I know there's reports of Selegiline being used for potentiation
I have some Moclebemide, a RIMA. Are MAO-A inhibitors more dangerous to fuck with than MAO-B in regards to drug interactions (more specifically SNRI action)
I know taking high doses of moclebemide on it's own results in too much SERT or NET
I was thinking of taking 150mg or even 75mg (half a pill) to potentiate DXM but I don't want SS
I'm currently on 300-400mg DXM and just cracking my second bottle (600mg to play with)
What's the risk of taking 75mg Moclobemide? 600mg was my theraputic dose, I didn't notice any effects to my state of mind until about 900mg (I never bothered trying 825mg.)
It has a very short half-life (1-2 hours) since there's none of it in my system (when it builds up the h/l increases)
Why are MAO-Bs used over MAO-As? MAO-B doesn't affect (nor)epinephrine and sert only dopamine therefore there's no risk of SS?
I have some Moclebemide, a RIMA. Are MAO-A inhibitors more dangerous to fuck with than MAO-B in regards to drug interactions (more specifically SNRI action)
I know taking high doses of moclebemide on it's own results in too much SERT or NET
I was thinking of taking 150mg or even 75mg (half a pill) to potentiate DXM but I don't want SS
I'm currently on 300-400mg DXM and just cracking my second bottle (600mg to play with)
What's the risk of taking 75mg Moclobemide? 600mg was my theraputic dose, I didn't notice any effects to my state of mind until about 900mg (I never bothered trying 825mg.)
It has a very short half-life (1-2 hours) since there's none of it in my system (when it builds up the h/l increases)
Why are MAO-Bs used over MAO-As? MAO-B doesn't affect (nor)epinephrine and sert only dopamine therefore there's no risk of SS?