Neuroprotection
Bluelighter
- Joined
- Apr 18, 2015
- Messages
- 1,088
is it true that symple hydrocarbon gases such as butane, propane, propylene and ethylene are psychoactive because of their action as simple asphyxiantes, depriving the brain of Oxygen resulting in neuron death or extreme damage.
Personally I think this is a simplistic explanation of their mechanism, though I don’t ever deny the risk of hypoxia induced brain damage or death from abusing hydrocarbon gases or any gaseous chemical for that matter.
First of all, if that was the case, then why do many moderate butane abusers still exist without any sines of serious brain damage, where as abusers of toluene and chlorinated solvents tend to develop parkinsons and MS like syndromes along with cognative problems.
Also, why are alkenes like ethylene, and propylene as well as cyclopropane stronger anaesthetics than their saturated alkane counterparts?
In fact ethylene and Propylene were once used as anaesthetics, but were discontinued due to concerns of flammability and their sweet yet irritating smell. In the case of ethylene, it was also noted that high concentrations, around 80% were required for deep anaesthesia thus entailing a risk of mild (usually harmless) Oxygen deficiency. Never the less, according to one article I read, it acted like Nitrous Oxide at much lower concentrations with stronger uforia but without oxidising capability, thus lacking risk of vitamin B12 deficiency associated with Nitrous.
I want to discuss this from a scientific and harm reduction perspective but I am not encouraging anyone to use inhalants particularly butane which carries additional risks of heart complications.
I am interested to know if anyone has tried Ethylene or Propylene spesifically. Before any comments about their flammability, please note that I understand that risk, also other drugs like ether have the same property.
Personally I think this is a simplistic explanation of their mechanism, though I don’t ever deny the risk of hypoxia induced brain damage or death from abusing hydrocarbon gases or any gaseous chemical for that matter.
First of all, if that was the case, then why do many moderate butane abusers still exist without any sines of serious brain damage, where as abusers of toluene and chlorinated solvents tend to develop parkinsons and MS like syndromes along with cognative problems.
Also, why are alkenes like ethylene, and propylene as well as cyclopropane stronger anaesthetics than their saturated alkane counterparts?
In fact ethylene and Propylene were once used as anaesthetics, but were discontinued due to concerns of flammability and their sweet yet irritating smell. In the case of ethylene, it was also noted that high concentrations, around 80% were required for deep anaesthesia thus entailing a risk of mild (usually harmless) Oxygen deficiency. Never the less, according to one article I read, it acted like Nitrous Oxide at much lower concentrations with stronger uforia but without oxidising capability, thus lacking risk of vitamin B12 deficiency associated with Nitrous.
I want to discuss this from a scientific and harm reduction perspective but I am not encouraging anyone to use inhalants particularly butane which carries additional risks of heart complications.
I am interested to know if anyone has tried Ethylene or Propylene spesifically. Before any comments about their flammability, please note that I understand that risk, also other drugs like ether have the same property.