No, he said something along the lines of "everyone always seems to be under the assumption that psilocybin metabolizes to psilocin in the body, but if there exists hard evidence I do not know of it", that said, he thinks it is very likely considering the sheer abundance of esterases ready to condense the bond.
Indeed, other similar compounds with an analogous story e.g. heroin and morphine serve as a strong indication that both pro-drug and drug are active but they may exhibit different pharmacokinetics and other properties that significantly alter the resulting experience. Ability to cross the BBB is an important one. (Of course there are big differences as well with heroin, its diacetyl instead of phosphoryl, and the major active metabolite has only one of the two acetyls cleaved).
Anyway how much good reason there may be to believe that psilocybin is converted into psilocin, there is less actual evidence that this is what happens as well in vitro using the lemon tek. It is what I believe though. I always used to think and say that a lot more was going on like the general change in acidity would aid absorption in the stomach. But the in vitro conversion theory has more things going for it, so I guess I had to change my mind. What experience I have with ROAs involving citrus juice actually remind me a lot more of 4-HO tryptamines than 4-AcO tryptamines: impossibly short comeup times, outrageous visuals and colors and really in-your-face as opposed to the 4-AcO smoothness and dilation.
This is quite off-topic though.