First of all hello to all members in this sub forum,
I am thinking about reaching the next bodybuilding level (>240lbs). Currently I am 220lbs (100kg) at 5'10 (1,80cm) with 13% bodyfat and my base stack for cycles is as follows:
1-1,5g test
4-6 IU pharma grade HGH
20-40 IU insulin per training day
Recently I have added 400mg nandrolone/deca for 8 weeks (bulk) followed by 8 weeks of 400-600mg trenbolone (cut). I did not notice much from 400mg nandrolone/deca. 400-600mg trenbolone was very effective during my cut.
Do you think it would be more effective and safer/healthier for adding more muscle mass (20lbs) at a minimum amount of fat to use the above base stack with 800-1000mg of deca/nandrolone 8-12 weeks (maybe at this dose I willl notice something) or 400-600mg of trenbolone 6 weeks. I would use trenbolone only 6 weeks, because of the strong adverse effect to lipids (never noticed any other sides). Which strategy would you recommend from your experience or knowledge?
Addition: Could it be possible that trenbolone could be less harmful than nandrolone for heart and blood vessels? You find many studies regarding nandrolone causing serious heart damage but less studies regarding trenbolone. Obviously there are some recent studies that aim for the potential usage of trenbolone for human purposes. Here are the recent studies.
Improvements in body composition, cardiometabolic risk factors and insulin sensitivity with trenbolone in normogonadic rats. (2015)
Thanks so much in advance.
I am thinking about reaching the next bodybuilding level (>240lbs). Currently I am 220lbs (100kg) at 5'10 (1,80cm) with 13% bodyfat and my base stack for cycles is as follows:
1-1,5g test
4-6 IU pharma grade HGH
20-40 IU insulin per training day
Recently I have added 400mg nandrolone/deca for 8 weeks (bulk) followed by 8 weeks of 400-600mg trenbolone (cut). I did not notice much from 400mg nandrolone/deca. 400-600mg trenbolone was very effective during my cut.
Do you think it would be more effective and safer/healthier for adding more muscle mass (20lbs) at a minimum amount of fat to use the above base stack with 800-1000mg of deca/nandrolone 8-12 weeks (maybe at this dose I willl notice something) or 400-600mg of trenbolone 6 weeks. I would use trenbolone only 6 weeks, because of the strong adverse effect to lipids (never noticed any other sides). Which strategy would you recommend from your experience or knowledge?
Addition: Could it be possible that trenbolone could be less harmful than nandrolone for heart and blood vessels? You find many studies regarding nandrolone causing serious heart damage but less studies regarding trenbolone. Obviously there are some recent studies that aim for the potential usage of trenbolone for human purposes. Here are the recent studies.
Improvements in body composition, cardiometabolic risk factors and insulin sensitivity with trenbolone in normogonadic rats. (2015)
Trenbolone Improves Cardiometabolic Risk Factors and Myocardial Tolerance to Ischemia-Reperfusion in Male Rats With Testosterone-Deficient Metabolic Syndrome. (2016)No evidence of adverse cardiac or hepatic effects was observed. In conclusion, improvements in body composition, lipid profile and insulin sensitivity (key risk factors for cardiometabolic disease) were achieved with six-week TREN treatment without evidence of adverse cardiovascular or hepatic effects that are commonly associated with traditional anabolic steroid misuse. Sex hormone suppression and benign prostate hyperplasia were confirmed as adverse effects of the treatment.
17β-Hydroxyestra-4,9,11-trien-3-one (trenbolone) exhibits tissue selective anabolic activity: effects on muscle, bone, adiposity, hemoglobin, and prostate. (2011)TREN treatment elicits favorable changes in body composition (specifically, in the reduction of visceral adiposity), lipid profile, insulin sensitivity, and myocardial tolerance to I-R in TEST-deficient male rats with the MetS. In comparison with TREN, the therapeutic effects of TEST were consistently less pronounced in these animals. Additionally, widespread replacement fibrosis in the myocardium and prostate hyperplasia were identified as consequences of TEST treatment and were markedly less pronounced in TREN-treated animals.
Tissue selectivity and potential clinical applications of trenbolone (17beta-hydroxyestra-4,9,11-trien-3-one): A potent anabolic steroid with reduced androgenic and estrogenic activity. (2010)Our findings indicate that TREN has advantages over supraphysiological testosterone and supports the need for future preclinical studies examining the viability of TREN as an option for androgen replacement therapy.
Compared to testosterone, 17beta-TBOH appears to induce less growth in androgen-sensitive organs which highly express the 5alpha reductase enzyme (e.g., prostate tissue and accessory sex organs). The reduced androgenic effects result from the fact that 17beta-TBOH is metabolized to less potent androgens in vivo; while testosterone undergoes tissue-specific biotransformation to more potent steroids, dihydrotestosterone and 17beta-estradiol, via the 5alpha-reductase and aromatase enzymes, respectively. Thus the metabolism of 17beta-TBOH provides a basis for future research evaluating its safety and efficacy as a means of combating muscle and bone wasting conditions, obesity, and/or androgen insensitivity syndromes in humans, similar to that of other SARMs which are currently in development.
Thanks so much in advance.