Hi Guys,
A lot of mental health drugs have a strong affinity for the H1 receptor (along with other desired affinitys like the HT or NE receptors) and this causes sedation and drowsiness. It is also known that the body adapts to the H1 blockage and over time the sedation and drowsiness wears off. So if the body adapts to the H1 blockage whats to say the body doesn't also adapt to the desired receptor affinitys?
My point is if the body adapts and the H1 blockage no longer causes drowsiness doesn't it stand to reason that the body also adapts to other receptor blockages (such as the serotonin re-uptake pump) and the desired anti-depressant effects wear off as well???
A lot of mental health drugs have a strong affinity for the H1 receptor (along with other desired affinitys like the HT or NE receptors) and this causes sedation and drowsiness. It is also known that the body adapts to the H1 blockage and over time the sedation and drowsiness wears off. So if the body adapts to the H1 blockage whats to say the body doesn't also adapt to the desired receptor affinitys?
My point is if the body adapts and the H1 blockage no longer causes drowsiness doesn't it stand to reason that the body also adapts to other receptor blockages (such as the serotonin re-uptake pump) and the desired anti-depressant effects wear off as well???
