I dont know of any studies done solely on the prolactinogenic activity of etizolam. I would guess that etizolam raises PRL acutely and should return to normal shortly after extended periods of abuse.
Evolutionary biologists believe that the prolactin was a safety mechanism in males which developed in the hunter gatherer days and there-before. Men would have sex, orgasm, then would temporarily lose their libido (as we do today). This would allow them to re-focus their attention on obtaining food rather than to keep on fucking all day. Less people would have survived in those days if men had not lost their interest in sex to go on to meet survival needs.
Dopamine and prolactin are related. Dopamine increases prolactin, and during sex dopamine is high. Orgasm causes a dopamine spike, and thereafter, theres is a relative absence of dopamine as prolactin spikes.
Hyperprolactinemia can cause sever loss of libido, and therefore, impotence is also a downstream effect. To keep prolactin suppressed, we can use dopamine agonists. In the US, we have bromocriptine, cabergoline, pramipexole, and ropinirole. Bromocriptine and cabergoline can cause heart valve deterioration, albeit at higher doses and for prolonged period of time. Still would you be comfortable taking it to treat a non-serious issue?
Pramipexole (Mirapex in the US) and ropinirole are two non-ergot dopamine agonists that do not cause the heart valve problem. I have much experience with both. Pramipexole is roughtly four times as potent as ropinirole. Ropinirole has less severe side effects in my opinion, and is cheaper. The patent just expired a few years ago and it is available generically. They have very similar dopamine subtype D2 binding affinities and should both do a good job lowering prolactin.