Yes, of cause, I see what you mean. But how is it metabolized? What parts come off in the body? If you remove the amide part it starts looking a bit like aminorex.
Yeah, I think it is metabolized
the same way as piracetam, which ends up 2-(2-oxopyrrolidin-1-yl) acetic acid.
You end up with this funky-ass molecule:
(2-oxo-4-phenylpyrrolidin-1-yl)acetic acid
...and what does
Pubchem have to say about it?
High throughput screening has shown it
doesn't kill a few scary types of bacteria.
Thanks, Obama! 
Yet it seems like every supplier under the sun offers it, and has no idea what it does. I'm guessing none of them actually have it on hand, and no one ever orders it.
Anyway, just thought it was a funny coincedence, did't think it would have any real bearing on it's effects. Or it would propably have been known already.
Definitely don't jump to conclusions like that with the racetams! In my opinion they as interesting a class as phenethylamines and tryptamines (albeit mostly in different ways), but horribly under-researched. You look through pubchem and see the occasional assay here and there, and almost nothing on SARs or finding new derivatives. It's almost like some kind of conspiracy to keep smart drugs under wraps
I'm only half kidding
There are even only a few papers on phenylpiracetam, and afaik none exploring potential derivatives despite showing pretty amazing effects in clinical trials.
I'd love to see what happens when you start substituting on the phenyl ring.
Really the only two racetams that have been heavily researched are Piracetam and Keppra, and we still barely have any idea how the fuck they work. Heck, Keppra is still the only known drug to target SV2A. And there are some really interesting ones out there, like Coluracetam... another first in class drug as a high-affinity choline uptake enhancer now in clinical trials that I'm also a big fan of.
Phenylpiracetam is actually supposed to be one of the racetams that really work. Stimulation from just one dose is supposed to be on par with a cup of coffe, just different obvisously.
I'm about to find out.
Yeah, it's pretty stimulating. I'd compare it more to dexedrine than coffee, it won't get you jittery. Most people claiming racetams don't work are expecting to notice some change in perception, and phenylpiracetam+oxiracetam+coluracetam are really the only ones that do that to any real degree, the former two as stimulants and the latter as a very, very weak psychedelic (just color enhancement, I wonder if that's why the named it). The stimulation on phenylpiracetam is pretty unlike anything else I've tried in the stimulant world, its very smooth and slightly euphoric, gives a big increase in heat/cold+exercise tolerance (why its banned in competition) despite being weakly adrenergic at most, increases cognitive tempo and reasoning abilities
a lot, and has modest improvements in both short and long term memory retention. I'd compare it to dexedrine but it's less euphoric, less focusing and far more pro-cognitive. I'd compare it to 2-FMA, but it's more euphoric and pro-cognitive, but less adrenergic and far less focusing. I'd compare it to Armodafinil, but it's far more effective at enhancing physical performance and less wake-promoting+focusing.
Yeah, it's basically just... phenylpiracetam.
The only downside is tolerance builds pretty fast, I have to cycle 3 days on and 5 off to return to baseline. The wikipedia article on it is a good summery of all the known info on it.
Racetams, Y U NO RESEARCHED BETTER!?
Edit:
In Soviet Russia, Carphedon research you! 
