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1-cyclopentyl-2-aminopropane dose range? Effects?

SkyblueMolly

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1-Cyclopentyl-2-AminoPropane. Let's call it CAP. It affects norepinephrine, epinephrine(adrenaline), and dopamine. Dimethylamylamine has plenty of effects on norepinephrine and epinephrine, but barely any effects on dopamine at all. So DMAA(dimethylamylamine) is like an adrenaline-like stimulant only. What would the dose range of 1-Cyclopentyl-2-AminoPropane be? 20mg to 60mg? Perhaps CAP would rival caffeine. I hope it does rival caffeine. Caffeine isn't a stimulant. 1-Cyclopentyl-2-AminoPropane is a stimulant, and it has some effects on dopamine. 1-Cyclopentyl-2-AminoPropane is norcyclopentamine, or a shortened cyclopentamine.
1-Cyclopentyl-2-AminoPropane.
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Caffeine isn't a stimulant.

Uh, reality check: it is?

Also are you talking about the aminopentane (prolintane) or the aminopropane? Cause you drew the aminopropane. Which would probably be very much like an intermediate between propylhexidrine and DMAA: tweaky and not that good.
 
Uh, reality check: it is?

Also are you talking about the aminopentane (prolintane) or the aminopropane? Cause you drew the aminopropane. Which would probably be very much like an intermediate between propylhexidrine and DMAA: tweaky and not that good.

I'm talking about the aminopropane. So the effects of 1-Cyclopentyl-2-AminoPentane would be between somewhere between a small dose of adderall and DMAA? Sort of like ritalin where you become over-neat, ocd, and anal retentive? That sounds like the guy from limitless when he's wired and ocd, not super high.
Caffeine fools adenosine receptors and reduces the perception of fatigue. That's not the same as stimulants that affect dopamine, norepinephrine, and maybe epinephrine causing actual stimulation (either physical, mental, or both). Caffeine isn't a stimulant. It only very mildly mimics stimulants. It's a fatigue perception reducer.
Fluorenol is an eugeroic. There are many classes of compounds that may mimic stimulants, but aren't stimulants.
 
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I'm talking about the aminopropane. So the effects of 1-Cyclopentyl-2-AminoPentane would be between somewhere between a small dose of adderall and DMAA? Sort of like ritalin where you become over-neat, ocd, and anal retentive? That sounds like the guy from limitless when he's wired and ocd, not super high.
Caffeine fools adenosine receptors and reduces the perception of fatigue. That's not the same as stimulants that affect dopamine, norepinephrine, and maybe epinephrine causing actual stimulation (either physical, mental, or both). Caffeine isn't a stimulant. It only very mildly mimics stimulants. It's a fatigue perception reducer.
Fluorenol is an eugeroic. There are many classes of compounds that may mimic stimulants, but aren't stimulants.

Doesn't caffeine also bring synapses closer together causing them to fire faster or something?


Ok, I looked it up and it seems that caffeine activates Ryanodine receptor 3 which causes a release of Ca2+ in the brain, which would certainly make it a stimulant, even by your definition.
 
Doesn't caffeine also bring synapses closer together causing them to fire faster or something?


Ok, I looked it up and it seems that caffeine activates Ryanodine receptor 3 which causes a release of Ca2+ in the brain, which would certainly make it a stimulant, even by your definition.

Maybe. But could 1-Cyclopentyl-2-AminoPentane and cyclopentamine be stronger than caffeine? It would be fun to have a coffee or the future. Sort of like ritalin jr or something. Maybe 1-Cyclopentyl-2-AminoPentane is that ritalin jr. %)
 
I highly doubt it, strongly adrenergic compounds like DMAA and propylhexidrine are discouraged from regular use as essentially everything but decongestants due to their massive effects on heart rate & bp...
 
Caffeine isn't a stimulant.
I'll try to put this as nicely as I can, but you are completely and utterly wrong. Caffeine isn't as potent as some other stimulants such as cocaine and amphetamine, but is IS a stimulant. The classification of a drug as a stimulant is not based on whether it has dopaminergic effects or produces euphoria, it is based on whether it increases the activity of the central nervous system.
 
Caffeine fools adenosine receptors and reduces the perception of fatigue. That's not the same as stimulants that affect dopamine

You may find it interesting to do some research on interactions between adenosine receptors and dopamine release/receptors, and you'd probably re-assess this rather simplistic view if you did.
 
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