Ha, 2,5-dimethoxy-4,1-diethylamino-benzene?
I'm having a tricky time naming that one, not a phenethylamine (2 amines), not a diphenethylamine (only 1 phenyl).
I might have a chance to try allylescaline, similar structure but double bond instead of the second amine.
My thoughts are that it would not 'need' to have a name based on a basis skeleton that defines the phenyl and two aminoethyls all at once.
While that is ideal, there is no actual reason for it unless it is part of a series or family in which all members have that skeleton. And with your
2,5-dimethoxy-1,4-diethylaminobenzene don't you actually already have named that skeleton?: 1,4-diethylaminobenzene?
Not sure if there is a common or trivial name for that 1,4 configuration.
I myself have wondered about just the plain 4-amino 2C-X, could not think of a reason why it wouldn't be active but after some searching it appears
I found out it is inactive though. Unfortunately I am not sure where I read it but I do think it was on BL... probably ADD then.
My gut says that the extra ethyl in between doesn't really improve things, though my gut is apparently not proving to be reliable on this subject.
Not the faintest idea about a secondary amine i.e. 2,5-dimethoxy-4-methylaminophenethylamine and 2,5-dimethoxy-4-ethylaminophenethylamine.
Would the amino be a relatively similar substitution like with the thiomescalines?
Nice on the allylescaline, I wonder about it ^. Also interested in 3C-AL and AL-NBOMe i.e. NBOMe-allylescaline.
The latter was available for sampling, if it turns out to be worthwhile we should be seeing a bit more of it.
A shop in my city (at least one, may be multiple) is selling RCs under the counter. I asked about it today using the word '4-fluor' and not flux (name for 4-FAor the word 4-fluoroamphetamine itself... they are capsules containing - "as far as the guy knew" - 125 mg or so, in any case what is considered a dose. He reacted as if nobody actually asks about the dose. Well it turns out it smells of cathinones, not sure what the reason is (I mean what the ID is) but I guess it could mean it is 4-FMC and he might know that. Anyway it is probably a smart thing to get it tested and confront the guy with the results pointing out differences and consequences.
Other samples I have been meaning to get tested are my etaqualone which is either bunk or cut according to the lack of effect it is said to have at appropriate doses, and another is my 2-DPMP. I have a suspicion that one could be cut as well, though I have no direct reasont to believe that. I have little desire to start assaying it myself but my ADHD-y friend who takes ritalin has shown great interest. The small doses and consequences of overdosing are good reasons to confirm ID and purity.
I found a shrink and expect to get medication in about 2 weeks when I get my first appointment, although it might take more than 1 session. There is somewhat of a hurry though - it will be a mood stabilizer, I have discussed as much with my psychologist. Neurontin self-medication (though discussed with my psychologist) has shown to produce quite encouraging results but I did not have a whole lot to test - only a couple of days to a week's worth. I made the stupid mistake of tapering very quickly and taking tramadol last weekend. This resulted in a surge of racing 10.000 thoughts (well not conscious defined thoughts but rather the feeling of such brain activity) that worried me quite a bit. It built up relatively quickly prompting me to take some neurontin and benzo's to avoid something seizure-like. The strange thing is that physically I felt pretty relaxed in the beginning, but at the peak of intensity there was an onset of anxious cramped rigidity. The sensation of my brain going into overdrive subsided when I took the neurontin and benzo's and alerted my roommate and talked about it a bit, at that point the weird sensation shifted towards my general chest area where I felt fluttery.
I realize how moronic that was, though relative to the ingestion of tramadol there was a serious delay to this episode, also I started really carefully with the tramadol. The main thing I was thinking about was resp. depression which I expected to be mild - which it really was up until I felt the necessity to take benzo's. After I calmed down from the overdrive-feelings I worried a little about the resp. depression, though I have definitely had far worse in the past and also I tend to overreact to the whole phenomenon of resp. despression from combinations. I guess the overreacting is a good line of defense, though obviously it can be a bit horrible at the time when it turns into a panic attack from the potential health hazard.
So now I can't be too sure about quick neurontin tapering itself being able to produce such effects or if the tramadol is to blame. Quitting benzo's like 5-6 days before this probably didn't help although I have felt little issue from that thanks to the Neurontin, also this 'attack' seems much too acute to stem from benzo's and their metabolites evacuating my system...I think that it was the tramadol and I should have expected something like this. So for me the option of Neurontin or Lyrica as medication still lies on the table. It makes sense to me that it would work for me since I have had chronic issues with GABAergics (such as multiple long GHB binges, a single xanax binge and a single valium binge quite a while ago and at times using phenibut a bit too much), ever since I have felt the need to self-medicate as something like chronic GABAergic withdrawal. So the effect of Neurontin on the GABA/glutamate equilibrium seems like a logical remedy. Glutamate is associated with higher thinking and one of my symptoms is an overwhelming amount of conscious thinking about things that should remain subconscious. Not sure if I am actually right about Neurontin being a theoretically logical solution but my therapist thinks that my level of understanding about these things in general at least gives me some credit when it comes to self-medication. Although of course officially he cannot say things like that and should emphasize that it is bad practice to self-medicate and that it is not really 'allowed', though the law doesn't seem to speak too strongly about it.
Another realistic medication I see for myself is mirtazepine. I am weary about true mood stabilizers and would like to think that they are too heavy a solution for something that is quite complex. I definitely want to keep away from things like lithium although I guess a big part of it is what I know about the contraindication with LSD. Perhaps depakote could be acceptable I don't know. I think I'd rather try SSRI's than heavy duty mood stabilizers like lithium.
Also I am not sure if I am understimating the long-term side-effects of Neurontin and Lyrica. Of the two Lyrica is more effective (I felt this in own experience) but I also think it has more serious potential side-effects.
Right now I have maybe like a day's worth of Neurontin left and less than a day's worth of Lyrica.
Trying Neurontin has allowed me to quit benzo's although my current situation is begging a little to be contained by something like benzo's. Maybe phenibut until I get proper medication, might be preferable to benzo's - and the risk of getting dependent on them again even if it is quite small doses like the last half year.
Anyway sorry if this is too much for a 'social thread', but I wouldn't be sure of the actual question if I wouldve made a thread about it.
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On another note in response to Pepper's mescaline comment: I seem to have synthetic and extract available to me although a little bit remotely. I have only tried synthetic myself, I can see how it is less complete than an alkaloid extract...
What are your comments? Would you prefer synthetic or extract? Is one of them more psychedelic/entheogenic and the other more purely entactogenic/empathogenic? Is an alkaloid mix usually more visual?
(except the PH papers). Gotta be a lot of seeds one would need though. Haven't tried smoking a opiate though, so it would be well worth it I'm sure, getting that famous rush...
