I am sure some of you know me and my silly meth posts a while back. I have been a member for a while, using meth for the last 2 years and have done a ton of research on amps/stims in the hope of combining my first hand experience with knowledge to formulate a complete picture and opinion, mostly for personal benefit.
I haven't been here much lately as my usage has diminished quite a bit over the last year, however I still use here and there and feel the need to reconcile some of my knowledge and experience on the topic so I can hopefully move on completely. Being an info nerd i feel I need to understand a topic fully before i feel i can "walk away" happy.
I would usually avoid making silly "why did meth do this and this posts", as they are never taken seriously, but there are still a few questions, based on my experiences, that I am still unsure about. I thought I would make this final post on the topic to see if there where any real answers or not.
These are a random collection of queries that I have been unable to explain to my self to a satisfactory level. Many of these may sound like familiar symptoms on face value but it is the overall circumstance and specific situation when considering all factors like time/dosage etc that makes them strange to me. I will explain further below when its appropriate.
1. Meth and the digestive system. Its common online that there are issues here, but I have not found a clear explanation. Usually I will experience total digestive shut down with only increased "gas" which can last as long as 4-5 days, usually only as long as I am under the influence directly. I would usually need to come off the drug completely before the digestive system will operate again. Of course this can be concerning and dangerous depending on how long it lasts. What exactly causes this effect?
2. Increased body temp on alcohol. This is tough to explain and can easily be written off as normal, however my experience is very specific. Yes its common that stims and amps cause increased body temperature, however there is a very specific and certain situation I would like to clarify.
Usually after about 1-2 full days of being high and onwards, and then consuming alcohol, I often enter a state of a perceived extreme increase in body temp resulting in profuse sweating which after many experiences and analysis, does not seem to have any cause related to external influences or meth alone. Even being very euphoric this feeling of overheating and sweating is noticeable concerning which is a good indicator to take notice.
To clarify, I could consume very high doses of good meth over 4-5 days without sleep, food, much water etc and as long as i never touched alcohol I would never experience intense heat and sweating, even if my BT was very high throughout.
However if i consume alcohol after about 24 hours of high dose meth use, it can often result in a significant "fever effect" causing very heaving sweating and extreme overheating/fever no matter the external temp. This is not your common meth temp issue, but noticeably different and extreme, which is the point i am trying to make.
Lastly, to throw a curve ball indicating its a very specific timing issue, I could consume very large amounts of meth and alcohol initially within the first 24 hour window and this would never ever occur. It seems to only occur if alcohol is used after 24-36 hours from the first dose. The required timing element specifically makes this very noticeable symptom stand out and very strange to me.
My thoughts are that the vasodilation from the alcohol combined with the increased body temp from the amp is to blame, but that is just an uneducated guess. I would like more confirmation and medical detail on this if at all possible?
3. I have often seen benzos mentioned as treatments of high does stim use and even overdoses, even as far as assisting with the direct health/physical effect of the stim.
My understanding however was the the benzo treated the psychological issues of the stim and/or merely masked (but didnt treat) the negative effects, since physically it cannot directly mitigate the stimulant or reduce the potential health risks associated with the stim itself.
For example, its often said that downers mask the symptoms of stimulants and thus its easier to overdose on either. This would indicate that no matter the amount of benzo used, the risk of XXX stim dosage is still as harmful, therefore the benzo didn't really help at all in terms of the health risk, only masked the symptoms. Is this correct or do benzos actually reduce the physical health/OD risk of stimulants directly in some way?
And if so, does alcohol often a similar benefit, and what would be the risk of alcohol use for this purpose as apposed to a benzo, due to the similar mechanism of action?
Overall I am very interested in the physical effects and risks of amps and alcohol together since in my experience it seems to help in all areas, however it is never recommended here. Why its a concern is not really clear to me? Its especially interesting since benzos are recommended, however alcohol is cautioned against even though they have very similar mechanisms of action?
Norepinephrine release and SNS stimulation. I would like to confirm if dilated pupils are specifically related to norepinephrine or SNS stmulation, dopamine release or both? If both, which has the greater effect or affinity for this effect on the pupils.
Another seemingly obvious question, however in my consistent experience, I will only ever experience an effect on pupils after its obvious that SNS stimulation has kicked into a high degree, IE 24-48 hours of continued high dose meth use. Even when extreme dopamine release and euphoria is obviously in effect in the beginning period of use, pupils are not effected until SNS stimulation is obvious such as vasoc and other physical factors.
My theory is that norepinephrine is mostly to blame for this physical symptom. For this reason I use my pupils to judge SNS stimulants and potential for upcoming negative effects
5. Lastly related to the above. I would like to clarify if the action on SNS stimulation from meth for example lasts longer then the effect on dopamine. In my experience the SNS stimulation always outlasts the desired dopamine stimulation and therefore when in a situation of redosing, such as a multi day meth binge, its possible to "add up" and compound the negative SNS stimulation without the desired dopamine effects keeping pace?
At least that is my regular experience in practice this seems to occur however im not sure exactly what is actually occurring physically. Perhaps they have equal durations of effect but dopamine runs out faster after some time and norepinephrine does not, causing the effect later in a binge? Can anybody clarify?
I think that's if for now. Thanks in advance.
I haven't been here much lately as my usage has diminished quite a bit over the last year, however I still use here and there and feel the need to reconcile some of my knowledge and experience on the topic so I can hopefully move on completely. Being an info nerd i feel I need to understand a topic fully before i feel i can "walk away" happy.
I would usually avoid making silly "why did meth do this and this posts", as they are never taken seriously, but there are still a few questions, based on my experiences, that I am still unsure about. I thought I would make this final post on the topic to see if there where any real answers or not.
These are a random collection of queries that I have been unable to explain to my self to a satisfactory level. Many of these may sound like familiar symptoms on face value but it is the overall circumstance and specific situation when considering all factors like time/dosage etc that makes them strange to me. I will explain further below when its appropriate.
1. Meth and the digestive system. Its common online that there are issues here, but I have not found a clear explanation. Usually I will experience total digestive shut down with only increased "gas" which can last as long as 4-5 days, usually only as long as I am under the influence directly. I would usually need to come off the drug completely before the digestive system will operate again. Of course this can be concerning and dangerous depending on how long it lasts. What exactly causes this effect?
2. Increased body temp on alcohol. This is tough to explain and can easily be written off as normal, however my experience is very specific. Yes its common that stims and amps cause increased body temperature, however there is a very specific and certain situation I would like to clarify.
Usually after about 1-2 full days of being high and onwards, and then consuming alcohol, I often enter a state of a perceived extreme increase in body temp resulting in profuse sweating which after many experiences and analysis, does not seem to have any cause related to external influences or meth alone. Even being very euphoric this feeling of overheating and sweating is noticeable concerning which is a good indicator to take notice.
To clarify, I could consume very high doses of good meth over 4-5 days without sleep, food, much water etc and as long as i never touched alcohol I would never experience intense heat and sweating, even if my BT was very high throughout.
However if i consume alcohol after about 24 hours of high dose meth use, it can often result in a significant "fever effect" causing very heaving sweating and extreme overheating/fever no matter the external temp. This is not your common meth temp issue, but noticeably different and extreme, which is the point i am trying to make.
Lastly, to throw a curve ball indicating its a very specific timing issue, I could consume very large amounts of meth and alcohol initially within the first 24 hour window and this would never ever occur. It seems to only occur if alcohol is used after 24-36 hours from the first dose. The required timing element specifically makes this very noticeable symptom stand out and very strange to me.
My thoughts are that the vasodilation from the alcohol combined with the increased body temp from the amp is to blame, but that is just an uneducated guess. I would like more confirmation and medical detail on this if at all possible?
3. I have often seen benzos mentioned as treatments of high does stim use and even overdoses, even as far as assisting with the direct health/physical effect of the stim.
My understanding however was the the benzo treated the psychological issues of the stim and/or merely masked (but didnt treat) the negative effects, since physically it cannot directly mitigate the stimulant or reduce the potential health risks associated with the stim itself.
For example, its often said that downers mask the symptoms of stimulants and thus its easier to overdose on either. This would indicate that no matter the amount of benzo used, the risk of XXX stim dosage is still as harmful, therefore the benzo didn't really help at all in terms of the health risk, only masked the symptoms. Is this correct or do benzos actually reduce the physical health/OD risk of stimulants directly in some way?
And if so, does alcohol often a similar benefit, and what would be the risk of alcohol use for this purpose as apposed to a benzo, due to the similar mechanism of action?
Overall I am very interested in the physical effects and risks of amps and alcohol together since in my experience it seems to help in all areas, however it is never recommended here. Why its a concern is not really clear to me? Its especially interesting since benzos are recommended, however alcohol is cautioned against even though they have very similar mechanisms of action?
Norepinephrine release and SNS stimulation. I would like to confirm if dilated pupils are specifically related to norepinephrine or SNS stmulation, dopamine release or both? If both, which has the greater effect or affinity for this effect on the pupils.
Another seemingly obvious question, however in my consistent experience, I will only ever experience an effect on pupils after its obvious that SNS stimulation has kicked into a high degree, IE 24-48 hours of continued high dose meth use. Even when extreme dopamine release and euphoria is obviously in effect in the beginning period of use, pupils are not effected until SNS stimulation is obvious such as vasoc and other physical factors.
My theory is that norepinephrine is mostly to blame for this physical symptom. For this reason I use my pupils to judge SNS stimulants and potential for upcoming negative effects
5. Lastly related to the above. I would like to clarify if the action on SNS stimulation from meth for example lasts longer then the effect on dopamine. In my experience the SNS stimulation always outlasts the desired dopamine stimulation and therefore when in a situation of redosing, such as a multi day meth binge, its possible to "add up" and compound the negative SNS stimulation without the desired dopamine effects keeping pace?
At least that is my regular experience in practice this seems to occur however im not sure exactly what is actually occurring physically. Perhaps they have equal durations of effect but dopamine runs out faster after some time and norepinephrine does not, causing the effect later in a binge? Can anybody clarify?
I think that's if for now. Thanks in advance.
