ChemicallyEnhanced
Bluelighter
Are humans not animals?..........
Are there two of you? You literally did a post very AGAINST animal testing and then sudden are posting very FOR it.
Are humans not animals?..........
But I was quoting you...and then you gave the opposite opinion? I'm confused.
No I didn't, I made a point of FACT.
It was you that brought up some subjects & I just told you I am fully aware of how NASA got benefit from the death of Jews etc & also what Japan got upto during WW".
Where did I say I was ok with these things?..........
Are humans not animals?..........
Are there two of you? You literally did a post very AGAINST animal testing and then sudden are posting very FOR it.
FFS
Where did I say it was ok to test stuff on animals?
Testing ANYTHING on animals is sick, sorry but if things have to be tested what the fuck is wrong with testing it on people that have done horrific crimes like raping kids etc? I have NO issue injecting some sex pervert with unknown drugs, fuck their pain & whatever may happen to them I say.
.....
The animals you seem to give less than a toss about are innocent, they have never harmed you or anyone else YET you seem to think it is ok to do horrific things to them, you better know "God" is just & has a really good memory is all I'm gonna say.
Firstly, I know you, zopiclone, are a meat eater. You are supporting industrial animal rearing for pleasure.The last part of my quote of you can be directly written to you regarding eating animals. chemically enhanced, are you vegan? if not, why is using animals to cure cancer bad but eating them or their products OK? I have been vegetarian for over 20 year. i'm also interested, if you developed type 1 diabetes, would you just die then? or cancer for that matter? if not, why is it OK for you to receive treatment for which animals have suffered, but the people who develop the treatment, out of genuine compassion for humanity, are damned to hell?
Sorry about off topic, but last month I drew some molecular structures of previously unknown compounds that can be synthesized with one reaction from natural precursors, they are listed in these threads:
Molecular docking apps
Yesterday, I tried to find online ligand-protein docking simulators to see it they could tell the difference between known dopamine reuptake inhibitors (methylphenidate, altropane, amfonelic acid...) and non-DRIs (ibuprofen, cyclizine, citalopram and some random organic compounds). I downloaded...www.bluelight.org
I Like to Draw Pictures of Random Molecules
^ It must have been a bit of work drawing all of those and making predictions... I tried to put a non-amine version of methylphenidate, with a sulfur atom in place of nitrogen, in the Swiss target prediction app as an input (the oxygen version is known to be a likely DAT ligand). Didn't get a...www.bluelight.org
The Swiss Institute of Bioinformatics target prediction application says they all are possible dopamine reuptake inhibitors. Do you know how to perform a real molecular docking simulation to get a better prediction of dopamine transporter binding affinity?
Not only is this off topic, it's off subforum... :D
foreigner, please answer my questions. where is your data? seriously i have the skills to analyse it and can publish, the world deserves not to suffer unnecessarily! do you diagnose breast cancer personally? what biomarkers do you use? how do you differentiate between types? how would you assess whether a cancer has metastasised? what difference does the 'underlying syndrome' make to treatment? when is patient cured? under what circumstances would you advise someone to go to a doctor- i.e. how do you differentiate between someone for whom '5/10 might work' and someone who needs 10/10? if 'maybe 5/10 might work sometimes' is an acceptable standard for alternative medicine, why are 15% of modern medical treatments having no proven efficacy an issue for you?
nobody is claiming that evidence based medicine doesn't have shortcomings, we just claim that it is based on evidence, so nice take down of a straw man.
Experimental findings support the evidence of a persistent leucopenia triggered by brain death (BD). This study aimed to investigate leucocyte behaviour in bone marrow and blood after BD in rats. BD was induced using intracranial balloon catheter inflation. Sham-operated (SH) rats were trepanned only. Thereafter bone marrow cells were harvested every six hours from the femoral cavity and used for total and differential counts. They were analysed further by flow cytometry to characterize lymphocyte subsets, granulocyte adhesion molecules expression and apoptosis/necrosis [annexin V/propidium iodide (PI) protocol]. BD rats exhibited a reduction in bone marrow cells due to a reduction in lymphocytes and segmented cells. Bone marrow lymphocyte subsets were similar in BD and SH rats (CD3, P = 0.1; CD4, P = 0.4; CD3/CD4, P = 0.4; CD5, P = 0.4, CD3/CD5, P = 0.2; CD8, P = 0.8 . Expression of L-selectin and beta2 -integrins on granulocytes did not differ (CD11a, P = 0.9; CD11b/c, P = 0.7; CD62L, P = 0.1). There were no differences in the percentage of apoptosis and necrosis (Annexin V, P = 0.73; PI, P = 0.21; Annexin V/PI, P = 0.29). In conclusion, data presented suggest that the downregulation of the bone marrow is triggered by brain death itself, and it is not related to changes in lymphocyte subsets, granulocyte adhesion molecules expression or apoptosis and necrosis.