It's hard to tell if you're being serious about your stance or just seeking to elicit a response/argument. I'm not trying to be a doomsayer regarding meth - the harms can be wildly overrated, certainly, and much literature (especially of the mainstream media variety) is patent hyperbole.
However you're quoting a paper from 1975 surveying deaths in the period 1940-75 - decades before many of the potential harms of meth became more clearly understood and recorded (indeed many of the mechanisms we still don't fully understand). Even the idea that the
abuse of sympathomimetic amines isn't substantially harmful to the cardiovascular system - even just from a theoretical standpoint - is an odd one to hold, given what we already know.
Based on the paper you quote, I assume you think the focus of concern should be restricted to imminent and direct morbidity from meth. But while acute risks are indeed worrying, they're certainly not the only concern for most meth/amp/stimulant users (although in OP's case, given his/her evident sensitivity to the acute intropic and chronotropic effects of meth, death is clearly a potential outcome (!), hence the HR advice given).
Nevertheless the dominant cardiovascular concern for meth/amp and other stimulant users is more typically the chronic harms - ones that build slowly over time like cardiomyopathies, valvular damage, aortic dilatation and so on. These can increase the risk of premature death, potentially taking years or even decades off a person's normal lifespan, despite not being obviously and directly related to morbidity at the time of death - and so inconveniently not registering in any coroner's report (assuming one's even written - which is rarely the case).
Despite that, and the consequent inadequacy of information we have to hand, even a tip-of-the-iceberg analysis suggests meth use is present in at least
5% of all patients presenting to the ER with heart failure [
https://www.ncbi.nlm.nih.gov/pubmed/18940295] and
40% of patients under the age of 45 admitted to hospital with cardiomyopathy [
https://www.ncbi.nlm.nih.gov/pubmed/17275458]. Acute coronary syndrome (ie unstable angina and acute myocardial infarction) was diagnosed in
25% of US emergency department patients presenting with chest pain following meth use [
https://www.ncbi.nlm.nih.gov/pubmed/12745036]
A selection of reports and studies discussing heart attacks (MI) resulting from meth/amp abuse can be found here:
https://www.ncbi.nlm.nih.gov/pubmed/15011896
https://www.ncbi.nlm.nih.gov/pubmed/12745036
https://www.ncbi.nlm.nih.gov/pubmed/10978660
https://www.ncbi.nlm.nih.gov/pubmed/10097363
https://www.ncbi.nlm.nih.gov/pubmed/17565561
Acute aortic dissection, another well-known complication of meth use, can be read about here:
https://www.ncbi.nlm.nih.gov/pubmed/9987866
https://www.ncbi.nlm.nih.gov/pubmed/10097363
https://www.ncbi.nlm.nih.gov/pubmed/7815027
Meth/amp is also strongly associated with cardiac arrhythmias and sudden cardiac death. You can read more about these cases here:
https://www.ncbi.nlm.nih.gov/pubmed/10344175
https://www.ncbi.nlm.nih.gov/pubmed/7588189
https://www.ncbi.nlm.nih.gov/pubmed/6482074
https://www.ncbi.nlm.nih.gov/pubmed/2979169
https://www.ncbi.nlm.nih.gov/pubmed/7840041
https://www.ncbi.nlm.nih.gov/pubmed/2293467
https://www.ncbi.nlm.nih.gov/pubmed/12877759
https://www.ncbi.nlm.nih.gov/pubmed/6519024
As for the more chronic pathologies, they also include underlying processes and mechanisms that lead to many of the acute outcomes listed above. So for example aortic dissection typically involves prolonged and repeated exposure to elevated BP and heart rate, leading to aortic dilatation, which slowly expands and weakens for years before the aorta finally ruptures in an acute episode. And prolonged tachycardia and catecholamine exposure can modify the behaviour of the heart's pacemaker and its elecrophysiological behaviour (affecting pulse conductivity, for example), leading to atrial fibrillation, which can then lead to clots, thrombus and ultimately MI, sudden death and so on.
However the classic chronic issues are coronary artery disease (CAD) and cardiomyopathy. Minimal to severe CAD is found in around 1-in-5 (20%) of meth users compared to 1-in-20 of the normal population, and occurs at a significantly younger age [
https://www.ncbi.nlm.nih.gov/pubmed/17565561].
But as quoted in that paper: "cardiac pathology takes time to develop [and] there may be a long ‘incubation’ period prior to methamphetamine-related death." As already suggested, this implies many of the harms inevitably go unrecorded or get blamed on traditional causes like ageing, poor genetics, a lack of exercise and unhealthy Western diets.
These concerns about the largely under-reported cardiovascular harms aren't just limited to American researchers either:
[
https://www.ncbi.nlm.nih.gov/pubmed/26706652]
In
Crystal Methamphetamine‐Associated Cardiomyopathy: Tip of the Iceberg? [
https://www.ncbi.nlm.nih.gov/pubmed/14705845], the authors summarise the cardiovascular-related harms quite nicely:
Evidence for all those harms/claims can be linked directly through the paper (I had to remove the messy hyperlinks unfortunately).
In
Cardiovascular Responses Elicited by the “Binge” Administration of Methamphetamine [
https://www.ncbi.nlm.nih.gov/pubmed/11907169], the authors have begun to investigate the impact of repeated binge dosing of meth on the cardiovascular system, since this more accurately reflects typical usage patterns, noting that:
I bring this tendency for binging to aggravate the heart's reaction to meth's pressor effect up as it's of direct concern to OP, who's heart is clearly quite sensitive as it is. Which is a very good reason for OP to further ponder the gravity of his/her addiction to meth, and to try even harder to stop. And all without even needing to consider the escalating risks to the brain, other organs, long-term mental health, intensified addiction and so on.