I think CBD blocks CB1 and increases CB2. THC alone may be a better option to increase CB1.
CBD does not totally block the effects of THC, it simply moderates them, that is to say, it does not block the receptors 100% at a normal dose. And most strains of weed sold for recreational use are mostly THC anyway. Even taking pure CBD does not produce typical effects of CB
1 blockade - it does not induce depression, anhedonia, or reduce appetite the same way rimonabant would.
" McPartland and Guy proposed that Cannabis and its many constituents work, in part, by “kick-starting” the [cannabinoid] system. "
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anandamide is just to activate CB1 right? it has no anxiety allieving effect on its own?
CB
1 activation, in moderation, can help stop anxiety, no matter how the receptor is activated, either anandamide or THC. The thing is, anandamide is rapidly broken down in the stomach and liver so you can't eat it as a drug.
. For FAAH inhibition Pterostilbene + black pepper and Maca combo.
I don't think that's likely to be very strong at inhibiting FAAH. I know resveratrol doesn't really penetrate BBB and produce a druglike action, I don't see why pterostilbene would. As fat as I know there are no effective natural FAAH inhibitors.
How the hell would I make my body create More/new cb1 receptors?
I posted several studies in your other thread: among other things, sugar, regular moderate to strong exercise, acute administration of opioids (not chronic/long-term use), acute administration of cannabis/THC (not long term usage), chronic stress, some antidepressant drugs, probiotics (yogurt and the like), polyunsaturated fatty acids. Some of which are healthier than others. And there are even more things that increase natural production of anadamide but not expression of CB
1 too...
You certainly don't have anything to lose, THC has never caused any deaths. If you don't want to do that then eat more sugars, fats, and get some strenuous exercise... for 20 mins or more.Time yourself. See how you feel after...