Many years ago I was given the diagnosis of schizophrenia despite I never satisfied neither the ICD-10 nor the DSM-IV-TR criteria for this illness and I never believed that I suffer from this disease. Recently it was found by an eminent neurologist that I was right. My current, and as all evidence indicates, right diagnosis is Lyme encephalopathy or postprocessual impairment of brain tissue due to spirochetal infection, i.e., neuroborreliosis. For nearly five years I was treated, non lege artis (neither positive nor negative symptomatology present), by an antipsychotic amisulpride. I took only a low dose of it, i.e., 200 mg daily, however, continuously for nearly five years. It's 18 days since I stopped taking amisulpride. When I wake up in the morning I feel good and I am able to work mentally quite satisfactorilly. But roughly at 2 p.m. the following symptoms emerge: bradypsychism, poor memory and concentration, physical exhaustion and dysthymia.
I would like to ask you to answer the following question: Is there an estimate of how long the molecules of amisulpride "sit" as antagonists at the dopamine receptors? What drug(s) could help to minimize the length of duration of this neuroleptic withdrawal syndrome?
Kindest Regards
coriolis
I would like to ask you to answer the following question: Is there an estimate of how long the molecules of amisulpride "sit" as antagonists at the dopamine receptors? What drug(s) could help to minimize the length of duration of this neuroleptic withdrawal syndrome?
Kindest Regards
coriolis