I don't know, I guess it wouldn't necessarily, it just strikes me as a warning sign. With drugs that are less erratic it makes it seem as though there is a more clear cut process going on:
x mg of Drug A administered -> Reaction A in brain -> Effect A felt
Whereas, iprocin seems to have different effects felt for the same dosage, I'm just wondering what gives rise to this change and what other consequences this gives rise to.
I know my flowchart is a drastic simplification of what really goes on, I'll admit I have next to no concrete knowledge of the processes synthetics/RCs actually undergo, this is just my brain wondering...
Perhaps it is just that iprocin is a fragile trip that can be completely aborted by small changes in set and setting, but there seems to be a trend that it is the 2nd or 3rd trips that people are having troubles with, not the first one. If it was just set and setting you would think there would be more reports of iprocin being inactive on the first try, given that people would be more likely to know a suitable set and setting after the first trip.
x mg of Drug A administered -> Reaction A in brain -> Effect A felt
Whereas, iprocin seems to have different effects felt for the same dosage, I'm just wondering what gives rise to this change and what other consequences this gives rise to.
I know my flowchart is a drastic simplification of what really goes on, I'll admit I have next to no concrete knowledge of the processes synthetics/RCs actually undergo, this is just my brain wondering...
Perhaps it is just that iprocin is a fragile trip that can be completely aborted by small changes in set and setting, but there seems to be a trend that it is the 2nd or 3rd trips that people are having troubles with, not the first one. If it was just set and setting you would think there would be more reports of iprocin being inactive on the first try, given that people would be more likely to know a suitable set and setting after the first trip.