Am I right in thinking that one could dramatically increase serum levels of N-arachidonoylethanolamine (anandamide) - anandamide especially but other endocannabinoids as well - by consuming the following readily obtainable substances:
Or do you suppose that fatty acid amide hydrolase is too abundant or too efficient for this combination to have a noticeable psychotropic effect? If so, are you aware of any fatty acid amide hydrolase inhibitors that can be obtained with relative facility?
- Ibuprofen (inhibits cyclooxygenase which converts arachidonic acid into various prostanoids.)
- Linoleic acid (converted into arachidonic acid)
- Lecithin (consists of: phosphatidylethanolamine, phosphatidylcholine, phosphatidylinositol, phosphatidic acid)
- Inositol
- 5-lipoxygenase inhibitor (Zileuton/Azelastine/Diethylcarbamazine/nordihydroguaiaretic acid) (arachidonate 5-lipoxygenase converts arachidonic acid into leukotrienes)
Or do you suppose that fatty acid amide hydrolase is too abundant or too efficient for this combination to have a noticeable psychotropic effect? If so, are you aware of any fatty acid amide hydrolase inhibitors that can be obtained with relative facility?