Is that why I always wake up in the middle of the night with an intense craving for sweet things? (I'm a heavy smoker).
Yeah, I need to get my cup size down....
opiates themselves do not delay healing.
opiates are given to patients after surgery or while experiencing pain because opiates promote rest/sleep/relaxation. The body will have a much tougher time healing from anything if the patient is experiencing constant pain. The body heals itself best when at rest/asleep.
I have no idea what kind of opiate use you are referring to here; if you're an opiate addict and neglectful in your sleep patterns and your nutrition habits, then it's likely that a burn or cut will heal much slower (compared to somebody who gets adequate sleep and decent nutrition.)
but no, as most here have stated - opiates themselves do not delay healing.
Probably b/c of your weight issue but I am no expert..
Of course, they're such good pain-killers it's possible to have something seriously wrong with you and not even notice it. Pain is a very good thing, even though it's painful, after all.
I guess it's true opiates cause cravings for sweet things; I and every one of my addict friends are all completely mad for sweets. (I like the really, really sour shit myself).
Morphine treatment resulted in a significant delay and reduction in both neutrophil and macrophage recruitment to the wound site. The delay and reduction in neutrophil reduction was attributed to altered early expression of keratinocyte derived cytokine and was independent of macrophage inflammatory protein 2 expression, whereas suppression of macrophage infiltration was attributed to suppressed levels of the potent macrophage chemoattractant monocyte chemotactic protein-1. When the effects of chronic morphine on later wound healing events were investigated, a significant suppression in angiogenesis and myofibroblast recruitment were observed in animals that received chronic morphine administration. Taken together, our findings indicate that morphine treatment results in a delay in the recruitment of cellular events following wounding, resulting in a lack of bacterial clearance and delayed wound closure.