N&PD Moderators: Skorpio
Metabolism of 3FMC and 2FMC, any possibilitys in slowing it?
Due to amphetamine shortage i'm using them as a replace for a few days but due to the short half life i have to take a lot for them being effective, i'm wondering by what metabolic route those things get metabolised and what meds can inhibit those enzyme's, would it be simular to amphetamine?
Thank you.
sekio
Bluelight Crew
Probably a few CYP enzymes that para-hydroxylate the ring, and MAO-A/B of course.
Playing with either is a bad plan, maybe try 4-fa or redosing? Continual usage of any ammphetamine isn't a good idea though.
I need amps for my ADHD, reuptake inhibitors and wellbutrin lack therapeutic effiacy in me.
sekio
Bluelight Crew
That's strange and to me sounds like justifying your DOC
I do know that reuptake inhibitors can have sometimes paradoxical effects, but I always figured they would also appear with releasing agents too. Remember, amph is a NDRI too!
I would be cautious about going into 3-FA territory with any sort of regularity due to possible 5-ht2b mediated cardiotoxicity ala fenfluramine/norfenfluramine...
Well amp is my DOC so i allways take to much so i have to thread my ADHD with this crap for the rest of the month, i know the risks involved in rc's tough and cycling differend one's minimises the risk, 5HT2B issues also take weeks to show up.
nuke
Bluelighter
That's strange and to me sounds like justifying your DOC
I do know that reuptake inhibitors can have sometimes paradoxical effects, but I always figured they would also appear with releasing agents too. Remember, amph is a NDRI too!
I would be cautious about going into 3-FA territory with any sort of regularity due to possible 5-ht2b mediated cardiotoxicity ala fenfluramine/norfenfluramine...
Trifluoromethyl groups generally do not act like fluoro groups on a benzene ring, 2c-f versus 2c-tfm being prime examples (former does not even appear to be an agonist or have affinity for the serotonin receptor)
