Count on it having the same, even if 1P would have activity of it's own besides being an LSD pro-drug (which Nichols says it doesn't and I trust the guy usually, I think any difference in effect would be due to pharmacokinetics), it would still be in addition to being an LSD pro-drug so you would always get the side-effects from LSD formed in your body. At least it's very unlikely 1P negates them, maybe slower kinetics could make things smoother but lack of psychedelic activity of 1P (at 5ht2a) doesn't mean it can't cause vasoconstriction in different ways by being able to bind at other receptors.
Though 5ht2a itself is implicated in vascular effects. So considering 1P is psychedelic and acts on 5ht2a it should also be expected to cause vasoconstriction. There are several aspects to activation of that receptor, LSA for example is much less potent than LSD but causes more vasoconstriction, probably in part due to alpha adrenoceptor activity.
Vasoconstriction isn't necessarily what causes body loads IMO, maybe eventually high physical activity and low blood flow could lead to cramping, but initially it should just produce tingling etc. Muscle tension probably plays a bigger role, things like that can be hard to avoid entirely. Just do what you can to keep anxiety / tension / cramped positioning as much as possible, relax, go do something and keep the blood flowing.. Maybe consider some theanine to ease up a bit.