Alcohol 2-Methyl-2-Butanol, any activity at GABA A?

[《Plasticity》]

I'm having a bit of trouble finding out if 2m2b has any sort of GABA A activity, more importantly if there is any cross-tolerance with benzos. I assume not, but it doesn't hurt to make sure. I ask because I came off a small benzo habit a little while back and ever since I can't consume alcohol as it throws me into serious rebound, I'm wondering if I will be good with consuming 2m2b. From what I understand the effects are primarily from GABA B agonism, but I'd like a confirmation of lacking GABA A activity. Perhaps this thread is better suited for NPD, but then again it's a simple one answer question.
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After a bit of digging I may have found the study with my answer, only I'm too dumb to understand exactly what it means . I'd really appreciate it if someone with a stronger grasp on pharmacology can decipher this for a layman. Here's a snippet from the abstract, TAA is Tertiary-Amyl Alcohol is 2m2b... all the same. So am I right when assuming this means 2m2b has even stronger GABA A agonism than alcohol? That doesn't sound right at all... but that's likely because it's not, as the type of effect had on GABA A isn't even spoken of as far as I can tell... and I'm not exactly sure what the correlation between this 36Cl- and GABA A is.

To more directly assess the effects of the ethers and their alcohol precursors on GABAA receptor function, the uptake of 36Cl- was measured in synaptoneurosomes, a preparation of closed membrane sacs comprised of pre- and postsynaptic membranes from adult rat cerebral cortex. of the compounds caused a concentration-dependent enhancement of muscimol-stimulated uptake of 36CI-, which diminished with further increasing concentrations. The potency of the enhancement by the compounds was in the rank order: MTBE = TAME > TAA = ETBE > TBA > ethanol. The half-maximally effective concentration (EC50) for the facilitation of muscimol-stimulated 36Cl- uptake ranged from 0.06 to 3 mM, and that for the higher-dose inhibitory effect (IC50) ranged from 3 to 50 mM.

http://www.pubfacts.com/detail/1510...lites-on-gamma-aminobutyric-acidA-GABAA-recep

The results demonstrated that the short-chain t-ethers and their t-alcohol metabolites inhibit binding at the convulsant site of the GABAAreceptor.

http://www.sciencedirect.com/science/article/pii/S0378427402000206?via=ihub#FIG3

Ahh, this was found in that last link... so GABA A antagonism? Apparently the strongest of the six tested in correlation to 2m2b's long carbon chain. This thread is so messy 8(