The facilitated exchange model was the dominant explanation for amphetamine-induced dopamine release in the 1980s and 1990s. Most reviews still include it as part of the mechanism of action of amphetamine. For example, a 2009 review wrote (
https://www.ncbi.nlm.nih.gov/pmc/articles/PMC2729543/): "These studies suggest that while
facilitated exchange diffusion may contribute to AMPH-mediated monoamine release, it cannot account for all experimental observations to date."
I'm a little surprised that you would say that you are not aware of any evidence that supports the facilitated exchange hypothesis, because any review of amphetamine action lists the evidence. For example, the same review from 2009:
"
Evidence in support of this model of AMPH-induced efflux demonstrates that AMPH accumulation in rat synaptosomes is saturable, temperature-dependent, and ouabain-sensitive, implicating an active transport mechanism for AMPH35. Additional evidence for an active transport mechanism has been supported by several electrophysiology studies illustrating AMPH’s ability to generate DA-like transporter associated currents."
The way that passage was written, the author is concluding that evidence exists that is consistent with the model. I'm not saying that I agree with the model, but I am aware of it, and I don't think there is enough evidence to discard it. And until we can completely disregard this potential aspect of amphetamine's action, calling the channel mode the efflux mode is going to generate confusion.