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Marijuana Mental Health

^ it's so bad it's entertaining IMO. No we don't have Fox news. Thankfully the BBC isn't that bad but I don't have a TV so I don't know about other channels. I know the Daily Mail is just as bad.

From what I've heard MSNBC is actually not that bad in comparison and go a long way to compensating for the Fox news propaganda machine. I may have got it mixed up but I think I saw a couple of cannabis reports done by them that were unbiased and showed medical cannabis in a positive light. It's just a shame Fox news are so popular when they resort to yellow journalism all the time. I've even heard of them photoshopping photos of people who's opinions they don't like to make them look ugly and goofy. Real journalists don't do that sort of bullshit.

Yeah the BBC news is on public television here, so is Democracy Now! Occasionally a report from Al Jazeera will be on, but rarely. So we have maybe an hour or two a couple times a week of decent news on all of the tv channels combined, it's ridiculous.

MSNBC is probably the least bad of the tv news channels, but they tend to have opinion pieces as much as they have actual news. They've aired a few documentaries about marijuana but I haven't seen any of them.

Americans seem to think that political commentators, sensationalist crap about social trends and celebrity news amounts to news coverage of important events, it's crazy how much time people fill with the garbage on tv, the public here is amazingly uninformed.
 
That's a very good question. From what I gather, we know that psychosis rates in countries and cultures where cannabis use is virtually non-existent is not lower than in countries where cannabis use is much more common. If cannabis use was a significant risk factor for the mental illness we would see higher rates of the illness in countries where cannabis use is far more common but we don't. You could say you can't compared different ethnic group to each other but as I understand it in the same groups of people rates of the mental illness don't increase when cannabis becomes more or less popular over time.
How would you rate it's effect on something like depression or anxiety disorder? It's interesting, I have a friend who becomes borderline suicidal when he attempts to stop smoking pot. He was like this for years before he ever started to smoke; pot became his remedy of sorts and he's stuck with it. He now has a girlfriend of 5 years, his own place, a job... so he isn't complaining really. I on the other hand become far more depressed in the times I dwell deeper into stoner-hood. This isn't such a bad thing as it allows me to increase my interest, insight and focus towards to my studies which is also my passion (which is also my username :D). Now I've seen a few studies in the past that didn't find significant correlations between marijuana use and most mental illness and the studies you've posted are consistent to that. However I do wonder if that's because the positive cases mix with the negative cases to in a sense balance things out statistically. In respect to schizophrenia this likely does not apply however with other sorts of mental illness I do raise the eyebrow a bit.


The research was done by professor Glyn Lewis with the university of Cardiff, who states no causal relationship between cannabis use and mental illness has been established. Another leading expert, Dr Stanley Zammit of the university of Bristol has also gone on record refuting the claim there is evidence of causality.


Here's the evidence you asked for:

Hickman et al 2009, a study of all published evidence, shows that the risk of a correlation between lifetime cannabis use and a single diagnosis of psychosis is at worst 0.013% and probably less than 0.003%. HERE is the link to the journal publication.



Also going back to the first point I made, you should take a look at this study funded by the Advisory Council on the Misuse of Drugs (ACMD) who commissioned scientists at Keele University to examine the evidence. They concluded that the rate and prevalence of schizophrenia was either “stable or declining”. HERE is the link.
Thanks for the detailed reply. Looks like you've done your research! I'd love to add more to the discussion in this regard but I must admit to my own ignorance and adhere to the standard of not talking out of my ass. =D



^ if that's aimed at me please carefully read what I said again. Strains, genetically have not been bred to be significantly stronger than they've been historically in the 60s and 70s. It's just that better growing conditions are a bit more common. It's an important distinction to make. The achievements made in thousands of years of cultivation of high quality landraces has not been surpassed by modern breeding techniques, despite their marked improvement over the basic selection and rouging farmers used to employ. I would also add that the marginal increases in potency have been grossly overstated and exaggerated again and again in the media. The modest improvements in potency that are mainly due to better growing conditions only account for small increases in potency in general as you will see if you look at the data. Far from the 25x or more increase in potency banded about by the media.

By the way OG Kush is just a fad that most growers and 'connoiseurs' (I hate using that term because it sounds pretentious) are sick to death of hearing about all the time. It's not even that good of a strain and is certainly not the most potent. And yes, a lot of the best cannabis of the 60s (particularly import cannabis from places like SE Asia) was definitely stronger than 'OG Kush'. Take well grown and prepared Thai Stick for example and compare it to OG Kush and then tell is more potent and better quality.
]Heh, I figured OG Kush maybe wasn't too special, just the first name that came to mind really. I think you might be misunderstanding what I'm saying here though. My point was that growers in the 60s/70s possibly weren't cultivating marijuana that was as consistently potent as it is today. Now that doesn't mean today's weed is stronger, rather that stronger strains (that always have existed) are simply more common today. A comparison would be how more people today live until 100 or 110 than they did in the past, but no one is living until 150. The internet has made it real easy for growers of all sorts --from the dimwitted to the highly intelligent-- to grow marijuana that is quite potent. I have to wonder if the lack of this resource hindered their ability to consistently provide high quality strains to the customer base. I also wonder if the seed distribution at that time consisted of seeds of lower potency strains. None of this may be true of course, I only raise the question because I've found the vast, vast majority of older people I have seshed with or discussed marijuana with feel this way about today's strains.
 
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How would you rate it's effect on something like depression or anxiety disorder? It's interesting, I have a friend who becomes borderline suicidal when he attempts to stop smoking pot. He was like this for years before he ever started to smoke; pot became his remedy of sorts and he's stuck with it. He now has a girlfriend of 5 years, his own place, a job... so he isn't complaining really. I on the other hand become far more depressed in the times I dwell deeper into stoner-hood. This isn't such a bad thing as it allows me to increase my interest, insight and focus towards to my studies which is also my passion (which is also my username :D). Now I've seen a few studies in the past that didn't find significant correlations between marijuana use and most mental illness and the studies you've posted are consistent to that. However I do wonder if that's because the positive cases mix with the negative cases to in a sense balance things out statistically. In respect to schizophrenia this likely does not apply however with other sorts of mental illness I do raise the eyebrow a bit.

Use of cannabis for anxiety is a very individual thing. For people who have smoked their whole life it's more likely to be of benefit that for a novice. Some people find it useful but others may not have such luck. But that's the same with conventional antidepressants. There are pros and cons to both really. Cannabis won't be beneficial for everyone that tries it and it's unrealistic to expect it to. I do think that unless you're using it for medical reasons there's a time and a place to smoke and for many smoking all day every day may very well make it difficult to function. Again, it depends on the person, because I know plenty of successful and well balanced people that smoke 24/7.

Heh, I figured OG Kush maybe wasn't too special, just the first name that came to mind really. I think you might be misunderstanding what I'm saying here though. My point was that growers in the 60s/70s possibly weren't cultivating marijuana that was as consistently potent as it is today. Now that doesn't mean today's weed is stronger, rather that stronger strains (that always have existed) are simply more common today. A comparison would be how more people today live until 100 or 110 than they did in the past, but no one is living until 150. The internet has made it real easy for growers of all sorts --from the dimwitted to the highly intelligent-- to grow marijuana that is quite potent. I have to wonder if the lack of this resource hindered their ability to consistently provide high quality strains to the customer base. I also wonder if the seed distribution at that time consisted of seeds of lower potency strains. None of this may be true of course, I only raise the question because I've found the vast, vast majority of older people I have seshed with or discussed marijuana with feel this way about today's strains.

I'm not surprised OG Kush was the first strain to come to your mind. Why? Because at the moment, apart from perhaps cheese, it's the most hyped strain in existence. You hear kush kush kush all the time and so people naturally wonder what all the fuss is about. It's not a bad strain but it certainly doesn't merit all the attention. It's saddening that seedbanks like DNA genetics who used to be highly regarded, started from having good ethics and being very wary of feminized seed and autoflowering gentics to eventually jumping on the bandwagon and starting to churn out kush this kush that at a rate far too fast for them to be actually breeding the strains. So for people that care about cannabis it can be rather disheartening.

Globalization has resulted in more strains than ever becoming more available to the masses. However this has not really improved the quality of the genetics. In the 60s people were mainly smoking landrace strains and in the 70s and 80s the pioneers started breeding the legendary strains into what we have today with strains such as northern lights, haze hybrids etc. Today almost all the strains, with the exception of ones produced by a minority of breeders like mandala seeds, ace seeds, cannabiogen etc. are just the old strains rehashed into new ones. I could show you a scan of one the catalogues from before the time of the internet from the 80s with many of the strains from which most hybrids of today are bred. Really not much has been achieved at all with breeders just using the same genetic stock that almost all modern strains are bred from. What I mean is there is very little original breeding work actually being done now. A well bred hybrid strain of two landrace strains is not necessarily going to be any less potent than a multi-way/poly hybrid so unless you have more people actually going to afghanistan, india, pakistan and taking landrace genetics you won't really be achieving anything that's not already been achieved. Like I was saying, potency is only one aspect of a strain and there's so much more to good breeding than producing high potent weed.

Really the main achievement between now and in the 60s and 70s is that short and stocky indica strains have been crossed with the old unwieldy sativa strains to produce hybrids that take the best attributes of each. This allows you to take the best traits of the sativa strains in a plant small enough to be grown indoors and with a shorter more realistic flowering time. This in and of itself revolutionized growing and allowed people to be able to grow indoors for the first time. The problem is capitalism, not modern growing. It has given too much of an incentive for people to take shortcuts and produce inferior cannabis just to make a quick buck. People should be breeding for the love of cannabis, not just to make a profit. Thankfully there are still good breeders around, it's just a case of educating people so they can make better more informed decisions about what to grow.
 
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When I first started smoking weed, I got too high and had a HORRIBLE trip. Now that I think of it I may have been psychotic. I completely lost all contact with reality, I died, and was rolling around the flames of hell. It was crazy. I stopped for a while and slowly eased back into it, then turned into a major pothead. It was great, until I started getting anxious after a few months. Every time I'd smoke I'd start to get not only anxious, but horribly depressed. It was really weird because I wasn't experiencing that kind of depression sober. I was however going through a lot of stress at the time and I think the weed just put me in touch with how I really felt about things, and I didn't like it. Nowadays I'm doing fine but I still don't enjoy smoking weed, it just doesn't make me feel good. I do miss enjoying it though :( ... maybe one day :)
 
^It's not for everyone, perhaps its not for you. If there is a lot of difficult stuff going on in your life its not necessarily going to make it easier for you to sort those issues out. If you are experiencing anxiety every time you smoke for whatever reason its probably a good idea to stay away from it. Maybe in the future when your life is at a different stage you will enjoy it again maybe not...

I was trying to find a reference about cannabis and mental health specifically with regard to people self medicating with cannabis the other day. I couldnt find the study I was looking for but I did find an overview put out by the Australian government. The view they put forward was that people suffering from depression may try and use cannabis to self medicate but they felt there was insufficient evidence to support the idea that cannabis is used for self medication in schizophrenic or psychotic individuals. The summary for shizophrenia is below and there is more information in the article itself.

Key points: Cannabis and schizophrenia
There are many factors that contribute to the emergence of psychotic disorders such as schizophrenia.
There is good evidence that cannabis makes some symptoms of psychosis transiently worse.
The evidence that the association of cannabis use with emerging psychosis is due to selfmedication
is not conclusive.
Cannabis use increases the risk of schizophrenia among those with other risk factors.
While it is likely that cannabis use leads to cases of schizophrenia that would not have occurred
otherwise, the number of such cases is probably small.

Its good to see they are remaining objective about the causality issue even though it isnt the view expressed in the media at the moment. I'm not sure how they came to the conclusion in the last dot point but I should probably read it again.

NCPIC Cannabis and Mental Health
 
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The conclusions relating to other mental health issues. Note it was a literature review and not a new study.

Key Points: Cannabis and depression, anxiety and other mental illnesses
Recent evidence suggests that early, frequent and continued cannabis use increases the risk of
experiencing depression in adulthood, although the evidence for early self-medication is stronger
for depression.
There is a lack of evidence suggesting that cannabis plays a causal role in the development of
anxiety disorders or bipolar disorder.
Cannabis appears to adversely influence the symptoms of mood and anxiety disorders in the long
term even though it may alleviate them during intoxication.
 
Wow AE, thanks for all the knowledge you have dropped about the potency of cannabis over the years on the previous page.

I was under the impression that cannabis now-a-days was stronger, so thanks for clarifying this out mate!
 
I had a single experience about a year ago after smoking a fair bit of cannabis over the period of 3 days.
I was having intense derealization and then all of a sudden there were multiple voices in my head arguing and i felt like a spectator.
In the state i was in i was convinced i somehow triggered latent schizophrenia but after laying off it for a week or so i felt back to normal and haven't experienced anything close to that again.

I'm not sure if this is valuable but this is the only experience i have on the topic.
 
I once smoked a bowl and hadn't slept for over a day. I started to hallucinate in the middle of the road. Somewhat like the onset of salvia. really really fucking freaked me out. Never happened again, dunno might have been a panic attack.
 
I had a single experience about a year ago after smoking a fair bit of cannabis over the period of 3 days.
I was having intense derealization and then all of a sudden there were multiple voices in my head arguing and i felt like a spectator.
In the state i was in i was convinced i somehow triggered latent schizophrenia but after laying off it for a week or so i felt back to normal and haven't experienced anything close to that again.

I'm not sure if this is valuable but this is the only experience i have on the topic.

What do you mean by "over the period of 3 days"? Did you smoke constantly for 3 days without sleeping? Otherwise I can't see why such an experience happened to you...

Even when I smoke all day for a week, I have never experienced voices in my head or anything. I can see about the derealization as I sometimes feel spaced out when waking up after a big night of toking.
 
for real i am diagnosed with panic disorder and take paxil for it....i have no ill effects from marijuana but if i drink? im useless for 3 days because i am anxious and paranoid as fuck....some people just arent built mentally to handle some good ass tree....you cant force it if it isnt working..love mary and she will love you back..
 
I've been smoking reefer for years and it has eradicated the depression that was dominating my pre-smoking life. I am a more social, happy, carefree and totally at peace with myself.
Cannabis is a good thing. It is also responsible for helping me quit cigarettes, it's a healthy and fun alternative. Peace & Pot
 
I once watched a documentary on marijuana and psychosis, I forgot exactly what one so unfortunately I can't really cite it or anything like that but it was extremely informative and unbaised. Basically what it said is that there are a lot of different cannabinoids in weed. d-9-THC is obviously one of them and it is pretty much the only ones that produces the "stoned" feeling we all know and love. THC may also be partially responsible for aiding the development of any possible psychotic illnesses that can be caused from weed smoking, while another cannabinoid, CBD (cannabidol or something like that) actually has a lot of potential for being an anti-psychotic medicine. The problem is that most CBD is a product of THC degradation, and today weed is grown to have the highest and most reselent THC content posible (well at least the good weed is). Thus the THC/CBD ratio in most good and even medicinal strains of weed is way to high to have much of an anti-psychotic benefit. In fact, therotically the effects of heavy and continued use of these strains should lean more towards the development of most likely small and less severe forms of psychosis. The good news is that this seems to be extremely rare and only happens to those who have a predisposition for psychotic disorders already.
This is all a result of the different receptors these cannabinoids target, THC is mainly a CB1 agonist, while CBD is a CB2 agonist. CB1 recepetors are located in parts of our brain that are very important for our cognitive thoughts processes such as critical/abstract thinking and memory formation: the hippocampus and cerebral cortex. Agonising these receptors is what gives us that euphoric head high (sSativa's tend to have higher THC/CBD ratio than indicas), but also tends to impair perception and memory. While CB2 agonist seem to play a much larger role aiding our immune system and pain management. They also seem to have a neuroprotective role over nerve cells, possibly by stimulating the production or activating existing glial cells. If this is true it would truly be a breakthrough in understanding potential postives of marijuana. Glial cells are integral to our mental health, they clean out dead nerve cells from our brain and also protect neurons in areas such as the hippocampus and cerebral cortex from any damage. The problem is that the complete effects of CB1 and CB2 anagonist/antagonist are still relatively unknown, and this is not because of any inabilties of the modern day scientific community, it's just that there are few labs who are willing to carry out such research, and they receive even less funding. What does seem to be known is that raising the CBD/THC ratio, instead of the other way around, seems to be more beneficial to our mental health, but would produce a less enjoyable high.
 
Cannabis doesn't really cause schizophrenia. There's no real evidence that proves it causes it anyway.

"Cannabis use has been associated with increased risk of psychiatric disorders. A 15-year follow-up of 50 465 Swedish male conscripts reported that those who had tried cannabis by age 18 years were 2·4 times more likely to be diagnosed with schizophrenia than those who had not.81 Risk increased with the frequency of cannabis use and remained significant after statistical adjustment for a few confounding variables. Those who had used cannabis ten or more times by 18 years of age were 2·3 times more likely to be diagnosed with schizophrenia than those who had not. Zammit and colleagues82 reported a 27-year follow-up of the same cohort. These investigators also showed a dose–response relation between frequency of cannabis use in individuals aged 18 years and risk of schizophrenia during the follow-up, and this association persisted after controlling for the effects of other confounding factors. They estimated that 13% of schizophrenia cases could be averted if cannabis use was prevented. These findings have been supported by longitudinal studies in the Netherlands,83 Germany,84 and New Zealand,85,86 all of which indicated that the association persisted after adjustment for confounders." from Lancet, Volume 374, Issue 9698, pp 1383-1391

That's from an article in the Lancet, which is a tier 1 peer reviewed publication. If cannabis didn't increase risk of schizophrenia as you say, I highly doubt the leading experts in the field who refereed the article would allow that into print.
 
^Association doesn't necessarily equal causation though. I agree that it is a risk factor but there are a heap of other things that have similar or greater levels of risk:

People with a family history of schizophrenia- up to 46 times greater risk of developing the illness
Being born in an urban environment- 2.4 times greater risk
Being born in winter- 1.1 times greater risk

according to:
Mortensen, P.B., et al., Effects of family history and place and season of birth on the
risk of schizophrenia. New England Journal of Medicine, 1999. 340: p. 603–608.
 
"Cannabis use has been associated with increased risk of psychiatric disorders. A 15-year follow-up of 50 465 Swedish male conscripts reported that those who had tried cannabis by age 18 years were 2·4 times more likely to be diagnosed with schizophrenia than those who had not.81 Risk increased with the frequency of cannabis use and remained significant after statistical adjustment for a few confounding variables. Those who had used cannabis ten or more times by 18 years of age were 2·3 times more likely to be diagnosed with schizophrenia than those who had not. Zammit and colleagues82 reported a 27-year follow-up of the same cohort. These investigators also showed a dose–response relation between frequency of cannabis use in individuals aged 18 years and risk of schizophrenia during the follow-up, and this association persisted after controlling for the effects of other confounding factors. They estimated that 13% of schizophrenia cases could be averted if cannabis use was prevented. These findings have been supported by longitudinal studies in the Netherlands,83 Germany,84 and New Zealand,85,86 all of which indicated that the association persisted after adjustment for confounders." from Lancet, Volume 374, Issue 9698, pp 1383-1391

That's from an article in the Lancet, which is a tier 1 peer reviewed publication. If cannabis didn't increase risk of schizophrenia as you say, I highly doubt the leading experts in the field who refereed the article would allow that into print.

Roger&Me you can have all the studies showing an association but as justsayno said, is association the same as causation? I think not.

You can do the research to verify this for yourself, but if you believe cannabis use causes schizophrenia how can you explain this?

Assessing the impact of cannabis use on trends in diagnosed schizophrenia in the United Kingdom from 1996 to 2005.
Frisher M, Crome I, Martino O, Croft P.

Department of Medicines Management, Keele University, Staffordshire, ST5 5BG, United Kingdom.

A recent systematic review concluded that cannabis use increases risk of psychotic outcomes independently of confounding and transient intoxication effects. Furthermore, a model of the association between cannabis use and schizophrenia indicated that the incidence and prevalence of schizophrenia would increase from 1990 onwards. The model is based on three factors: a) increased relative risk of psychotic outcomes for frequent cannabis users compared to those who have never used cannabis between 1.8 and 3.1, B) a substantial rise in UK cannabis use from the mid-1970s and c) elevated risk of 20 years from first use of cannabis. This paper investigates whether this has occurred in the UK by examining trends in the annual prevalence and incidence of schizophrenia and psychoses, as measured by diagnosed cases from 1996 to 2005. Retrospective analysis of the General Practice Research Database (GPRD) was conducted for 183 practices in England, Wales, Scotland and Northern Ireland. The study cohort comprised almost 600,000 patients each year, representing approximately 2.3% of the UK population aged 16 to 44. Between 1996 and 2005 the incidence and prevalence of schizophrenia and psychoses were either stable or declining. Explanations other than a genuine stability or decline were considered, but appeared less plausible. In conclusion, this study did not find any evidence of increasing schizophrenia or psychoses in the general population from 1996 to 2005.
http://www.ncbi.nlm....pubmed/19560900

In my opinion that sort of data is far more telling than research like the study you cited, Roger&Me. Schizophrenia levels started to level off and then declined from 1996 to 2005 roughly the timeframe indoor grown "skunk" started taking over. Kind of hard to explain that one away.

You have to realize very high THC/no CBD ganja has been around in Tropical Asia for millenia so this recent refer madness stuff is complete BS.
 
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Latest Research On Cannabis and Schizophrenia Runs Contrary to Mainstream Media Hype


http://blog.norml.org/2010/05/26/la...renia-runs-contrary-to-mainstream-media-hype/

The mainstream media loves to spill ink hyping the allegation that cannabis causes mental illness, particularly schizophrenia. In fact, it was in March when international media outlets declared that cannabis use ‘doubled’ one’s risk of developing the disease. Yet when research appears in scientific journals rebuking just this sort of ‘reefer madness,’ it generally goes unreported.

Such is the case with a pair of just-published studies slated to appear in the journal Schizophrenia Research. The first study, conducted by a team of researchers at various New York state hospitals, the Yale University School of Medicine, and the National Institutes of Mental Health assessed whether there exists a causal association between cannabis use and the age of onset of psychosis in patients hospitalized for the first time for an episode of schizophrenia.

Despite previous media claims to the contrary, researchers concluded:
“Although the onset of cannabis use disorder preceded the onset of illness in most patients, our findings suggest that age at onset of psychosis was not associated with cannabis use disorders. Previous studies implicating cannabis use disorders in schizophrenia may need to more comprehensively assess the relationship between cannabis use disorders and schizophrenia, and take into account the additional variables that we found associated with cannabis use disorders.”

A separate study slated for publication in the same journal assessed the cognitive skills of schizophrenic patients with a history of cannabis use compared to non-users. Authors reported that patients with a history of cannabis use “demonstrated significantly better performance on measures of processing speed, verbal fluency, and verbal learning and memory” compared to abstainers. cannabis use was also associated with better overall GAF (Global Assessment of Functioning) scores compared to those of non-users.

Authors concluded: “The results of the present analysis suggest that (cannabis use) in patients with SZ (schizophrenia) is associated with better performance on measures of processing speed and verbal skills. These data are consistent with prior reports indicating that SZ patients with a history of CUD (cannabis use disorders) have less severe cognitive deficits than SZ patients without comorbid CUD. … The present findings also suggest that CUD in patients with SZ may not differentially affect the severity of illness as measured by clinical symptomatology.”

Both study’s findings are in line with previous (though virtually unreported) research indicating that cannabis is unlikely to instigate incidences of schizophrenia in the general population, that cannabis use among patients with the disease is associated with higher cognitive function, and that at least some schizophrenics find subjective relief from symptoms of the illness by using pot. Nonetheless, odds are the nobody from the mainstream media will be champing at the bit to report on them.



study:

Are cannabis use disorders associated with an earlier age at onset of psychosis? A study in first episode schizophrenia.

Sevy S, Robinson DG, Napolitano B, Patel RC, Gunduz-Bruce H, Miller R, McCormack J, Lorell BS, Kane J.

The Zucker Hillside Hospital of the North Shore Long Island Jewish Health System, Psychiatry Research, USA; Albert Einstein College of Medicine, Department of Psychiatry, USA.

Also:



http://medicalmarijuana.procon.org/view.answers.php?questionID=000220


Does marijuana use cause lasting schizophrenia, psychosis, or other mental disorders?

Colin Blakemore, PhD, ScD, Chair of the Department of Physiology at the University of Oxford, wrote in a Dec. 27, 2002 email to ProCon.org:
"It is conceivable that excessive use of cannabis sometimes contributes to acute schizophrenic episodes. But it is difficult to believe that cannabis is a strong risk factor for this disorder, because there is no evidence that the incidence of schizophrenia has risen dramatically over the past 50 years, in parallel with the huge increase in cannabis use.

Young schizophrenic patients are often heavy cigarette smokers too, but no-one would suggest that tobacco causes schizophrenia."

Paul Armentano, Senior Policy Analyst at the National Organization for the Reform of Marijuana Laws (NORML), stated in an Aug. 2, 2007 press release titled "NORML Responds to New Rash of Pot and Mental Health Claims":
"Despite the enormous popularity of cannabis in the 1960s and 1970s in numerous Western cultures, rates of psychotic disorders haven't increased since then in any of these societies. Individuals suffering from mental illness such as schizophrenia tend to use all intoxicants - particularly alcohol and tobacco - at greater rates than the general population. Not surprisingly, many of these individuals also use cannabis."

Lynn Zimmer, PhD, late Professor Emeritus at the Queens College, noted in her 1997 book Marijuana Myths, Marijuana Facts:
"Given that the incidence of schizophrenia declined substantially in Western societies in the 1970s, at the same time cannabis use was rising, it seems highly unlikely that marijuana causes schizophrenia in otherwise healthy people....
Cannabis psychosis is self-limiting, disappearing in a few days with or without medical treatment. Toxic psychosis probably occurs more commonly in individuals with preexisting psychiatric disorders....
Marijuana temporarily alters mood, thought, emotions, and perception, sometimes quite dramatically. None of marijuana's effects cause people to behave in any particular manner.
In the midst of a toxic psychosis, people may become agitated and frightened. In response to acute panic, people may become withdrawn and inactive.
Neither of these states eliminates the social and moral restraints that guide human behavior."
 
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Debunking the Myth of a Link Between Marijuana and Mental Illness


Despite media claims that marijuana can cause psychosis or schizophrenia, there's no science to back it up?page=1

http://www.alternet.org/drugs/15177...f_a_link_between_marijuana_and_mental_illness
July 25, 2011 |


Prohibitionists have a long history of exploiting tragedy to further their own drug war agenda. Case in point: Members of Congress in the 1980s seized upon the overdose of basketball star Len Bias to enact sweeping legislative changes establishing mandatory minimum sentencing in drug crimes, random workplace drug testing for public employees, and the creation of the Drug Czar’s office.

So it was hardly surprising to see anti-drug zealots return to this tried-and-true playbook in the days immediately following the shooting this past January of Arizona Congresswoman Gabrielle Giffords and 18 civilians. Only hours after alleged shooter Jared Lee Loughner was taken into custody, pundits on the political far right opined that the 22-year-old former pot smoker had been driven mad by weed.

For example, less than 24-hours after the shooting former George W. Bush speech-writer David Frum posed the question, “Did pot trigger the Giffords shooting?” to which the longtime conservative commentator answered, “Increasingly, experts seem to be saying ‘yes.’”

Frum’s accusation appeared to gain a modicum of respectability one month later when the mainstream media highlighted a report in The Archives of General Psychiatry that purported to have linked marijuana use with psychosis.

“It is increasingly clear that marijuana is a cause of schizophrenia,” the study’s lead researcher, Matthew Large of Prince of Wales Hospital in New South Wales, Australia, told the online publication Web MD in February. (In a separate interview he said he was “horrified” by suggestions that the plant should be legalized and regulated.) Large further insisted, “[T]he schizophrenia caused by cannabis starts earlier than schizophrenia with other causes.”

Or not.

In truth, the supposed new ‘study’ contained no new findings at all. Rather, Large and his team simply reviewed previously published research – much of it decades old.

“There are no new data. I want to emphasize that. This is a meta-analysis, which means it (reviews) the studies that were already out there,” SUNY Albany psychology professor Mitch Earleywine, author of the book Understanding Marijuana: A New Look at the Scientific Evidence, explained on the NORML Audio Stash days after the report’s release. “What you’re not hearing in the media is that in fact, this (reported association) is probably early-onset folks self-medicating (with cannabis).”

There are several published reports to back up Earleywine’s suspicion. For instance, a 2005 study of 1,500 subjects that appeared in the scientific journal Addiction reported that the development of “psychotic symptoms in those who had never used cannabis before the onset of (such) symptoms … predicted future cannabis use.”

Other studies reinforcing Earleywine’s ‘self-medication’ theory include a 2008 study published in the International Journal of Mental Health Nursing which found that schizophrenics typically report using cannabis to reduce anxiety and “improve their mental state.” Marijuana use has also been associated with clinically objective benefits in some schizophrenics. Recently, a 2010 report in the journalSchizophrenia Research found that schizophrenic patients with a history of cannabis use demonstrate higher levels of cognitive performance compared to nonusers. Researchers in that study concluded, “The results of the present analysis suggest that (cannabis use) in patients with SZ (schizophrenia) is associated with better performance on measures of processing speed and verbal skills. These data are consistent with prior reports indicating that SZ patients with a history of (cannabis use) have less severe cognitive deficits than SZ patients without comorbid (cannabis use).”

A 2011 meta-analysis published online by the journal Schizophrenia Research also affirmed that schizophrenics with a history of cannabis use demonstrate “superior neurocognitive performance” compared to non-users. Investigators at the University of Toronto, Institute of Medical Sciences reviewed eight separate studies assessing the impact of marijuana consumption on cognition, executive function, learning, and working memory in schizophrenic subjects. Researchers determined that the results of each of the performance measurements suggested “superior cognitive functioning in cannabis-using patients as compared to non-using patients.”

Investigators stopped short of attributing subjects' cannabis use to the improved outcomes, hypothesizing instead that patients with superior cognitive skills may be more likely to acquire cannabis than subjects with lesser abilities. “t is difficult to determine whether it is cannabis itself that triggers alterations in neuropsychological functioning or if drug-using patients represent a subset of the schizophrenia population who exhibit better neurocognitive performance,” they wrote. Nevertheless, they concluded that it would be reasonable to assume that “cannabis likely has modest … effects on neurocognitive function in schizophrenia.”

Other clinical literature also casts doubt on Large’s claim that marijuana use accelerates mental illness. In a study published last year, a team that included researchers affiliated with the Albert Einstein College of Medicine, Yale University, and the National Institute of Mental Health assessed whether lifetime pot use was associated with an earlier age of onset of symptoms in schizophrenic patients. They concluded, "Although cannabis use precedes the onset of illness in most patients, there was no significant association between onset of illness and (cannabis use) that was not accounted for by demographic and clinical variables.”

The researchers also criticized the findings of previously published studies that purported to have uncovered a ‘pot trigger’ for mental illness. “Previous studies implicating cannabis use disorders in schizophrenia may need to more comprehensively assess the relationship between cannabis use disorders and schizophrenia.”

Unlike Earleywine, however, the researchers in this study were not convinced that a large percentage of schizophrenic patients are ‘self-medicating’ with pot. “We … found that about half of our subjects discontinued the use of cannabis when their psychotic symptoms worsened,” said Dr. Serge Sevy of the Zucker Hillside Hospital, who led the study. “(But) unfortunately, our study did not include questions about (patients’) reasons for using or discontinuing cannabis. I cannot provide the percentage of patients who discontinued cannabis use because of a worsening of psychosis … (versus those who) became too impaired to obtain cannabis.”

As for Large’s most serious claim, that juvenile marijuana use “is a cause of schizophrenia,” most experts on the subject – and most scientific reviews of the matter – disagree.

For example, authors of a 2009 study published in Schizophrenia Research said definitively that increased cannabis use by the public has not been followed by a proportional rise in diagnoses of schizophrenia or psychosis. Investigators at the Keele University Medical School in Britain compared trends in marijuana use and incidences of schizophrenia in the United Kingdom from 1996 to 2005. Researchers reported that the "incidence and prevalence of schizophrenia and psychoses were either stable or declining" during this period, even the use of cannabis among the general population was rising.

"[T]he expected rise in diagnoses of schizophrenia and psychoses did not occur over a 10 year period," they concluded. "This study does not therefore support the specific causal link between cannabis use and incidence of psychotic disorders. ... This concurs with other reports indicating that increases in population cannabis use have not been followed by increases in psychotic incidence."

In April, scientists at the University Hospital of Child and Adolescent Psychiatry in Bern, Switzerland also published clinical trial data indicating that cannabis use plays virtually no role in the early onset of psychosis in younger patients. Researchers assessed the differences in the age of onset of psychosis among 625 patients admitted to the Early Psychosis Prevention and Intervention Centre in Melbourne, Australia. They reported, “Only cannabis use … starting at age 14 was associated with an earlier age at onset at a small effect size.” Overall, the age at onset for patients with first-episode psychosis “was not significantly different” among patients with a history of cannabis use versus non-users.

These results don’t particularly surprise Dr. Julie Holland, clinical assistant professor of psychiatry at the NYU School of Medicine and the editor of The Pot Book: A Complete Guide to Cannabis – It’s Role in Medicine, Politics, Science, and Culture. “The bottom line here is no one knows exactly what causes schizophrenia, and scientists have been looking for decades,” she says. “The best explanation is a ‘stress diathesis’ model, where people have a genetic tendency toward schizophrenic illness, and then something triggers its appearance. But unless you have the genes, you won't get the illness. Cannabis won't change one’s genetic predisposition.”

Holland does caution that people with a predisposition toward schizophrenia “tend to have a stronger, more psychotic-like reaction to cannabis, but that is different from the idea that pot actually gives you schizophrenia, which is completely untrue.” As for the severity of these potential psychotic symptoms, Holland states, “When the drug wears off, so do its effects. There is no lasting psychosis from pot.”

Retired associate professor of psychiatry at Harvard Medical School, Dr. Lester Grinspoon, has studied both cannabis and schizophrenia for over 40 years, authoring the books Schizophrenia: Psychopharmacology and Psychotherapy and Marihuana The Forbidden Medicine. His expert opinion largely echoes the views of Drs. Holland and Earleywine.

“Schizophrenia is largely a genetically determined disorder. However, not all people who have this genetic makeup develop the disorder. So, we have been searching for other variables that must be involved but so far with little success,” he explains. “Recently we have seen the publication of a number of papers that point the finger toward cannabis. Because my work in schizophrenia was first undertaken in the 1960s when marijuana was first observed to be increasingly widely used by young people, I was always careful to include the possibility that the patient had previously smoked marijuana in my history taking. I can't tell you how many patients this involved, but it was certainly measured in the hundreds and not once did I find that it could be considered causal. Its use, on a few occasions, seemed like an attempt to alter an insufferable internal environment, much as people with schizophrenia often do with alcohol and tobacco.”

Ultimately, however, even if such a causal connection between cannabis use and mental illness were to one day be established, this finding alone would do little to support pot prohibition. In fact, the policy implications of such a determination should be just the opposite.

Health risks connected with drug use – when scientifically documented – should not be seen as legitimate reasons for criminal prohibition, but instead, as reasons for legal regulation. After all, there are numerous adverse health consequences associated with alcohol, and it’s precisely because of these effects that the product is legally regulated and its use is restricted to specific consumers and settings. Similarly, if there are legitimate mental health risks associated with use of cannabis by certain individuals then a regulated system would best identify and educate these people so that they may refrain from its use. Placed in this context, drug warriors’ fear-mongering surrounding the issue of marijuana and mental health does little to advance the cause of tightening prohibition, and provides ample ammunition to wage for its repeal.
 
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Here's a reality check:

Drinking too much coffee can make you hear voices, warn scientists


http://www.dailymail.co.uk/health/a...fee-bring-hallucinations-warn-scientists.html

If you keep hearing things that aren’t there, you’re probably drinking too much coffee.

Just five cups a day could be enough to make your ears play tricks on you, according to researchers.

n an experiment, volunteers who had consumed ‘high levels’ of caffeine thought they were listening to Bing Crosby singing White Christmas even though the song was not being played.

The researchers described caffeine as ‘the most commonly used psychoactive drug’.

They said the study showed that the health risks of too much coffee need to be addressed.

In the tests, 92 volunteers were asked to listen to a constant fuzzy sound known as white noise.

Professor Simon Crowe, of La Trobe University in Melbourne, said: ‘We also told them that within the white noise there may be parts of the song White Christmas and if you hear it, press a button.

‘We didn’t include White Christmas in the white noise but found that more people who were very stressed and had high levels of caffeine thought they heard the song.

‘The combination of caffeine and stress affect the likelihood of an individual experiencing a psychosis-like symptom.’

The team from the university’s School of Psychological Sciences found that five cups of coffee a day was enough to trigger this.

Professor Crowe added: ‘Caution needs to be exercised with the use of this overtly “safe” drug.’
 
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Here's a reality check:

Drinking too much coffee can make you hear voices, warn scientists


http://www.dailymail.co.uk/health/a...fee-bring-hallucinations-warn-scientists.html

If you keep hearing things that aren’t there, you’re probably drinking too much coffee.

Just five cups a day could be enough to make your ears play tricks on you, according to researchers.


There is a BIG fucking difference between thinking you heard something and actually hearing someone/somethingthing tell you to do things.

I love MJ don't get me wrong, but it seriously fucks with some people.
 
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