ShaggyFin
Bluelighter
- Joined
- Jan 15, 2013
- Messages
- 751
Here are various research Papers that prove that Cannabinoids cure almost all forms of Cancer. It can also be used for various other Medical purposes (Pain, Seizures, etc)
General Cancer
https://www.researchgate.net/public...noids_as_Cell_Death_and_Anticancer_Modulators
https://www.researchgate.net/publication/24427518_Cannabinoids_in_the_treatment_of_cancer
https://www.researchgate.net/public...eripheral_CB1CB2_Cannabinoid_Receptor_Ligands
Lung Cancer:
https://www.researchgate.net/public...astasis_via_intercellular_adhesion_molecule-1
Gliomas
https://www.researchgate.net/publication/5892365_Cannabinoids_and_Gliomas
Melanoma
https://www.researchgate.net/public...CB1_Receptor_as_Drug_Target_in_Human_Melanoma
Breast Cancer
https://www.researchgate.net/public..._tumor_growth_and_metastasis_of_breast_cancer
FAAH Inhibitors
https://www.researchgate.net/public...nities_from_regulating_endocannabinoid_levels
https://www.researchgate.net/publication/236250821_The_endocannabinoid_signaling_system_in_cancer
Cannabinoid Reuptake Inhibitors (CRIs)/Endo-Cannabinoid Reuptake Inhibitors (ECRIs) & Fatty Acid Amide Hydrolayse Inhibitors (FAAH Inhibitors)
Your brain has natural Cannabinoids that it produces to regulate many things, these are called EndoCannabinoids.
Here is a link to the Wiki about EndoCannabinoids function in the brain:
https://en.wikipedia.org/wiki/Endocannabinoid_system
But there is now a Molecule out there that is not a cannabinoid called LY-2183240. What this Molecule is said to do is this:
Acts both as a potent inhibitor of the reuptake of the endocannabinoid anandamide, and as an inhibitor of fatty acid amide hydrolase (FAAH), the primary enzyme responsible for degrading anandamide. This leads to markedly elevated anandamide levels in the brain, and LY-2183240 has been shown to produce both analgesic and anxiolytic effects in animal models.
Here is what Anandamine is (a natural Cannabinoid that your brain produces: http://en.wikipedia.org/wiki/Anandamide
In 1992, in Raphael Mechoulam's lab, the first such compound was identified as arachidonoyl ethanolamine and named anandamide, a name derived from the Sanskrit word for bliss and -amide. Anandamide is derived from the essential fatty acid arachidonic acid. It has a pharmacology similar to THC, although its chemical structure is different. Anandamide binds to the central (CB1) and, to a lesser extent, peripheral (CB2) cannabinoid receptors, where it acts as a partial agonist. Anandamide is about as potent as THC at the CB1 receptor.[41] Anandamide is found in nearly all tissues in a wide range of animals.[42] Anandamide has also been found in plants, including small amounts in chocolate.
AM 404 (Formed in the body when Tylenol is taken)
UCM 707
AM1172
VDM11
VDM13
OMDM1
OMDM2
LY 2183240
LY 2318912
O-2093
Inhibitors of fatty acid amide hydrolase (FAAH)
Carbamate FAAH inhibitors
OL-135
URB 597
URB 532
Bisarylimidazole derivative
Natural
Phytocannabinoids
Delta(9)-tetrahydrocannabinol (THC)
Delta(-THC
Cannabidiol
Cannabigerol
Cannabichromene
Cannabicyclol
Cannabielsoin
Cannabitriol
Miscellaneous
Endogenous
Endocannabinoids
N-arachidonoylethanolamide (anandamide; CB1-CB2 partial agonist)
2-arachidonoylglycerol (CB1 complete agonist, CB2 agonist)
2-arachidonoylglyceryl ether (noladin ether; CB1 complete agonist)
O-arachinoyl-ethanolamine (virodhamine; CB2 partial agonist, CB1 antagonist, inverse agonist)
N-arachidonyl-dopamine (CB1 agonist)
Docosatetraenoylethanolamide?
Oleamide?
N-Oleoyl dopamine?
Dihomo-linolenoylethanolamide?
Endocannabinoid-related compounds
Fatty acid derivatives
Oleamide
Oleoylethanolamide
2-oleoylglycerol
Stearoylethanolamide
Palmitoylethanolamide
2-palmitoylglycerol
Linoleoylethanolamide
2-linoleoylglycerol
Archidonoyl-aminoacid
Synthetic
Cannabinoid receptor agonists
Classical cannabinoids
Delta (-THC (CB1-CB2 agonist)
HU-210 (CB1-CB2 agonist)
AM411 (CB1 agonist)
O-1184 (CB1 agonist, CB2 inverse agonist)
O-1057 (complete CB1-CB2 agonist)
Non-classical cannabinoids
CP-55 940 (complete CB1-CB2 agonist)
JWH-015 (CB2 agonist)
L-768242 (CB2 agonist)
Specific CB-2 receptor agonists
AM-1241
HU-308
L-759633
L-759656
JWH-015
JWH-133
GW405833
Eicosanoids
R-(+)-WIN-55, 212-2 (complete CB1-CB2 agonist)
Aminoalkylindoles
R-(+)-methanandamide
Arachidonoyl-2¢-chloroethylamide
Arachidonylcyclopropylamide
O-1812
2-arylimino-5,6 dihydro-4H-1, 3-thiazines
Arylsulfonamides (CB1 agonists)
Cannabinoid receptor antagonists
Diarylpyrazoles
SR141716A (rimonabant; CB1 antagonist, inverse agonist)
AM251 (CB1 antagonist, inverse agonist)
SR147778 (CB1 antagonist, inverse agonist)
AM281 (CB1 antagonist, inverse agonist)
SR144528 (CB2 antagonist, inverse agonist)
Substituted benzofuranes
LY 320135 (CB1 antagonist)
Aminoalkylidoles
AM 630 (CB2 antagonist, partial CB1 agonist)
Triazole derivatives
LH-21 (CB1 antagonist)
AM404, also known as N-arachidonoylaminophenol,[1] is an active metabolite of paracetamol (acetaminophen), responsible for all or part of its analgesic action
AM-404 is created in the body when you take Tylenol, so taking Tylenol helps Cannabinoids work
WHY IS THERE NOT A THREAD ABOUT PEOPLE WHO HAVE TAKEN THCV????
General Cancer
https://www.researchgate.net/public...noids_as_Cell_Death_and_Anticancer_Modulators
https://www.researchgate.net/publication/24427518_Cannabinoids_in_the_treatment_of_cancer
https://www.researchgate.net/public...eripheral_CB1CB2_Cannabinoid_Receptor_Ligands
Lung Cancer:
https://www.researchgate.net/public...astasis_via_intercellular_adhesion_molecule-1
Gliomas
https://www.researchgate.net/publication/5892365_Cannabinoids_and_Gliomas
Melanoma
https://www.researchgate.net/public...CB1_Receptor_as_Drug_Target_in_Human_Melanoma
Breast Cancer
https://www.researchgate.net/public..._tumor_growth_and_metastasis_of_breast_cancer
FAAH Inhibitors
https://www.researchgate.net/public...nities_from_regulating_endocannabinoid_levels
https://www.researchgate.net/publication/236250821_The_endocannabinoid_signaling_system_in_cancer
Cannabinoid Reuptake Inhibitors (CRIs)/Endo-Cannabinoid Reuptake Inhibitors (ECRIs) & Fatty Acid Amide Hydrolayse Inhibitors (FAAH Inhibitors)
Your brain has natural Cannabinoids that it produces to regulate many things, these are called EndoCannabinoids.
Here is a link to the Wiki about EndoCannabinoids function in the brain:
https://en.wikipedia.org/wiki/Endocannabinoid_system
But there is now a Molecule out there that is not a cannabinoid called LY-2183240. What this Molecule is said to do is this:
Acts both as a potent inhibitor of the reuptake of the endocannabinoid anandamide, and as an inhibitor of fatty acid amide hydrolase (FAAH), the primary enzyme responsible for degrading anandamide. This leads to markedly elevated anandamide levels in the brain, and LY-2183240 has been shown to produce both analgesic and anxiolytic effects in animal models.
Here is what Anandamine is (a natural Cannabinoid that your brain produces: http://en.wikipedia.org/wiki/Anandamide
In 1992, in Raphael Mechoulam's lab, the first such compound was identified as arachidonoyl ethanolamine and named anandamide, a name derived from the Sanskrit word for bliss and -amide. Anandamide is derived from the essential fatty acid arachidonic acid. It has a pharmacology similar to THC, although its chemical structure is different. Anandamide binds to the central (CB1) and, to a lesser extent, peripheral (CB2) cannabinoid receptors, where it acts as a partial agonist. Anandamide is about as potent as THC at the CB1 receptor.[41] Anandamide is found in nearly all tissues in a wide range of animals.[42] Anandamide has also been found in plants, including small amounts in chocolate.
AM 404 (Formed in the body when Tylenol is taken)
UCM 707
AM1172
VDM11
VDM13
OMDM1
OMDM2
LY 2183240
LY 2318912
O-2093
Inhibitors of fatty acid amide hydrolase (FAAH)
Carbamate FAAH inhibitors
OL-135
URB 597
URB 532
Bisarylimidazole derivative
Natural
Phytocannabinoids
Delta(9)-tetrahydrocannabinol (THC)
Delta(-THC
Cannabidiol
Cannabigerol
Cannabichromene
Cannabicyclol
Cannabielsoin
Cannabitriol
Miscellaneous
Endogenous
Endocannabinoids
N-arachidonoylethanolamide (anandamide; CB1-CB2 partial agonist)
2-arachidonoylglycerol (CB1 complete agonist, CB2 agonist)
2-arachidonoylglyceryl ether (noladin ether; CB1 complete agonist)
O-arachinoyl-ethanolamine (virodhamine; CB2 partial agonist, CB1 antagonist, inverse agonist)
N-arachidonyl-dopamine (CB1 agonist)
Docosatetraenoylethanolamide?
Oleamide?
N-Oleoyl dopamine?
Dihomo-linolenoylethanolamide?
Endocannabinoid-related compounds
Fatty acid derivatives
Oleamide
Oleoylethanolamide
2-oleoylglycerol
Stearoylethanolamide
Palmitoylethanolamide
2-palmitoylglycerol
Linoleoylethanolamide
2-linoleoylglycerol
Archidonoyl-aminoacid
Synthetic
Cannabinoid receptor agonists
Classical cannabinoids
Delta (-THC (CB1-CB2 agonist)
HU-210 (CB1-CB2 agonist)
AM411 (CB1 agonist)
O-1184 (CB1 agonist, CB2 inverse agonist)
O-1057 (complete CB1-CB2 agonist)
Non-classical cannabinoids
CP-55 940 (complete CB1-CB2 agonist)
JWH-015 (CB2 agonist)
L-768242 (CB2 agonist)
Specific CB-2 receptor agonists
AM-1241
HU-308
L-759633
L-759656
JWH-015
JWH-133
GW405833
Eicosanoids
R-(+)-WIN-55, 212-2 (complete CB1-CB2 agonist)
Aminoalkylindoles
R-(+)-methanandamide
Arachidonoyl-2¢-chloroethylamide
Arachidonylcyclopropylamide
O-1812
2-arylimino-5,6 dihydro-4H-1, 3-thiazines
Arylsulfonamides (CB1 agonists)
Cannabinoid receptor antagonists
Diarylpyrazoles
SR141716A (rimonabant; CB1 antagonist, inverse agonist)
AM251 (CB1 antagonist, inverse agonist)
SR147778 (CB1 antagonist, inverse agonist)
AM281 (CB1 antagonist, inverse agonist)
SR144528 (CB2 antagonist, inverse agonist)
Substituted benzofuranes
LY 320135 (CB1 antagonist)
Aminoalkylidoles
AM 630 (CB2 antagonist, partial CB1 agonist)
Triazole derivatives
LH-21 (CB1 antagonist)
AM404, also known as N-arachidonoylaminophenol,[1] is an active metabolite of paracetamol (acetaminophen), responsible for all or part of its analgesic action
AM-404 is created in the body when you take Tylenol, so taking Tylenol helps Cannabinoids work
WHY IS THERE NOT A THREAD ABOUT PEOPLE WHO HAVE TAKEN THCV????