ponch
Bluelighter
Have a few benzos lying around for such occasions Si, I rarely touch any kind of stimulant without them these days. They really are a life saver.
Specialspack... relapse is speaking the truth when he says its not available to anyone and he tells us why in his trip report.
Whatever you think of him, at least he's proving a Harm Reduction message by recommending that this chemical isn't one that everyone should be able to get. With a name like Special Spack, maybe you ought to reevaluate some things about yourself!
Edit:
I understand you were probably saying taking 3,4-dichloro-methylphenidate in combination with other drugs is a bad idea,
and I agree, but (correct me if im wrong but) the addition of valium at the tail end of this trip report would only serve to reduce the which was already very high, so this is quite sensible HR along with a mention that it shouldnt be available to everyone because of the high potency. Ketamine may or may not have contributed to an overstimulation but I'm pretty sure these work on different neurochemical systems NMDA and Dopamine (Ketamine & 34DCMP Respectively)
I don't think there's a need to be so brash with relapse.. this is a relatively unknown chemical, and he offered some insight.
I'm aware of the possible longevity of this stuff so I'll leave the rest in the bag, but as of, say 11am this morning, I have 5mg in the bloodstream. I shall update a little later, as I'd like to engage in some light musical work
Bloody hell 8) - I was merely pointing out the irony inherent in someone castigating irresponsible drug users on the one hand, whilst on the other merrily sampling a barely-studied potent stimulant on a cocktail of subutex, pregabalin, ketamine and valium.
With all due respect, pharmacologically, you don't know what you're talking about. Just because the drugs have different primary CNS targets, doesn't mean that they won't have potential interactions. Armchair pharmacology is never a good idea when exploring new substances...
I'm not denying that this report offers some limited insight, but to call it harm reduction is a bit of a joke.
better than MDPV
Bloody hell 8) - I was merely pointing out the irony inherent in someone castigating irresponsible drug users on the one hand, whilst on the other merrily sampling a barely-studied potent stimulant on a cocktail of subutex, pregabalin, ketamine and valium.
With all due respect, pharmacologically, you don't know what you're talking about. Just because the drugs have different primary CNS targets, doesn't mean that they won't have potential interactions. Armchair pharmacology is never a good idea when exploring new substances...
I'm not denying that this report offers some limited insight, but to call it harm reduction is a bit of a joke.
An unusual aspect of 4-CA metabolism is the reported conversion of the drug to oxygen-containing products. A phenolic product was identified by Parli and Schmidt (1975) as being 3-chloro-4-hydroxyphenylisopropylamine. This would seem to invoke the NIH shift as an explanation for the migration of the chloro atom. Even more remarkable is the report (Sherman and Gal, 1976) of the isolation of 3,4-dimethoxyphenylisopropylamine following the intraventricular injection of 4-CA. This represents the formation in vivo of a weak but accepted pressor and psychotomimetic. When the mechanism of its formation is understood, a chemical link may be at hand tying the simpler phenylisopropylamine stimulants to the methoxylated psychotomimetics. There were no reports from the clinical studies of 4-CA that suggested any psychotomimetic action.