First off, let's talk terminology for a second. In the context of this thread and for that matter, the vast majority of our communications on this site:
Opioid - Referring to substances/pharmaceuticals that are defined as Opioid Agonists. There are different Opioid receptors, though they are vastly different in the effects they produced when agonized. For our purposes, we are discussing substances/pharmaceuticals that primarily produce their effects by agonizing the Mu Opioid Receptor or, MOR for short. I'm not going to get into the other receptors types because I don't feel they're incredibly relevant here.
For the uninitiated, the simple explanation is that the MOR is the receptor responsible for all of the effects that you think and feel when you think of the word Opioid; the desired effects; the effects that cause people to become addicted; the effects that mitigate pain and misery.
The vast majority of "Opioids" discussed here on Bluelight are MOR agonists and less commonly, we will discuss substances that work upon other receptors, but to a much lesser extent. For instance, Pentazocine (Talwin) was once a very popular drug of abuse that was both a Mu and a Kappa agonist. There are no drugs like this, at this time, that I'm aware of that are similar in this way.
I have read a lot about Kratom. I've talked to at least 100 serious Kratom addicts in my time... probably more. I'm getting fuckin old here guys.
The active constituent of Kratom is a family of alkaloids known as Mitragynine(s). The two primary alkaloids that produce Mu receptor agonism are the eponymous Mitragynine and 7-Hydroxymitragynine or 7-OH colloquially. Mitragynine is more plentiful by volume, but 7-OH is a more potent overall chemical, so both play a significant role in the totality of effects produced by the substance.
After reading and talking with a lot of people over the course of years, I'm very tempted to say that Kratom and therefore Mitragynine are little more than your garden variety Opioid(s). I do believe that as a plant, there is possibly more at work than just said agonism, though I feel that this auxilliary stuff would be of modest relevance to the person using them.
I've yet to see anything that convinces me that Kratom is anything but a somewhat exotic (to us) Opioid that has yet to be made illegal, thereby filling a vacuum in the market for folks who don't wish to use Fentanyl or are respected citizens who couldn't afford to be caught with hard drugs. We all want to believe that our Opioid habit is something other than what it is. I've been there.
To answer OP's question directly:
Kratom isn't going to "increase your tolerance" gram for gram more than any other Opioid, you feel me? We are doing apples to apples accounting here. We are just dealing with apples of differing potencies.
Don't look at it as, "this is how addicted I am to Tramadol, but this is how addicted I am to Fentanyl; it's all just about your total number of Morphine Milligram Equivalents or MME. This is the system they will use in hospital and medical clinics to standardize a person's total Opioid intake.
The only problem is, we have not even an educated guess at determining the strength of your Kratom and even more so for the Fentanyl you're using.
With that being said, Kratom didn't increase your tolerance per se, you just took more Opioids and that's what raised your tolerance. Which Opioid it was has little importance relative to your total number of MME's. I hope that helps. Let me know if you have any questions guys.
