Just because it looks like no one addressed that question, I assume he was abbreviating
N-
Acetyl
Cysteine, which primarily benefits heavy smokers and drinkers, and can be your liver's greatest ally
In fact, I think there are some "hangover pills" being sold today that are basically just overpriced NAC in a cap...
-
And as for daily use of threshold entheo/entacto's, I've been keeping
a steady routine of MDAI intake ~2-3 doses a day ~4 days a week for nearly 2 months now and haven't noticed any decrease in effectiveness.
Perhaps AMT would follow a similar course?
Makes me wonder about all these reports you hear from people "
if you take serotonin-releasing compounds frequently, you will gradually dilute your native 5HT receptor density, experience an increase in depression symptoms, etc etc".
Hasn't been remotely the case for me.
Perhaps one cannot rationally apply this rule of thumb to
all serotonin releasing compounds?
Each different 5HT-releasing drug will affect a different spread of neurological regions. Is it not possible there exists a compound (AMT? MDAI? MDMAI? etc.) which binds to just the right nuclei ripe with, say,
Glial cells that are complex enough to modulate the health/functionality of the Reward System neurons, such that they may safely endure a permanent state of empathogenic toxicosis
Although my personal experience with frequent use of MDAI has so far been favorable, I also take daily doses of Bacopa Monnieri and Rhodiola Rosea extract, which are both known to affect a great deal of elements in a person's 5HT system, including but
not limited to
cell density.
DMT, also, has a fair pile of data suggesting it changes one's native 5HT system dramatically over time, and this is good era for anyone really interested in becoming a regular DMT voyager to have that chance without much difficulty (myself included
).
What a veritable cauldron of competing (stewing?) cognitive alterations some of our brains are, eh?