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Tryptamines The Big & Dandy 5-MeO-DiPT Thread

Originally posted by bomb:
IMHO, foxy can easily/sometimes be more fun than LSD. (though I would take LSD 9 times out of 10 over 5-meo-dipt) especially when mixed with pot. Why do you ask? well LSD can lead you away to hell if you let it, and foxy is much less inclined to do so. It is also much more controlable which is a plus if you are out in public (aka clubbing :) )

My opinion exactly. The benefit of foxy is that it gives me a nice body high, interesting audio effects, and mild visuals with minimal mind fuck. These effects, combined with its controllability, make it a good party drug. Unlike with acid, the lack of mindfuck actually enables you to hold intelligible conversations with non-tripping people. If you are taking a dose that gives you unpleasant side effects, chances are that the dose is too high. Foxy is not supposed to be an intense, acid-like psychedelic, and if you try to have an acid-like trip off it, chances are you will take a dose that is WAY more than optimal. I get threshold effects at about 10 mg, and my preferred dosage is 15-20 mg. Anything above 20 mg is simply TOO MUCH for me, even though I could have an intense trip off a higher dose. When I accidentally took 25-30 mg of foxy, the body high was so intense that I could hardly feel certain parts of my body. It was certainly NOT worth the insane visuals I got. If I wanted visuals like that, I'd just take 5 hits of acid and not have to deal with the body load.
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Every time i've done LSD i've done foxy 15 times. Like most drugs its very opinionated. One person loves it the next hates it. As for the numbness, I find it gives the same physical disconnected feeling that come with mush and 2-CB but not like a painkiller like K. However the numbness is felt in my hands mostly which i enjoy. But hey, thats just me.
 
80mgs? I gpt my ass kicked on taking 8mgs and then, liking it, took another 8mgs about four hours later. What was 80mgs like? I can't imagine. It took me almost 2 months to feel normal (baseline) again. But I feel that 5-Meo-Dipt effects people diffrently.
 
I also have a feeling there are batches of 5-MeO-DiPT that are either cut or poorly made. That would account for why some people take up to 100 mg of that one, or 300 mg of AMT.
 
Originally posted by Bytor:
80mgs? I gpt my ass kicked on taking 8mgs and then, liking it, took another 8mgs about four hours later. What was 80mgs like? I can't imagine. It took me almost 2 months to feel normal (baseline) again. But I feel that 5-Meo-Dipt effects people diffrently.
Sheer terror. That is the only word to describe it
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5-Meo-Dipt "Foxy" - Unpredictable!

I started experimenting with Foxy about 2 months ago. I have tripped on it about 8-10 times, so I would say I am fairly experienced. I usually do anywere from 20-30 mgs.
But the past 2 times I did Foxy, something weird happened. It had always taken efect really quick on me...around 30 minutes. I always felt it before everyone else I was tripping with. However, all the sudden it started taking 2-3 hours. The first time that happened, I ended up taking a second dose because I thought I had accidently taken an empty capsule the frist time and when both kicked in I was so rocked. It wasn't a good experience. So the next time this happened I just waited. It took over 2 hours to take effect...while all of my friends were tripping, I was still totally sober.
Has this happend to anyone else?? There dosn't seem to be any logical explanation for it. I am going to a rave this weekend and I plan to trip, but I don't know if I should take the foxy 2 hours before the rave so I'm not waiting around sober or if it will be normal again. I love Foxy, but I hate how unpredictable it is!!
[ 22 October 2002: Message edited by: JessiePoo2 ]
 
This is just conjecture, but if you have done 5-meo-dipt ten times in two months the problem you are having could very well be due to a tolerance buildup. Try giving it a break for a couple of weeks and see if that makes a difference.
 
It could just be breaking down with age, if you're storing it improperly. Is it cold, dark and dry? If not, its probably degrading.
 
I have foxy near 2 years old that still has (seemingly) near full potency.
I agree with the tolerance idea. I've seen a post on here claiming foxy has no tolerance, but I have had a different experience taking it 2 nights in a row, with only about half effect the second night.
Actually I did it back to back weekends not long ago and it came on pretty quick--less than 30 minutes--the first weekend. 5-6 days later, when I did the same amount, it took about an hour to get to that point. But each time I do foxy, it is a little different in so many ways. Of these above 2 occasions, the first time I was rather active the whole time, while the second time I was immobile the 2nd half of the trip, just wanted to lay still and relax. Both good, just different.
And I only do 8mg, so it is easy to be off 1mg or so between doses and I figure that may have some effect. All in all, hard to conclude anything from my experiences, except that I think there is some tolerance issue.
Oh, and it sounds like you are doing too much. I mean, jeez, would you roll every weekend for 8 weeks? Because that is too much. Foxy is different, but there is no reason to think it is safer than e. I'd take a break if I were you.
~psychoblast~
 
I thought of all those reasons too, but I never eat at least 6 hours before taking it. And I do store it in a dark, dry place. I guess it must be a tolerence thing, but then my friends have all done it the same amount as me and I'm the only one this happens to. I know I have been doing it a lot, I took last weekend off...when I used to roll I did that a lot more though!!
 
What out for a change of diet as well, foxy is not as well documented as many other drugs, and a change in diet may decrease absorption rates. Just an assumption.
 
5-MeO-DiPT body load in relation to ethnic background

Alright, when I first started taking 5-MeO-DiPT at medium doses (before I learned how nice IV is) I had an enormous body load...Actually most of the people who took simular doses reacted the same, except for the people who's family history lead back to Ireland...Could there be a connection? I'm nordic in background and most of the other's who suffered along with me are decended from mainland Europe...Has anyone else experianced anything close to this or is it just coninsidence?
 
I'm from Sweden as well. Only taken it once, at a pretty low dose (around 10mg), and I had no mentionable body load. Minor restlessnes, and slight nausea during the comeup, but nothing more.
 
dutch no body load noticed, at dosage ranging from 10 to 2x 10 mgs taken apart. strangely a lot of people complain about the bodyload on internet. me & friends noticed none. strange
 
I'm mostly Irish, English and German.
Sometimes I experience a body load from 5-MeO-DiPT. Sometimes I don't.
I also know someone who has a 100% Irish background and he experienced a significant body load from 10mgs and vomitted.
[ 09 November 2002: Message edited by: Love In Vein ]
 
Well, somewhere around 10% of white people have a deficiency in some enzyme (CY-P450, I'm pretty sure) which affects people's reactions to various drugs. I remember reading this 10% figure in some book dealing with MDMA (PIHKAL perhaps, or Nick Saunders' book) which claimed that this deficiency made MDMA hit these people harder than usual. Apparantly this same enzyme deficiency also makes codeine less effective (they have a hard time metabolizing it into morphine, so it tends to make them nauseous more than narcotized). It also affects how DXM works (not surprising, since DXM is an opioid and this enzyme seems to play a big role in opioid metabolism). Grapefruit juice also acts to inhibit this enzyme.
Its an enzyme that affects MANY drugs, and different levels of this and related enzymes (theres a whole bunch of them, I think they're referred to collectively as cytochrome somethingorother) can have a drastic impact on how many drugs work, from MDMA to opioids to various heart medications. Its certainly possible that this enzyme plays a role either in 5-MeO-DIPT's metabolism or perhaps somewhere in the chain of physiological reactions started by the drug. We know surprisingly little about how psychedelics in general work, and practically nothing about the pharmacodynamics of 5-MeO-DIPT specifically.
As far as I know though, this particular enzyme deficiency isn't limited to Irish or other Keltic peoples... rather, the 10% figure is I believe something common to all white Europeans. Also the deficiency also occurs in non-whites, its just that its more common in people with white ancestry. I don't know if its a dominant or recessive trait and how it would affect people of mixed ancestry. I also don't know how accurate that 10% number is... the book was a book dealing with psychedelics, not genetics or enzymatic metabolism. I'm pretty sure that CY-P450 deficiency is something that occurs more frequently with whites than other groups but I'm not sure if 10% is really an accurate statistic.
Also, I'm not sure on the racial scope... Assuming the 10% figure is correct, does this apply to just white Europeans, or does it apply to Aryan people as a whole? And by this, I am using Aryan in its proper sense and not the misuse of the term by the Nazis... the proper definition of Aryan refers to people descended from a group of people who probably originated somewhere around the Black Sea or the Caucasus Mountains. These people then spread out in several directions in different migratory waves. We can tell where their descendants ended up because they all speak related languages, known collectively as Indo-European. The term Aryan properly includes most white Europeans (Kelts, Germans, Slavs, Latins, Greeks) but also includes many of the people of India, Pakistan, Iran, Afghanistan, and some of the people of the central Asian steppes. Is the enzyme deficiency as common with them? Dunno. Then, there's also white Europeans who pre-dated the Aryans... for example, the Basques, and probably the Etruscans and Minoans... and there are also later arrivals such as the Finns and Estonians, who are actually related to the Asiatic people of Siberia, and more distantly (based on some linguistic evidence) to the Koreans and Japanese. And of course, theres been so much interbreeding over the centuries, there's probably no people anywhere in the world who are genetically like the original Aryans. So, what does all this mean for the genetics of this enzyme deficiency? Hell if I know. Did it originate with the Aryans, and if so, does it occur as often in non-white descendants of the Aryans such as the people of India? Maybe it was something picked up through intermarriage with the pre-Aryan peoples of Europe, and is therefore found in all white Europeans whether of Aryan or pre-Aryan origins... or maybe it was a genetic mutation that occured in the Aryans who migrated to Europe and so doesn't affect Basques, Finns, etc. You'd want to ask a geneticist if you really care about all this crap.
Its also possible that there is some other unknown enzyme or receptor condition or god only knows what that has nothing to do with CY-P450 but plays a role in the pharmacology of 5-MeO-DIPT. If so this could be something found in Irish or other Keltic people. It wouldn't be that unusual. In addition to the CY-P450 deficiency common with whites, many Native Americans are deficient in the enzyme alcohol dehydrogenase which turns alcohols into aldehydes. This makes these people unusually sensitive to alcohol and prone to alcoholism - though of course the genocidal conditions on Indian Reservations probably contributes more to the epidemic of alcoholism than any genetic factors! There are other various odd reactions to drugs and foods that tend to be found mosty in specific races, so it would not be completely unreasonable to speculate that people of Irish descent may have some kind of enzyme or neuroreceptor mutation that makes them react to 5-MeO-DIPT differently.
Personally, I have lots of Irish ancestry, but I've also got ancestors from Germany, Holland, England and Indonesia, so that makes it hard to draw any conclusions from my experiences. I found low doses (4-8mg) to be a mild and somewhat interesting semi-psychedelic sensory enhancer that could perhaps have some recreational value for some people... if future research shows it to be a safe drug, and I came across some, I would consider taking it again at a dose around 5mg just for fun. I found that doses above 8mg did begin to create fully psychedelic experiences, but I found it pretty unusable in that capacity because along with the increase in psychedelic qualities came an increase in body load - mostly, intestinal upset. It seems to have an effect opposite of opioids in that it seems to speed up intestinal movements, causing gas and often severe diarrhea that can last hours. This didnt happen every time I did it, but it happened often enough and was unpleasant enough that I would never take this substance again at a dose above 8mg. In fact, I dont think I'd want to even go past 5mg. My idea of a good psychedelic experience does not involve spending 3 or 4 hours stuck on a toilet shitting out every last bit of liquid in my intestines.
Anyway, until we know everything about how 5-MeO-DIPT works and what chemicals in the body it reacts with, and can do tests on different pure-blooded representatives of different ethnic groups to determine their levels of those chemicals, this is something that we can only speculate about based on anecdotal evidence... and since there's probably not that many people using 5-MeO-DIPT who happen to be pure-blooded Irish, I don't think speculation is even possible. We don't have enough anecdotal evidence about human reaction to this drug in general to even think about how some populations may react differently from the norm. We don't know what the norm is, and probably won't for many many many years - if ever.
 
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