I just don't see how 200g of fibre daily is even possible. I can reach 50g at most if i snack between meals and supplement but it's difficult to get more than 40g while keeping up with other stuff like protein and fats. the thing is that all fibrous foods also contain lots of carbs and sugar and in modern urban society most of the population gets nowhere near the amount of exercise that hunter-gatherer types would have been getting daily. For them, getting veggies would mean pulling roots from the ground and meat would mean stalking prey for hours a day. For us, getting meat and veggies is simply a matter of sitting in the car and driving to the grocery store where it's all conveniently been collected for purchase. So even if we were able to take in 200g of fibre a day, the question is how much physical activity would we have to add to burn through the energy surplus that comes with it.
Yeah 200g would be pretty hard to attain but the lower end of the scale should be easily attainable.
As for the consequences of consuming that much energy - this is only conjecture but... If what some of the research is pointing towards turns out to be correct - that the diversity of bacteria is a key determiner for a whole raft of diseases like diabetes and even obesity - then maybe, just maybe, the fixation with calorie intake might be less important than we've previously believed. Maybe the problem with food isn't so much about how much you eat but rather the quality of what you eat. These are early days and I doubt anyone would advocate that people just throw out our previous understandings about nutrition but just maybe. One researcher was making the claim that this understanding about our relationship to our microbiome could add years, even decades, to the life-span of an individual.
IF that turns out to be true then the benefits of eating large amounts of fibre may well outweigh the risks. Maybe. Time will tell.
I may have missed it somewhere but are we talking about soluble fibre, insoluble, or both together?
Don't think that was specified in the Catalyst episodes I was watching - I'm working from an assumption that it includes both.
And, something I saw yesterday, new research on the link between microbiota and allergies:
https://theconversation.com/changes-to-bugs-in-the-gut-could-prevent-food-allergies-30814
=====================================================
Changing the bacteria in the gut could treat and prevent life-threatening allergies, according to research published in the Proceedings of the National Academy of Sciences (PNAS) journal today.
“These findings are a game changer for understanding how allergies develop,” said Dr Simon Keely, senior lecturer in immunology and microbiology at the University of Newcastle. “The number of hospital admissions due to reactions to food have increased dramatically over the past 10 to 20 years.”
The study authors examined how changes in the trillions of bacteria that normally populate the gastrointestinal tract influence allergic responses to food. They started by inducing peanut sensitivity in mice by giving them antibiotics soon after birth.
Antibiotics disturb the harmony between the bacteria and immune system of the gut, creating an allergic sensitisation to peanuts. They prime the immune system to mistakenly recognise peanuts as a threat.
The researchers then introduced a peanut solution directly into the mice’s gastrointestinal tract through a feeding tube. When the rodents were exposed to this solution, they became sensitive to peanuts.
A group of bacteria called Clostridia was then introduced into the gut of the mice and the researchers found it got rid of the peanut sensitivity. They believe the findings will inform the development of similar approaches for allergy prevention in humans.
Although the research was done in mice, Professor Katie Allen, paediatric gastroenterologist and allergist at Murdoch Childrens Research Institute, said the results were profound for allergy research because they showed proof of concept of the importance of gut bacteria and their interactions with the developing immune system.
Dr Keely said the study demonstrated previously unrecognised pathways by which the immune system interacts with the bacteria in the gut.
“When you disrupt that interaction, you become more susceptible to developing allergy,” he said.
The study underlines how bacteria in the gut and the immune system are intrinsically linked, he said, adding “they regulate each other. If you disturb one, you disturb the other.”
But both Allen and Keely highlighted some of the limitations of the research.
“We can’t say that gut bacteria that are shown to be protective for mice will also be protective for humans but it is an interesting concept nonetheless,” said Professor Allen.
Dr Keely said humans have a much more diverse diet than mice and tended to be exposed to a very broad environment of bacteria, unlike mice, which tend to live in relatively clean cages.
The good news is that this research suggests the potential for modifying gut bacteria in humans as a way of prevention of allergic disease – in particular food sensitisation, Professor Allen added.
Professor Allen said researchers have been looking at many ways of preventing allergy including kick starting the immune system in newborn babies, studying migration changes on human allergies and studies in probiotics.
This current study provides further supportive evidence for the role of gut bacteria in allergy development, she said. The most important thing to take away from it is that there may be group of bugs that will be helpful in protecting against allergic sensitization and therefore also food allergy.