being analogous. AND, if brain damage is significant, I still feel that this medication provides a valuable tool for many people, just as it might be possible to reduce or eliminate the development of opiate tolerance, it would be possible through NMDA antagonists, which assuming the secondary medication was safe as well, reduce or eliminate striatal dopamine depletion and thus any significant damage that might result.
I actually can't find the article about striatal dopamine in mice sorry. Also it's possible that amphetamine use could trigger the release of free radicals, but again that would indicate that treatment should be augmented not discontinued.
http://www.ncbi.nlm.nih.gov/pubmed/1436384 Article about NMDA receptor antagonists
http://amphetamines.com/adhd/drugsbrain.html Overview of ADD medication and possible risks.
http://www.sciencedirect.com/scienc...3891e563313bea51bad43fc821ce2751&searchtype=a article about frontal cortex damage increasing the brains receptivity to amphetamine, which I think is quite indicative of amphetamines value in dealing with neural incongruence.
Oh and http://www.ncbi.nlm.nih.gov/pubmed/16736241 article titled methylphenidate exerts no neurotoxic, but neuroprotective effects in vitro.
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