iksaxophone
Bluelighter
- Joined
- Jul 6, 2015
- Messages
- 201
Studies suggest that NSAIDs such as ibuprofen, acetaminophen, and indomethacin interact with the cannabinoid receptors.(http://www.ncbi.nlm.nih.gov/pmc/articles/PMC3056416/)
If this is so, why is it that:
a) They only potentiate the effects of more classic CB agonists, rather than exhibiting psychoactive THC-like effects, or the (admittedly anecdotal) perceptable effects of cannabidiol? That is, why do they only seem to be active on inflammation?
b) How is it that they are so much more toxic than other chemicals that effect the CB system (herniation for example, when cannabinoids are fairly good for the digestive tract)?
If this is so, why is it that:
a) They only potentiate the effects of more classic CB agonists, rather than exhibiting psychoactive THC-like effects, or the (admittedly anecdotal) perceptable effects of cannabidiol? That is, why do they only seem to be active on inflammation?
b) How is it that they are so much more toxic than other chemicals that effect the CB system (herniation for example, when cannabinoids are fairly good for the digestive tract)?