Every "antipsychotic"/neuroleptic will render dopamine agonists or dopamine reuptake inhibitors useless. This types of drugs are, among otherr things "dopamine antagonists". This is cause for psychosis working hypothesis is that underlining cause is "dopamine imbalance". But unlike "serotonin imbalance hypothesis" used for explaining depression, in "dopamine imbalance hypothesis" too high levels of dopamine (not too low of serotonin like in the case of depression) are causing the disorder. So if you are taking "antipsychotic" in doses that are usually prescribed for some psychotic disorder then you can forget amphetamines properly working - cause your dopamine receptors are blocked (saturated by molecule that does not activate them).
Best analogy would be naltrexone and opioids. If you're on 50mg of oral naltrexone and you take it in the morning - taking opioids (hydrocodone, ocycodone, morphine...) later that day will not "work". They will be "blocked" by opioid antagonist that you took in the morning. If you want to feel pleasurable effects that bring euphoria like analgesia, sedation, anxiolisys, warmth...only thing you can do is wait until naltrexone leaves your opioid receptors. And that will depend on how much of opioid antagonist you have circulating your body. In this scenario where one takes 50mg oral naltrexone in the morning by the evening of the next day opioids should be "working" again.
For dopamine receptor antagonists (antipsychotics) it will depend on the type and it's "duration of action" (which is closely related to half-life of a particular agent) but overall story is very similar. Untill it leaves your body in a meaningful percentage you will not feel the effects of Aderall or other dopaminergic drugs. We are all different but after 5-6 half lifes percentage of the drug in your bloodstream falls below 1% and has negligible effects.
I could and probably should made this post shorter but I hope that you don't mind my rant and that I have answered your question.
Take care!