A guess putting the half-life at ~20 hours.
Luvox is a good med for OCD, I've read.
These kind of symptoms are a bit troubling. I guess there's the everyday "did you tell your doctor" thing.
That's the way to go, telling your provider, unless it's extreme that you can't wait 1-2 days for a response.
When you say "tearing up your stomach" that sounds awful, like you have an ulcer or are forming one (from the breakdown of the mucous membrane that protects the stomach, a lining on the outisde).
Insomnia is no good, too. When we get tired, our daily challenges and grievances are greatly magnified. Sleep is a must, especially for people who are in the mental health system and who are trying to improve their emotional well-being.
If you can wait until you tell your doctor without injury (which is impossible for me to guess or gauge, but I guess can be roughly estimated by your comfort) then I'd do so.
Well we all have our own meds that have worked for us. There are many meds of the same class because they all have variants that aren't well-explained by pure science. Anecdotes are likely less useful than general mechanism.
I was on Remeron for a bit. It's a tetracyclic, I believe. It tends to work a bit earlier than many other AD's. Its penchant for inducing sleep follows a relatively unique array of mechanisms. Some downstream opioidergic activity may have to do with its antidepressant effects. I believe it being an adrenergic autoreceptor agonist action essentially mimics how opioids reduce NE/E, also, along with being soporific. Similarly in histaminergic action, which it has gnarly strength at H1. This has the effect of classical conditioning on these latter two, and close comparison on the former, forming quite a med just by these effects for opioid withdrawal.
We see its range of effects on the wikipedia page. 5-HT2a antagonist is said to lead to more D1 binding in the neocortex and to facilitate cognition. I'm guessing it is an anti-emetic.
Well lamictal is a mood stabilizer. We're talking mostly different ballgames here, as mirtazepine is an antidepressant.
Some said that lamictal is better for depression than for mania, but I think that was since disproved. As other mood stabilizers, it has some effect to increase the GABA:glutmate ratio, or, more inhibitory and less excitatory activity (the two being the main inhibitory and excitatory nt's in the CNS, respectively). Yeah so blocking axonal NA+ channel influx effectively reduces the chance that a given neuron will fire and pass on the neurochemical message. But we see on Wikipedia that apparently it doesn't have some mechanisms that other mood stabilizers have.
I'd rather take remeron if I was depressed, and rather take lamictal if I more so had issues with mood stability.
