On a different note. I've quite recently started taking/experimenting with Nitrazepam(Mogadon), and on a couple of occasions have awoken all of the sudden. With my whole body being in a paralyzed state with me being completely awake and open eyed.. and its taken ALL of my strength to even move a muscle let alone pull myself out of it
For myself. Basically being very awake, with the pleasure of being able to indulge in the opie high at the same time. Quite a different feeling as I usually never mix oc with anything else.
On a different note. I've quite recently started taking/experimenting with Nitrazepam(Mogadon), and on a couple of occasions have awoken all of the sudden. With my whole body being in a paralyzed state with me being completely awake and open eyed.. and its taken ALL of my strength to even move a muscle let alone pull myself out of it
Completely freaked me out and has NEVER EVER happened with any other benzo. Listing alphra, bromaze, clonaze, diaze, nimetaze, temaze, zopidem which all have been used to/for sleep aside from nimetaze.
Any thoughts? Explanations on this?
When I had nitrazepam it was usually mixed with 2mg alprazolam and 20mg temazepam. But yeah I have pretty bad insomnia and throughout the day i would have taken another 4mg of alprazolam and a bunch of diazepam.
Whoops, yep. Somehow got into the habit of calling it a benzo.. been corrected a few times now but still can't help it!
Well its basically the only prescription med that I've come across so far that works best with my body in order to get a proper nights sleep. Never needed to take more than 10mg too![]()
MIMSPharmacodynamics. Zolpidem belongs to the imidazopyridine group of compounds and is structurally unrelated to other hypnotic agents. Zolpidem selectively binds the omega-1 receptor subtype (also known as the benzodiazepine-1 subtype) which is the alpha unit of the GABA-A receptor complex. Whereas benzodiazepines non-selectively bind all three omega receptor subtypes, zolpidem preferentially binds the omega-1 subtype. The modulation of the chloride anion channel via this receptor leads to the specific sedative effects demonstrated by zolpidem, i.e. the preservation of deep sleep (stages 3 and 4 slow wave sleep).