MDMA + PEA + Strychnine

[motaburn]

hi

tommorow someone will be testing this combination pack.
i decided to show you the ingredients before the person ingest the stuff, in case someone here have any idea, advice, about bad, good, perhaps dangerous interactions..



T+0 :
- a heaped teaspoon of 'vital greens' (to alkalise the stomach in order for the mdma to get better absorbed.)
- 1g ginger extract (to feel good and maybe potentiate the effect)
- 2g magnesium glycinate (to reduce jaw clenching and mdma neurotoxicity, antioxidant)
- 100mg Co-enzyme Q10 (potentiate the roll)
- a teaspoon of bee pollen
- 1g Vit C as Calcium ascorbate

T+ 45 mins :
125mg of tested mdma
with grapefruit juice

T+ 100 mins :
- 500mg cayenne pepper capsule (dopamine release)
- 2.5g phenylethylamine or PEA (dopamine and norepinephrine release)
- 2g magnesium glycinate

T+120 mins :
50mg ground strychnos nux-vomica seeds (the friends love this stuff for a little uplift)

T+ 7hours :
the boring stuff
-100 mg 5-HTP + 1g EGCG (green tea extract)
and some vit B6 + vit B 12 + magnesium + vit C
- Milk Thristle (to help the liver do his job)
- 1 or 2g melatonin (antioxidant)
- 1g L-tyrosine


well I don't want to get to much into the supplements, but if you think there could be any bad interactions between MDMA, Strychnine-brucine, PEA and maybe cayenne..
thanks

 

[Folley]

That seems completely unnecessary, as well as having quite the potential for danger.


Just take the MDMA. You don't need all that other shit.

 

[JWills20]

as well as having quite the potential for danger.

Care to explain why this would be dangerous?

@OP Personally I wouldn't bother taking so many supplements, but if you wanna then all power to you.

- 500mg cayenne pepper capsule (dopamine release)
- 2.5g phenylethylamine or PEA (dopamine and norepinephrine release)

Can you discuss how these two serve to cause dopamine & norepinephrine release? I've never heard of them and I'm usually dubious about supplements actually causing catecholamine release. It's highly unusual, and usually constrained to psychoactive drugs.

 

[SilentRoller]

PEA doesn't cause DAT and NET release as such, but rather undergoes a slow conversion in the body to dopamine (much like l-tyrosine in that respect). There is no point taking PEA like to plan to, just stick with MDMA and some ALA as an antioxidant.

 

[JWills20]

PEA doesn't cause DAT and NET release as such, but rather undergoes a slow conversion in the body to dopamine (much like l-tyrosine in that respect). There is no point taking PEA like to plan to, just stick with MDMA and some ALA as an antioxidant.

I see. Yeah you're right SR, any monoamine precursor is unlikely to realistically change much acutely. Pre-cursors won't magically turn into lots of dopamine to release in hours.

I'd agree with SR's second point too. Sticking to a few anti-oxidants is adequate if your aim is strictly HR. I suppose if you're looking to 'boost' the experience then supplements aren't really the way. Look into nootropics if you wanna enhance the roll. I find a stack of noopept/piracetam/caffeine to really boost the overall effect of the caffeine. I'd imagine nootropic drugs would also boost MDMA.

 

[SilentRoller]

I'd imagine nootropic drugs would also boost MDMA

Absolutely correct! YMMV of course. Both noopept and piracetam make the roll far more clearheaded thus removing the mashyness. I didn't enjoy the 'no mashyness' feeling, although I did find the down-slope a lot nicer and gentle.

Just going back to tyrosine quickly - I did find that if I dropped about 1.5g of tyrosine around 30-40 minutes before dropping, the MDMA high was ridiculously rushy and intense. However, the price for this was that the back-end of the MDMA experience was fucking horrible (together with the comedown + days after), possibly due to the increased dopamine levels/release causing increased down-regulation/damage of 5HT neurons.

Basically, from a HR perspective, it wasn't great.

 

[Folley]

Care to explain why this would be dangerous?

Reading up a bit more, perhaps not dangerous.. though I still wouldn't recommend taking a toxin alongside MDMA.


That aside, he's eating nearly 5 grams of supplements before he even doses.. taking that many different chemicals is going to put a lot of stress on the liver and honestly isn't going to accomplish much in the end.

 

[Wicked702]

Is Calcium Ascorbate acidic, basic, or neutral? If acidic (which I *think* it is), won't it cancel out the vital greens?

 

[Brendles]

agreed, all those supplements won't amplify shit compared to a cap of noopept or a couple grams of piracetam. Just take the dose. and I'd keep the magnesium on ya though. I personally like St. Johns Wort more than I do 5-HTP for coming down.

Edit: Supplement city

 

[ebola?]

PEA doesn't cause DAT and NET release as such, but rather undergoes a slow conversion in the body to dopamine (much like l-tyrosine in that respect).

This is not true: MAO metabolizes PEA quite rapidly, little to none of it being metabolized into dopamine (it's really with substitutions like the methylenedioxy bridge that you end up with 3,4-ring hydroxylation). If it weren't broken down so rapidly, PEA would cause DA and NE release.

ebola