Whilst I don't typically hang about in the MDMA subforum, as, being autistic, I don't really find the artificially induced entactogenic state to be..well, it feels forced on me and artificial, as if running 'NT' as an operating system on emulator software (I.e the drug) on autistic wetware. Its not the way I naturally am, and whilst it can have its values, its typically a shallow experience to me (unlike the entactogen AMT, thats also a full on 5H2a receptor agonist trip in the card-carrying psychedelic sense, that I respond better to) and I find I really don't like the idea of something, even temporarily, making me closer to 'non autistic/NT-like' (no offense to the neurotypicals here, its just a personal area of comfortability thing), kind of gives me the creeps in that sensee.
I noticed this thread, though. And should point out that longterm use of this might be dangerous. This is very, VERY close to fenfluramine, the now withdrawn diet drug stimulant, thats also a strong serotonergic drug. In the case of fenfluramine vs this, its simply a 1 for 1 replacement of the 3-trilfluoromethyl substituent present in fenfluramines structure for a 3-fluoro, still a highly electron withdrawing group, fenfluramine is 3-TFM-N-ethylamphetamine. It had a very long half life, and so did a similarly active metabolite, the N-desethylated norfenfluramine, and both were possessed of powerful agonist effects at 5HT2b receptors, which in people taking it resulted in sustained, intense 5HT2b receptor agonism that had a tendency towards causing hypertrophic growth of IIRC the tricuspid valve, within the heart, with a thickening of the valve flaps and resultant cardiac problems, as 5HT2b receptors.
Now I'm not certain that this is a strong 5HT2b agonist, or for 3-fluoro (N-methyl)amphetamine, rather I'm basing this warning off the fact that both these are N-ethylated amphetamines with a single ring-substitution, located at the 3' position phenyl carbon that is of a highly electron withdrawing nature. And so is an aryl fluoride like this.
So it isn't beyond the realms of the foreseeable that this too could be a strong 5HT2b receptor agonist as well.